Rapid loss of microvascular integrin expression during focal brain ischemia reflects neuron injury.
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Integrins in the optic nerve head: potential roles in glaucomatous optic neuropathy (an American Ophthalmological Society thesis)Aging and the neurovascular unitMagnetic resonance imaging of ischemia viability thresholds and the neurovascular unit.A novel three-dimensional system to study interactions between endothelial cells and neural cells of the developing central nervous system.An emerging role of mast cells in cerebral ischemia and hemorrhage.Severe blood-brain barrier disruption and surrounding tissue injuryHemostasis and alterations of the central nervous systemPlasmin-dependent modulation of the blood-brain barrier: a major consideration during tPA-induced thrombolysis?Relationship of neurovascular elements to neuron injury during ischemia.Focal cerebral ischemia induces active proteases that degrade microvascular matrix.The integrin family of cell adhesion molecules has multiple functions within the CNS.Interendothelial claudin-5 expression depends on cerebral endothelial cell-matrix adhesion by β(1)-integrinsExtracellular proteolytic pathophysiology in the neurovascular unit after stroke.The neurovascular unit and combination treatment strategies for strokeDabigatran abrogates brain endothelial cell permeability in response to thrombin.Extracellular matrix and matrix receptors in blood-brain barrier formation and stroke.Cellular mechanisms of neurovascular damage and repair after strokeDisruption of dystroglycan-laminin interactions modulates water uptake by astrocytesLaminins containing the β2 and γ3 chains regulate astrocyte migration and angiogenesis in the retina.Tissue-type plasminogen activator and the low-density lipoprotein receptor-related protein induce Akt phosphorylation in the ischemic brainThe rapid decrease in astrocyte-associated dystroglycan expression by focal cerebral ischemia is protease-dependent.Responses of endothelial cell and astrocyte matrix-integrin receptors to ischemia mimic those observed in the neurovascular unitUpregulation of fibronectin and the α5β1 and αvβ3 integrins on blood vessels within the cerebral ischemic penumbra.Regulation of cerebral vasculature in normal and ischemic brainVirchow's triad: the vascular basis of cerebral injuryLipopolysaccharide-induced blood brain barrier permeability is enhanced by alpha-synuclein expressionUltrastructural and temporal changes of the microvascular basement membrane and astrocyte interface following focal cerebral ischemia.Inflammation and the neurovascular unit in the setting of focal cerebral ischemia.Integrin-mediated regulation of neurovascular development, physiology and disease.Implications of vascular endothelial growth factor for postischemic neurovascular remodeling.The neurovascular unit in the setting of stroke.Heterogeneity in the penumbra.The cell-matrix interface: a possible target for treating retinal vascular related pathologies.Glial influence on the blood brain barrier.Novel insights into the development and maintenance of the blood-brain barrier.Brain endothelial cell specific integrins and ischemic stroke.The central role of aquaporins in the pathophysiology of ischemic stroke.Bleeding in the brain: Amyloid-beta may keep clots awayActivation systems for latent matrix metalloproteinase-2 are upregulated immediately after focal cerebral ischemia.Increased expression of the beta4 and alpha5 integrin subunits in cerebral blood vessels of transgenic mice chronically producing the pro-inflammatory cytokines IL-6 or IFN-alpha in the central nervous system.
P2860
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P2860
Rapid loss of microvascular integrin expression during focal brain ischemia reflects neuron injury.
description
2001 nî lūn-bûn
@nan
2001年の論文
@ja
2001年学术文章
@wuu
2001年学术文章
@zh-cn
2001年学术文章
@zh-hans
2001年学术文章
@zh-my
2001年学术文章
@zh-sg
2001年學術文章
@yue
2001年學術文章
@zh
2001年學術文章
@zh-hant
name
Rapid loss of microvascular in ...... chemia reflects neuron injury.
@en
Rapid loss of microvascular in ...... chemia reflects neuron injury.
@nl
type
label
Rapid loss of microvascular in ...... chemia reflects neuron injury.
@en
Rapid loss of microvascular in ...... chemia reflects neuron injury.
@nl
prefLabel
Rapid loss of microvascular in ...... chemia reflects neuron injury.
@en
Rapid loss of microvascular in ...... chemia reflects neuron injury.
@nl
P2093
P2860
P1476
Rapid loss of microvascular in ...... chemia reflects neuron injury.
@en
P2093
Copeland BR
Seiffert D
del Zoppo GJ
P2860
P304
P356
10.1097/00004647-200107000-00009
P577
2001-07-01T00:00:00Z
P5875
P6179
1014345220