Renal primary cilia lengthen after acute tubular necrosis. - Wikidata - awgldk - TriplyDB

Renal primary cilia lengthen after acute tubular necrosis.

Renal primary cilia lengthen after acute tubular necrosis.

http://www.wikidata.org/entity/Q37367710

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Delayed graft function in the kidney transplant Measuring the primary cilium length: improved method for unbiased high-throughput analysis.Effects of TORC1 Inhibition during the Early and Established Phases of Polycystic Kidney Disease Role of epidermal primary cilia in the homeostasis of skin and hair follicles Primary cilia dynamics instruct tissue patterning and repair of corneal endothelium The zebrafish foxj1a transcription factor regulates cilia function in response to injury and epithelial stretch Target-of-rapamycin complex 1 (Torc1) signaling modulates cilia size and function through protein synthesis regulation.Carcinogens induce loss of the primary cilium in human renal proximal tubular epithelial cells independently of effects on the cell cycle A novel mutation causing nephronophthisis in the Lewis polycystic kidney rat localises to a conserved RCC1 domain in Nek8 Shear stress-dependent regulation of apical endocytosis in renal proximal tubule cells mediated by primary cilia.Primary cilia disappear in rat podocytes during glomerular development.A chemical screen identifies class a g-protein coupled receptors as regulators of cilia.Nucleofection disrupts tight junction fence function to alter membrane polarity of renal epithelial cells.The fate of bone marrow-derived cells carrying a Polycystic Kidney Disease mutation in the genetically normal kidney Development of a method for the measurement of primary cilia length in 3D.Bridging the gap: a Canadian perspective on translational kidney research.Genome-wide association study of acute kidney injury after coronary bypass graft surgery identifies susceptibility loci.Cystogenesis and elongated primary cilia in Tsc1-deficient distal convoluted tubules.Unilateral nephrectomy elongates primary cilia in the remaining kidney via reactive oxygen species.Primary cilia and kidney injury: current research status and future perspectives The fate of the primary cilium during myofibroblast transition Wnt-signaling pathways in progressive renal fibrosis.Ultrastructure of cilia and flagella - back to the future!Visualizing renal primary cilia.The ciliary flow sensor and polycystic kidney disease.Flagellar Synchronization Is a Simple Alternative to Cell Cycle Synchronization for Ciliary and Flagellar Studies.The Biology of Ciliary Dynamics.Reduction of oxidative stress during recovery accelerates normalization of primary cilia length that is altered after ischemic injury in murine kidneys.Role of primary cilia in non-dividing and post-mitotic cells.Primary Cilia in Cystic Kidney Disease.Identification of signaling pathways regulating primary cilium length and flow-mediated adaptation.Galectin-3, a novel centrosome-associated protein, required for epithelial morphogenesis.Primary cilia elongation in response to interleukin-1 mediates the inflammatory response.Chronic Hypobaric Hypoxia Modulates Primary Cilia Differently in Adult and Fetal Ovine Kidneys.Human kidney proximal tubule-on-a-chip for drug transport and nephrotoxicity assessment.Tendon cell ciliary length as a biomarker of in situ cytoskeletal tensional homeostasis.The CPLANE protein Intu protects kidneys from ischemia-reperfusion injury by targeting STAT1 for degradation.Increased Primary Cilia in Idiopathic Pulmonary Fibrosis.Endothelial dysfunction in pulmonary arterial hypertension: loss of cilia length regulation upon cytokine stimulation.Can Tissue Cilia Lengths and Urine Cilia Proteins Be Markers of Kidney Diseases?

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P2860

Renal primary cilia lengthen after acute tubular necrosis.

http://www.wikidata.org/entity/Q37367710

description

article científic

@ca

article scientifique

@fr

articolo scientifico

@it

artigo científico

@pt

bilimsel makale

@tr

scientific article published on 16 July 2009

@en

vedecký článok

@sk

vetenskaplig artikel

@sv

videnskabelig artikel

@da

vědecký článek

@cs

name

Renal primary cilia lengthen after acute tubular necrosis.

@en

Renal primary cilia lengthen after acute tubular necrosis.

@nl

type

label

Renal primary cilia lengthen after acute tubular necrosis.

@en

Renal primary cilia lengthen after acute tubular necrosis.

@nl

prefLabel

Renal primary cilia lengthen after acute tubular necrosis.

@en

Renal primary cilia lengthen after acute tubular necrosis.

@nl

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Journal of the American Society of Nephrology

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Renal primary cilia lengthen after acute tubular necrosis.

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Elizabeth Verghese

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Junli Zhuang

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Prudence A Hill

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Raphael Weidenfeld

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Robyn G Langham

http://www.w3.org/2001/XMLSchema#string

P2860

Cilia and the cell cycle?

Inversin, the gene product mutated in nephronophthisis type II, functions as a molecular switch between Wnt signaling pathways

Restoration of tubular epithelial cells during repair of the postischemic kidney occurs independently of bone marrow-derived stem cells.

Localization of proliferating cell nuclear antigen, vimentin, c-Fos, and clusterin in the postischemic kidney. Evidence for a heterogenous genetic response among nephron segments, and a large pool of mitotically active and dedifferentiated cells.

Influence of cyclosporin, tacrolimus and rapamycin on renal function and arterial hypertension after renal transplantation.

Pathophysiology of ischemic acute renal failure.

Defects in ciliary localization of Nek8 is associated with cystogenesis.

The vertebrate primary cilium is a sensory organelle.

Dedifferentiation and proliferation of surviving epithelial cells in acute renal failure.

Acute kidney injury and aberrant planar cell polarity induce cyst formation in mice lacking renal cilia.

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2147-2153

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scholarly article

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10.1681/ASN.2008101105

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10

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20

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Sharon D. Ricardo

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2009-07-16T00:00:00Z

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26676526

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19608704

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2754102

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