Control of sister chromatid recombination by histone H2AX. - Wikidata - awgldk - TriplyDB

Control of sister chromatid recombination by histone H2AX.

Control of sister chromatid recombination by histone H2AX.

http://www.wikidata.org/entity/Q41549984

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Human SMC5/6 complex promotes sister chromatid homologous recombination by recruiting the SMC1/3 cohesin complex to double-strand breaks Rad17 recruits the MRE11-RAD50-NBS1 complex to regulate the cellular response to DNA double-strand breaks SIRT1 redistribution on chromatin promotes genomic stability but alters gene expression during aging Dynamics of DNA damage response proteins at DNA breaks: a focus on protein modifications MicroRNA-138 modulates DNA damage response by repressing histone H2AX expression DNA damage-dependent acetylation and ubiquitination of H2AX enhances chromatin dynamics Targeting ATM-deficient CLL through interference with DNA repair pathways Impact of histone H4 lysine 20 methylation on 53BP1 responses to chromosomal double strand breaks Structural Insight into p53 Recognition by the 53BP1 Tandem Tudor Domain Genetic analysis of Saccharomyces cerevisiae H2A serine 129 mutant suggests a functional relationship between H2A and the sister-chromatid cohesion partners Csm3-Tof1 for the repair of topoisomerase I-induced DNA damage.Minding the gap: the underground functions of BRCA1 and BRCA2 at stalled replication forks γ-H2AX kinetics as a novel approach to high content screening for small molecule radiosensitizers Histone H2AX and Fanconi anemia FANCD2 function in the same pathway to maintain chromosome stability RECQL5 and BLM exhibit divergent functions in cells defective for the Fanconi anemia pathway Double strand break repair functions of histone H2AX Role of mammalian Mre11 in classical and alternative nonhomologous end joining GammaH2AX and cancer PhiC31 integrase induces a DNA damage response and chromosomal rearrangements in human adult fibroblasts.Residual gammaH2AX foci as an indication of lethal DNA lesions.Enhanced H2AX phosphorylation, DNA replication fork arrest, and cell death in the absence of Chk1.Differential requirement for H2AX and 53BP1 in organismal development and genome maintenance in the absence of poly(ADP)ribosyl polymerase 1.MRE11-RAD50-NBS1 complex dictates DNA repair independent of H2AX.Histone H2AX stabilizes broken DNA strands to suppress chromosome breaks and translocations during V(D)J recombination Bisbenzamidine derivative, pentamidine represses DNA damage response through inhibition of histone H2A acetylation.An RNF168 fragment defective for focal accumulation at DNA damage is proficient for inhibition of homologous recombination in BRCA1 deficient cells.Cell cycle-dependent induction of homologous recombination by a tightly regulated I-SceI fusion protein.BRCA1 controls homologous recombination at Tus/Ter-stalled mammalian replication forks Senescence evasion by MCF-7 human breast tumor-initiating cells.DNA-PK phosphorylation of RPA32 Ser4/Ser8 regulates replication stress checkpoint activation, fork restart, homologous recombination and mitotic catastrophe.KAP-1 promotes resection of broken DNA ends not protected by γ-H2AX and 53BP1 in G₁-phase lymphocytes.Deletion of BRCA2 exon 27 causes defects in response to both stalled and collapsed replication forks H2AX Phosphorylation: Its Role in DNA Damage Response and Cancer Therapy.Histone gammaH2AX and poly(ADP-ribose) as clinical pharmacodynamic biomarkers.H2AX post-translational modifications in the ionizing radiation response and homologous recombination.Mechanisms of double-strand break repair in somatic mammalian cells.Microhomology-mediated End Joining and Homologous Recombination share the initial end resection step to repair DNA double-strand breaks in mammalian cells MDC1 interacts with Rad51 and facilitates homologous recombination.Replication and homologous recombination repair regulate DNA double-strand break formation by the antitumor alkylator ecteinascidin 743.PARP1-driven poly-ADP-ribosylation regulates BRCA1 function in homologous recombination-mediated DNA repair.Defining a genotoxic profile with mouse embryonic stem cells

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P2860

Control of sister chromatid recombination by histone H2AX.

http://www.wikidata.org/entity/Q41549984

description

2004 nî lūn-bûn

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2004年の論文

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2004年学术文章

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2004年学术文章

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2004年學術文章

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2004年學術文章

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2004年學術文章

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name

Control of sister chromatid recombination by histone H2AX.

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Control of sister chromatid recombination by histone H2AX.

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Control of sister chromatid recombination by histone H2AX.

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Control of sister chromatid recombination by histone H2AX.

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Anyong Xie

http://www.w3.org/2001/XMLSchema#string

Craig H Bassing

http://www.w3.org/2001/XMLSchema#string

Frederick W Alt

http://www.w3.org/2001/XMLSchema#string

Inbo Shim

http://www.w3.org/2001/XMLSchema#string

Ingeborga Jarzyna

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Nadine Puget

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Ralph Scully

http://www.w3.org/2001/XMLSchema#string

Shobu Odate

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P2860

Crystal structure of the nucleosome core particle at 2.8 A resolution

MDC1 is a mediator of the mammalian DNA damage checkpoint

Sister chromatids are preferred over homologs as substrates for recombinational repair in Saccharomyces cerevisiae

Chk2 phosphorylation of BRCA1 regulates DNA double-strand break repair

Involvement of the cohesin protein, Smc1, in Atm-dependent and independent responses to DNA damage

Megabase chromatin domains involved in DNA double-strand breaks in vivo

Cre reporter strains produced by targeted insertion of EYFP and ECFP into the ROSA26 locus

Genetic requirements for the single-strand annealing pathway of double-strand break repair in Saccharomyces cerevisiae

A role for Saccharomyces cerevisiae histone H2A in DNA repair.

Postreplicative recruitment of cohesin to double-strand breaks is required for DNA repair.

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15610743

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2991138

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