Amyloid-beta-(1-42) increases ryanodine receptor-3 expression and function in neurons of TgCRND8 mice. - Wikidata - awgldk - TriplyDB

Amyloid-beta-(1-42) increases ryanodine receptor-3 expression and function in neurons of TgCRND8 mice.

Amyloid-beta-(1-42) increases ryanodine receptor-3 expression and function in neurons of TgCRND8 mice.

http://www.wikidata.org/entity/Q38308416

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The ER stress factor XBP1s prevents amyloid-beta neurotoxicity The role of ryanodine receptor type 3 in a mouse model of Alzheimer disease Calcium-sensitive regulation of monoamine oxidase-A contributes to the production of peroxyradicals in hippocampal cultures: implications for Alzheimer disease-related pathology.Genetic interactions found between calcium channel genes modulate amyloid load measured by positron emission tomography.Calcium signaling and amyloid toxicity in Alzheimer disease.Susceptibility to Calcium Dysregulation during Brain Aging.Role of presenilins in neuronal calcium homeostasis Early calcium dysregulation in Alzheimer's disease: setting the stage for synaptic dysfunction.Amyloid-beta: a crucial factor in Alzheimer's disease.Stabilizing ER Ca2+ channel function as an early preventative strategy for Alzheimer's disease.Neuronal store-operated calcium entry pathway as a novel therapeutic target for Huntington's disease treatment.Endoplasmic reticulum Ca(2+) handling in excitable cells in health and disease Altered ryanodine receptor expression in mild cognitive impairment and Alzheimer's disease.Changes in the physiology of CA1 hippocampal pyramidal neurons in preplaque CRND8 mice.Mitochondrial- and endoplasmic reticulum-associated oxidative stress in Alzheimer's disease: from pathogenesis to biomarkers.C1q-induced LRP1B and GPR6 proteins expressed early in Alzheimer disease mouse models, are essential for the C1q-mediated protection against amyloid-β neurotoxicity.Dysregulation of neural calcium signaling in Alzheimer disease, bipolar disorder and schizophrenia Tau pathology-mediated presynaptic dysfunction The pathogenesis of Alzheimers disease is it a lifelong "calciumopathy"?The dysregulation of intracellular calcium in Alzheimer disease.Control of intracellular calcium signaling as a neuroprotective strategy Neuronal calcium signaling, mitochondrial dysfunction, and Alzheimer's disease.New therapeutic targets in Alzheimer's disease: brain deregulation of calcium and zinc.Consequences of inhibiting amyloid precursor protein processing enzymes on synaptic function and plasticity.Calcium regulation of neural rhythms, memory and Alzheimer's disease.Neuronal calcium signaling: function and dysfunction.Ryanodine receptors: physiological function and deregulation in Alzheimer disease.Amyloid beta receptors responsible for neurotoxicity and cellular defects in Alzheimer's disease.Post-translational remodeling of ryanodine receptor induces calcium leak leading to Alzheimer's disease-like pathologies and cognitive deficits.Dysregulation of neuronal calcium homeostasis in Alzheimer's disease - A therapeutic opportunity?Emerging pathways driving early synaptic pathology in Alzheimer's disease.Mechanism of Ca2+ disruption in Alzheimer's disease by presenilin regulation of InsP3 receptor channel gating.Molecular and functional identification of a mitochondrial ryanodine receptor in neurons.Phosphorylation of histone H2AX in the mouse brain from development to senescence.Ca2+/calmodulin-dependent protein kinase II promotes neurodegeneration caused by tau phosphorylated at Ser262/356 in a transgenic Drosophila model of tauopathy.Phosphoproteome analysis of an early onset mouse model (TgCRND8) of Alzheimer's disease reveals temporal changes in neuronal and glia signaling pathways.Calcium signalling and Alzheimer's disease.Early Alterations of Hippocampal Neuronal Firing Induced by Abeta42.Critical review: involvement of endoplasmic reticulum stress in the aetiology of Alzheimer's disease.Crosstalk between Endoplasmic Reticulum Stress and Protein Misfolding in Neurodegenerative Diseases

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Amyloid-beta-(1-42) increases ryanodine receptor-3 expression and function in neurons of TgCRND8 mice.

http://www.wikidata.org/entity/Q38308416

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Amyloid-beta-(1-42) increases ...... on in neurons of TgCRND8 mice.

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Charlene Supnet

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David Westaway

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Hong Kong

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Jeff Grant

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Michael Mayne

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P2860

Deletion of the ryanodine receptor type 3 (RyR3) impairs forms of synaptic plasticity and spatial learning

The amyloid hypothesis of Alzheimer's disease: progress and problems on the road to therapeutics

Modulation of a voltage-gated calcium channel linked to activation of glutamate receptors and calcium-induced calcium release in the catfish retina

Calcium release from presynaptic ryanodine-sensitive stores is required for long-term depression at hippocampal CA3-CA3 pyramidal neuron synapses.

Calcium signaling in the ER: its role in neuronal plasticity and neurodegenerative disorders.

Presenilin, Notch, and the genesis and treatment of Alzheimer's disease.

Gene regulation and DNA damage in the ageing human brain.

Molecular genetics of Ca(2+) stores and intracellular Ca(2+) signalling.

Calcium dysregulation, IP3 signaling, and Alzheimer's disease.

Advancing age alters the expression of the ryanodine receptor 3 isoform in adult rat superior cervical ganglia.

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38440-38447

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10.1074/JBC.M606736200

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50

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281

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2006-10-18T00:00:00Z

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