Antibodies against CD14 protect primates from endotoxin-induced shock - Wikidata - awgldk - TriplyDB

Antibodies against CD14 protect primates from endotoxin-induced shock

Antibodies against CD14 protect primates from endotoxin-induced shock

http://www.wikidata.org/entity/Q37360210

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CD1d- and MR1-Restricted T Cells in Sepsis Peptidoglycan and lipoteichoic acid modify monocyte phenotype in human whole blood Structural and functional consequences of alveolar cell recognition by CD8(+) T lymphocytes in experimental lung disease.Characterization of a murine monoclonal antibody to Cryptococcus neoformans polysaccharide that is a candidate for human therapeutic studies.CD14 inhibition efficiently attenuates early inflammatory and hemostatic responses in Escherichia coli sepsis in pigs.Pathogenesis of septic shock in Pseudomonas aeruginosa pneumonia Leptospiral lipopolysaccharide activates cells through a TLR2-dependent mechanism.Role of plasma, lipopolysaccharide-binding protein, and CD14 in response of mouse peritoneal exudate macrophages to endotoxin Critical role of CD14 for production of proinflammatory cytokines and cytokine inhibitors during sepsis with failure to alter morbidity or mortality What are the microbial components implicated in the pathogenesis of sepsis? Report on a symposium.Porphyromonas gingivalis lipopolysaccharide: an unusual pattern recognition receptor ligand for the innate host defense system.Towards a rational development of anti-endotoxin agents: novel approaches to sequestration of bacterial endotoxins with small molecules.Nrf2 is a critical regulator of the innate immune response and survival during experimental sepsis.Liposomal lipopolysaccharide initiates TRIF-dependent signaling pathway independent of CD14.The bacterial endotoxin lipopolysaccharide has the ability to target the brain in upregulating its membrane CD14 receptor within specific cellular populations.Novel therapies for sepsis: antiendotoxin therapies.Recent developments in the treatment of sepsis.Receptors, mediators, and mechanisms involved in bacterial sepsis and septic shock.Methemoglobin is an endogenous toll-like receptor 4 ligand-relevance to subarachnoid hemorrhage.Role of CD14 in responses to clinical isolates of Escherichia coli: effects of K1 capsule expression CD14 contributes to warm hepatic ischemia-reperfusion injury in mice Tumor necrosis factor- alpha production to lipopolysaccharide stimulation by donor cells predicts the severity of experimental acute graft-versus-host disease.Chimeric anti-CD14 IGG2/4 Hybrid antibodies for therapeutic intervention in pig and human models of inflammation.Systemic CD14 inhibition attenuates organ inflammation in porcine Escherichia coli sepsis Double blockade of CD14 and complement C5 abolishes the cytokine storm and improves morbidity and survival in polymicrobial sepsis in mice.The differential impact of disulfide bonds and N-linked glycosylation on the stability and function of CD14.Characterization of an intravenous lipopolysaccharide inflammation model in broiler chickens.Lipopolysaccharide induces expression of fibronectin alpha 5 beta 1-integrin receptors in human monocytic cells in a protein kinase C-dependent fashion.Pathobiology of lipopolysaccharide.Changes of inflammation-associated cytokine expressions during early phase of experimental endotoxic shock in macaques Effect of CD14 blockade on endotoxin-induced acute lung injury in mice.Dietary L-arginine supplementation attenuates lipopolysaccharide-induced inflammatory response in broiler chickens.Effects of systemic immunogenic insults and circulating proinflammatory cytokines on the transcription of the inhibitory factor kappaB alpha within specific cellular populations of the rat brain.IC14, a CD14 specific monoclonal antibody, is a potential treatment for patients with severe sepsis Evidence of participation of soluble CD14 in the host response to microbial invasion of the amniotic cavity and intra-amniotic inflammation in term and preterm gestations

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P2860

Antibodies against CD14 protect primates from endotoxin-induced shock

http://www.wikidata.org/entity/Q37360210

description

article científic

@ca

article scientifique

@fr

articolo scientifico

@it

artigo científico

@pt

bilimsel makale

@tr

scientific article published on October 1996

@en

vedecký článok

@sk

vetenskaplig artikel

@sv

videnskabelig artikel

@da

vědecký článek

@cs

name

Antibodies against CD14 protect primates from endotoxin-induced shock

@en

Antibodies against CD14 protect primates from endotoxin-induced shock.

@nl

type

label

Antibodies against CD14 protect primates from endotoxin-induced shock

@en

Antibodies against CD14 protect primates from endotoxin-induced shock.

@nl

prefLabel

Antibodies against CD14 protect primates from endotoxin-induced shock

@en

Antibodies against CD14 protect primates from endotoxin-induced shock.

@nl

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P1433

Journal of Clinical Investigation

P1476

Antibodies against CD14 protect primates from endotoxin-induced shock

@en

P2093

A M Moriarty

http://www.w3.org/2001/XMLSchema#string

D J Leturcq

http://www.w3.org/2001/XMLSchema#string

G Talbott

http://www.w3.org/2001/XMLSchema#string

R J Ulevitch

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R K Winn

http://www.w3.org/2001/XMLSchema#string

T R Martin

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P2860

Structure and function of lipopolysaccharide binding protein

CD14, a receptor for complexes of lipopolysaccharide (LPS) and LPS binding protein

Interferon gamma receptor deficient mice are resistant to endotoxic shock.

Lipopolysaccharide activation of human endothelial and epithelial cells is mediated by lipopolysaccharide-binding protein and soluble CD14.

Gram-negative sepsis and the adult respiratory distress syndrome.

Isolation of a lipopolysaccharide-binding acute phase reactant from rabbit serum.

Glycosyl-phosphatidylinositol-anchored or integral membrane forms of CD14 mediate identical cellular responses to endotoxin

Septic shock in humans. Advances in the understanding of pathogenesis, cardiovascular dysfunction, and therapy.

A region of human CD14 required for lipopolysaccharide binding.

CD14 and innate recognition of bacteria.

P304

1533-1538

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scholarly article

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10.1172/JCI118945

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7

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98

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1996-10-01T00:00:00Z

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14371293

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8833900

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507584

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