H9c2 cardiac myoblasts undergo apoptosis in a model of ischemia consisting of serum deprivation and hypoxia: inhibition by PMA.
about
Molecular mechanism of monoamine oxidase A gene regulation under inflammation and ischemia-like conditions: key roles of the transcription factors GATA2, Sp1 and TBPCombinations of Kinase Inhibitors Protecting Myoblasts against HypoxiaMagnesium Lithospermate B Protects Cardiomyocytes from Ischemic Injury Via Inhibition of TAB1-p38 Apoptosis SignalingGlutaredoxin regulates apoptosis in cardiomyocytes via NFkappaB targets Bcl-2 and Bcl-xL: implications for cardiac agingDifferential profile of Nix upregulation and translocation during hypoxia/ischaemia in vivo versus in vitroOverexpression of Exosomal Cardioprotective miRNAs Mitigates Hypoxia-Induced H9c2 Cells Apoptosis.Serum depletion induces changes in protein expression in the trophoblast-derived cell line HTR-8/SVneo.Eukaryotic translation elongation factor 1A induces anoikis by triggering cell detachmentThe cardioprotective efficacy of TVP1022 in a rat model of ischaemia/reperfusion.H9c2 and HL-1 cells demonstrate distinct features of energy metabolism, mitochondrial function and sensitivity to hypoxia-reoxygenation.Lentiviral vector mediated modification of mesenchymal stem cells & enhanced survival in an in vitro model of ischaemia.Filamin C: a novel component of the KCNE2 interactome during hypoxia.Cardiac overexpression of metallothionein rescues cold exposure-induced myocardial contractile dysfunction through attenuation of cardiac fibrosis despite cardiomyocyte mechanical anomalies.Factors released from embryonic stem cells inhibit apoptosis in H9c2 cells through PI3K/Akt but not ERK pathway.Cathepsin A inhibition attenuates myocardial infarction-induced heart failure on the functional and proteomic levelsSurface-modified particles loaded with CaMKII inhibitor protect cardiac cells against mitochondrial injury.BH3-only protein Bim is upregulated and mediates the apoptosis of cardiomyocytes under glucose and oxygen-deprivation conditions.Low level tumor necrosis factor-alpha protects cardiomyocytes against high level tumor necrosis factor-alpha: brief insight into a beneficial paradox.Factors released from embryonic stem cells inhibit apoptosis of H9c2 cells.Role of NADPH oxidase in H9c2 cardiac muscle cells exposed to simulated ischaemia-reperfusion.Mitochondria transmit apoptosis signalling in cardiomyocyte-like cells and isolated hearts exposed to experimental ischemia-reperfusion injury.Effect of serum and oxygen concentration on gene expression and secretion of paracrine factors by mesenchymal stem cells.Herba Cistanche extract enhances mitochondrial glutathione status and respiration in rat hearts, with possible induction of uncoupling proteins.Vascular endothelial growth factor reduced hypoxia-induced death of human myoblasts and improved their engraftment in mouse muscles.
P2860
Q28259446-4D0E633F-6F60-45C5-8505-9269855F2068Q28547870-F0AAE0C5-0B66-48B6-8288-8373255B44B3Q28564909-01782466-1BF2-46EC-9A1E-B46A0AB986D2Q28568477-6EE7C21B-E4A0-4C00-8FB1-B869C860BACDQ28578192-3097DF94-7FBA-4E47-9AA8-3F23186720C9Q33624309-7B96A3ED-A1C5-4F76-900C-4874235F9989Q33634767-3BCE86B1-9DDD-41B8-94C0-AE342E1128EEQ34259550-E4E85195-695B-41E5-B154-B9370AE61D73Q35036171-6BE1ABC8-D529-4D4F-9580-A07935CD6346Q35299240-8DC3C6C7-9289-4C6F-AA37-8871A4153A95Q35579260-B53CAC6E-C07D-434B-A56F-905847CB4ACCQ35950069-F2037BAF-270C-44E6-A624-6003118E5BBBQ36084149-63B801CF-F094-4635-9262-95B3FA945BA9Q36844587-472F24B1-9326-48A2-9060-A2D261AFEC60Q36955513-8B6B12A8-9ACA-42DD-8870-6097B5758FD4Q38717638-FA84EDEF-86A9-4ACE-86F8-7896888040EFQ38947578-C2E1131C-7C62-4640-9995-7A6E077335FEQ38998367-496FEEAD-8B44-44D8-A05C-7C197C2227FBQ39633829-351CEBD2-DB83-4894-91A3-EB6E3192F80EQ39946205-B5A52ED8-1031-4974-8B26-BC0FF37D7050Q40108880-FD259DFF-B215-4EFF-8820-077A718D7A4FQ41537789-BF3549CA-BB11-464D-8C08-B6246A7B6B66Q42964477-1D754A96-B227-429B-8187-4F8BEFDB57A4Q45873666-DDEF5C8C-0E5B-4D0A-B45B-41324871B43F
P2860
H9c2 cardiac myoblasts undergo apoptosis in a model of ischemia consisting of serum deprivation and hypoxia: inhibition by PMA.
description
2003 nî lūn-bûn
@nan
2003年の論文
@ja
2003年論文
@yue
2003年論文
@zh-hant
2003年論文
@zh-hk
2003年論文
@zh-mo
2003年論文
@zh-tw
2003年论文
@wuu
2003年论文
@zh
2003年论文
@zh-cn
name
H9c2 cardiac myoblasts undergo ...... nd hypoxia: inhibition by PMA.
@en
type
label
H9c2 cardiac myoblasts undergo ...... nd hypoxia: inhibition by PMA.
@en
prefLabel
H9c2 cardiac myoblasts undergo ...... nd hypoxia: inhibition by PMA.
@en
P2093
P2860
P1433
P1476
H9c2 cardiac myoblasts undergo ...... and hypoxia: inhibition by PMA
@en
P2093
Annalisa Facchini
Carlo Guarnieri
Claudio Marcello Caldarera
Emanuele Giordano
Francesca Bonafè
Francesca Bonavita
P2860
P356
10.1016/S0014-5793(03)00029-2
P407
P577
2003-02-01T00:00:00Z