Attenuation of the slow component of delayed rectification, action potential prolongation, and triggered activity in mice expressing a dominant-negative Kv2 alpha subunit.
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Regulation of the Kv2.1 potassium channel by MinK and MiRP1Kv2 subunits underlie slowly inactivating potassium current in rat neocortical pyramidal neuronsKv5, Kv6, Kv8, and Kv9 subunits: No simple silent bystandersIon channel macromolecular complexes in cardiomyocytes: roles in sudden cardiac deathInhibition of HERG1 K(+) channels by the novel second-generation antihistamine mizolastineMarkov models of use-dependence and reverse use-dependence during the mouse cardiac action potentialThe Inhibitory Effects of Ca2+ Channel Blocker Nifedipine on Rat Kv2.1 Potassium ChannelsLocalization of Kv4.2 and KChIP2 in lipid rafts and modulation of outward K+ currents by membrane cholesterol content in rat left ventricular myocytesMouse models of long QT syndrome.MicroRNA-133a protects against myocardial fibrosis and modulates electrical repolarization without affecting hypertrophy in pressure-overloaded adult heartsMechanisms of Kv2.1 channel inhibition by celecoxib--modification of gating and channel blockMolecular basis of functional voltage-gated K+ channel diversity in the mammalian myocardium.Estrogen contributes to gender differences in mouse ventricular repolarizationMediation of neuronal apoptosis by Kv2.1-encoded potassium channels.Molecular identification and functional roles of a Ca(2+)-activated K+ channel in human and mouse hearts.Mice with cardiac overexpression of peroxisome proliferator-activated receptor γ have impaired repolarization and spontaneous fatal ventricular arrhythmiasEnhanced cardiac PI3Kα signalling mitigates arrhythmogenic electrical remodelling in pathological hypertrophy and heart failureIntracellular Ca2+ release underlies the development of phase 2 in mouse ventricular action potentials.Diverse phenotypes of outward currents in cells that have survived in the 5-day-infarcted heart.Exercise training and PI3Kα-induced electrical remodeling is independent of cellular hypertrophy and Akt signalingProlonged Action Potential and After depolarizations Are Not due to Changes in Potassium Currents in NOS3 Knockout Ventricular Myocytes.Cardiac expression of the CREM repressor isoform CREM-IbΔC-X in mice leads to arrhythmogenic alterations in ventricular cardiomyocytesA role for DPPX modulating external TEA sensitivity of Kv4 channels.Understanding ion channel biology using epitope tags: progress, pitfalls, and promise.PPARalpha-mediated remodeling of repolarizing voltage-gated K+ (Kv) channels in a mouse model of metabolic cardiomyopathy.Molecular Basis of Functional Myocardial Potassium Channel Diversity.SP6616 as a new Kv2.1 channel inhibitor efficiently promotes β-cell survival involving both PKC/Erk1/2 and CaM/PI3K/Akt signaling pathways.Reactive oxygen species-induced activation of p90 ribosomal S6 kinase prolongs cardiac repolarization through inhibiting outward K+ channel activity.Distinct cellular and molecular mechanisms underlie functional remodeling of repolarizing K+ currents with left ventricular hypertrophy.Deleting the accessory subunit KChIP2 results in loss of I(to,f) and increased I(K,slow) that maintains normal action potential configuration.Misinterpretation of the mouse ECG: 'musing the waves of Mus musculus'.Dissection of the voltage-activated potassium outward currents in adult mouse ventricular myocytes: I(to,f), I(to,s), I(K,slow1), I(K,slow2), and I(ss).Localization and mobility of the delayed-rectifer K+ channel Kv2.1 in adult cardiomyocytes.Overlapping binding sites of structurally different antiarrhythmics flecainide and propafenone in the subunit interface of potassium channel Kv2.1.Chronic atrial fibrillation does not further decrease outward currents. It increases them.Structural determinants of the regulation of the voltage-gated potassium channel Kv2.1 by the modulatory α-subunit Kv9.3.Molecular mechanisms of regulation of fast-inactivating voltage-dependent transient outward K+ current in mouse heart by cell volume changes.Electrophysiological characteristics of heart ventricular papillary muscles in diabetic histidine decarboxylase knockout and wild-type mice.Correlation of repolarization of ventricular monophasic action potential with ECG in the murine heart.Effect of androgen deficiency on mouse ventricular repolarization.
P2860
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P2860
Attenuation of the slow component of delayed rectification, action potential prolongation, and triggered activity in mice expressing a dominant-negative Kv2 alpha subunit.
description
1999 nî lūn-bûn
@nan
1999年の論文
@ja
1999年論文
@yue
1999年論文
@zh-hant
1999年論文
@zh-hk
1999年論文
@zh-mo
1999年論文
@zh-tw
1999年论文
@wuu
1999年论文
@zh
1999年论文
@zh-cn
name
Attenuation of the slow compon ...... nt-negative Kv2 alpha subunit.
@en
type
label
Attenuation of the slow compon ...... nt-negative Kv2 alpha subunit.
@en
prefLabel
Attenuation of the slow compon ...... nt-negative Kv2 alpha subunit.
@en
P2093
P356
P1433
P1476
Attenuation of the slow compon ...... nt-negative Kv2 alpha subunit.
@en
P2093
P304
P356
10.1161/01.RES.85.7.623
P577
1999-10-01T00:00:00Z