Increases in p53 expression induce CTGF synthesis by mouse and human hepatocytes and result in liver fibrosis in mice. - Wikidata - awgldk - TriplyDB

Increases in p53 expression induce CTGF synthesis by mouse and human hepatocytes and result in liver fibrosis in mice.

Increases in p53 expression induce CTGF synthesis by mouse and human hepatocytes and result in liver fibrosis in mice.

http://www.wikidata.org/entity/Q39508688

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Role of Noncoding RNAs as Biomarker and Therapeutic Targets for Liver Fibrosis TGF-β signaling in tissue fibrosis: redox controls, target genes and therapeutic opportunities MicroRNAs in fibrosis: opportunities and challenges Involvement of inflammation and its related microRNAs in hepatocellular carcinoma Mcl-1 and Bcl-xL regulate Bak/Bax-dependent apoptosis of the megakaryocytic lineage at multistages TGF-β-induced hepatocyte lincRNA-p21 contributes to liver fibrosis in mice Contribution of p53 to metastasis.Tissue-specific and age-dependent effects of global Mdm2 loss Inhibition by curcumin of multiple sites of the transforming growth factor-beta1 signalling pathway ameliorates the progression of liver fibrosis induced by carbon tetrachloride in rats Circulating microRNAs as a Fingerprint for Liver Cirrhosis.Identification of therapeutic covariant microRNA clusters in hypoxia-treated cardiac progenitor cell exosomes using systems biology.Endothelial p53 deletion improves angiogenesis and prevents cardiac fibrosis and heart failure induced by pressure overload in mice.Connective-Tissue Growth Factor (CTGF/CCN2) Induces Astrogenesis and Fibronectin Expression of Embryonic Neural Cells In Vitro.miR-130b-3p Modulates Epithelial-Mesenchymal Crosstalk in Lung Fibrosis by Targeting IGF-1 Protection from liver fibrosis by a peroxisome proliferator-activated receptor δ agonist Role of MicroRNAs in Fibrosis.Role of Pin1 protein in the pathogenesis of nonalcoholic steatohepatitis in a rodent model MicroRNAs in Liver Health and Disease Activin A-Smad Signaling Mediates Connective Tissue Growth Factor Synthesis in Liver Progenitor Cells The LATS2 tumor suppressor inhibits SREBP and suppresses hepatic cholesterol accumulation Up-regulation of circulating miR-20a is correlated with hepatitis C virus-mediated liver disease progression.Rutin Attenuates Hepatotoxicity in High-Cholesterol-Diet-Fed Rats.Pathophysiological Significance of Hepatic Apoptosis Glycyrrhizic acid attenuates CCl₄-induced hepatocyte apoptosis in rats via a p53-mediated pathway The MDM2-p53-pyruvate carboxylase signalling axis couples mitochondrial metabolism to glucose-stimulated insulin secretion in pancreatic β-cells MicroRNA Expression Profiling in CCl₄-Induced Liver Fibrosis of Mus musculus.Processing of miR17-92 Cluster in Hepatic Stellate Cells Promotes Hepatic Fibrogenesis During Alcohol-Induced Injury.Transforming growth factor-β signaling in hepatocytes promotes hepatic fibrosis and carcinogenesis in mice with hepatocyte-specific deletion of TAK1.The Bcl-2 homology domain 3 (BH3)-only proteins Bim and bid are functionally active and restrained by anti-apoptotic Bcl-2 family proteins in healthy liver.p53 promotes inflammation-associated hepatocarcinogenesis by inducing HMGB1 release.DNA damage-induced sustained p53 activation contributes to inflammation-associated hepatocarcinogenesis in rats.Hepatic SATB1 induces paracrine activation of hepatic stellate cells and is upregulated by HBx.Is p53 Involved in Tissue-Specific Insulin Resistance Formation?p53 induces miR199a-3p to suppress SOCS7 for STAT3 activation and renal fibrosis in UUO.Mouse models of Mdm2 and Mdm4 and their clinical implications.MicroRNAs: their involvement in fibrosis pathogenesis and use as diagnostic biomarkers in scleroderma.Molecular mechanisms of oncogene-induced inflammation and inflammation-sustained oncogene activation in gastrointestinal tumors: an under-appreciated symbiotic relationship.Cellular and molecular actions of CCN2/CTGF and its role under physiological and pathological conditions.Mutant p53 in concert with an interleukin-27 receptor alpha deficiency causes spontaneous liver inflammation, fibrosis, and steatosis in mice miR-19a, -19b, and -26b Mediate CTGF Expression and Pulmonary Fibroblast Differentiation.

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P2860

Increases in p53 expression induce CTGF synthesis by mouse and human hepatocytes and result in liver fibrosis in mice.

http://www.wikidata.org/entity/Q39508688

description

2011 nî lūn-bûn

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2011年論文

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2011年论文

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Increases in p53 expression in ...... ult in liver fibrosis in mice.

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Increases in p53 expression in ...... ult in liver fibrosis in mice.

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Increases in p53 expression in ...... ult in liver fibrosis in mice.

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Increases in p53 expression in ...... ult in liver fibrosis in mice.

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Increases in p53 expression in ...... ult in liver fibrosis in mice.

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P1433

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Increases in p53 expression in ...... ult in liver fibrosis in mice.

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P2093

Akira Tomokuni

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Atsushi Hosui

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Hayato Hikita

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Hinako Tsunematsu

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Hiroaki Nagano

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Hisashi Ishida

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Masaharu Takigawa

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Minoru Shigekawa

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Naoki Hiramatsu

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Norio Hayashi

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P2860

Connective tissue growth factor (CTGF) acts as a downstream mediator of TGF-beta1 to induce mesenchymal cell condensation

Glutaminase 2, a novel p53 target gene regulating energy metabolism and antioxidant function

Repression of the miR-17-92 cluster by p53 has an important function in hypoxia-induced apoptosis

In vivo activation of the p53 pathway by small-molecule antagonists of MDM2

TIGAR, a p53-inducible regulator of glycolysis and apoptosis

Profibrogenic transforming growth factor-beta/activin receptor-like kinase 5 signaling via connective tissue growth factor expression in hepatocytes

Molecular evolution of a microRNA cluster

De-repression of CTGF via the miR-17-92 cluster upon differentiation of human glioblastoma spheroid cultures

miR-133 and miR-30 regulate connective tissue growth factor: implications for a role of microRNAs in myocardial matrix remodeling

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3343-3356

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8

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121

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Takahiro Kodama

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2011-07-11T00:00:00Z

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21747166

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3148730

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