Induction and repair of DNA double strand breaks: the increasing spectrum of non-homologous end joining pathways.
about
DNA ligase III and DNA ligase IV carry out genetically distinct forms of end joining in human somatic cellsCDK targeting of NBS1 promotes DNA-end resection, replication restart and homologous recombinationRobust chromosomal DNA repair via alternative end-joining in the absence of X-ray repair cross-complementing protein 1 (XRCC1)Leukocyte DNA damage after reduced and conventional absorbed radiation doses using 3rd generation dual-source CT technologyNovel Biological Approaches for Testing the Contributions of Single DSBs and DSB Clusters to the Biological Effects of High LET RadiationDNA damage and repair in plants - from models to cropsAlternative Okazaki Fragment Ligation Pathway by DNA Ligase IIIDNA-PK: a dynamic enzyme in a versatile DSB repair pathwaySources of DNA double-strand breaks and models of recombinational DNA repairDNA double-strand-break complexity levels and their possible contributions to the probability for error-prone processing and repair pathway choiceAntigenic variation and the generation of diversity in malaria parasitesHoming endonucleases: DNA scissors on a missionMicroRNA and signal transduction pathways in tumor radiation responseATP driven structural changes of the bacterial Mre11:Rad50 catalytic head complexStructure of Mre11–Nbs1 complex yields insights into ataxia-telangiectasia–like disease mutations and DNA damage signalingTRF2 interaction with Ku heterotetramerization interface gives insight into c-NHEJ prevention at human telomeresHuman LIGIV is synthetically lethal with the loss of Rad54B-dependent recombination and is required for certain chromosome fusion events induced by telomere dysfunctionAn inverse switch in DNA base excision and strand break repair contributes to melphalan resistance in multiple myeloma cellsDNA ligases I and III cooperate in alternative non-homologous end-joining in vertebratesDNA repair deficiency as a susceptibility marker for spontaneous lymphoma in golden retriever dogs: a case-control studyDeregulation of DNA double-strand break repair in multiple myeloma: implications for genome stabilitySingle nucleotide polymorphisms as susceptibility, prognostic, and therapeutic markers of nonsmall cell lung cancerThe miR-99 family regulates the DNA damage response through its target SNF2HCoupling endonucleases with DNA end-processing enzymes to drive gene disruption.Systems biology analysis of Drosophila in vivo screen data elucidates core networks for DNA damage repair in SCA1.RAG2's acidic hinge restricts repair-pathway choice and promotes genomic stability.Cancer risk at low doses of ionizing radiation: artificial neural networks inference from atomic bomb survivors.Werner syndrome protein positively regulates XRCC4-like factor transcriptionPARP3 affects the relative contribution of homologous recombination and nonhomologous end-joining pathways.A fine-scale dissection of the DNA double-strand break repair machinery and its implications for breast cancer therapy.Requirement for Parp-1 and DNA ligases 1 or 3 but not of Xrcc1 in chromosomal translocation formation by backup end joining.Non-SMC elements 1 and 3 are required for early embryo and seedling development in Arabidopsis.Non-homologous end joining: emerging themes and unanswered questions.Mycobacterium tuberculosis Ku can bind to nuclear DNA damage and sensitize mammalian cells to bleomycin sulfate.Screening of Duchenne muscular dystrophy (DMD) mutations and investigating its mutational mechanism in Chinese patients.High frequency targeted mutagenesis using engineered endonucleases and DNA-end processing enzymes.DNA repair kinetics in SCID mice Sertoli cells and DNA-PKcs-deficient mouse embryonic fibroblasts.The role of ATM in the deficiency in nonhomologous end-joining near telomeres in a human cancer cell line.A multistep genomic screen identifies new genes required for repair of DNA double-strand breaks in Saccharomyces cerevisiae.Human RECQ1 interacts with Ku70/80 and modulates DNA end-joining of double-strand breaks
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Induction and repair of DNA double strand breaks: the increasing spectrum of non-homologous end joining pathways.
description
article científic
@ca
article scientifique
@fr
articolo scientifico
@it
artigo científico
@pt
bilimsel makale
@tr
scientific article published on 15 February 2011
@en
vedecký článok
@sk
vetenskaplig artikel
@sv
videnskabelig artikel
@da
vědecký článek
@cs
name
Induction and repair of DNA do ...... mologous end joining pathways.
@en
Induction and repair of DNA do ...... mologous end joining pathways.
@nl
type
label
Induction and repair of DNA do ...... mologous end joining pathways.
@en
Induction and repair of DNA do ...... mologous end joining pathways.
@nl
prefLabel
Induction and repair of DNA do ...... mologous end joining pathways.
@en
Induction and repair of DNA do ...... mologous end joining pathways.
@nl
P1433
P1476
Induction and repair of DNA do ...... mologous end joining pathways.
@en
P2093
Emil Mladenov
George Iliakis
P356
10.1016/J.MRFMMM.2011.02.005
P407
P577
2011-02-15T00:00:00Z