Modulation of excitation-contraction coupling by isoproterenol in cardiomyocytes with controlled SR Ca2+ load and Ca2+ current trigger.
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Kinetics of FKBP12.6 binding to ryanodine receptors in permeabilized cardiac myocytes and effects on Ca sparksCalcium movements inside the sarcoplasmic reticulum of cardiac myocytesIon Channels in the HeartAbnormal Ca(2+) cycling in failing ventricular myocytes: role of NOS1-mediated nitroso-redox balancePhosphodiesterase 4B in the cardiac L-type Ca²⁺ channel complex regulates Ca²⁺ current and protects against ventricular arrhythmias in miceHeart failure with preserved ejection fraction.Phosphorylation of the ryanodine receptor mediates the cardiac fight or flight response in mice.Functional antagonism of β-adrenoceptor subtypes in the catecholamine-induced automatism in rat myocardium.Basal and β-adrenergic regulation of the cardiac calcium channel CaV1.2 requires phosphorylation of serine 1700Using models of the myocyte for functional interpretation of cardiac proteomic data.CaMKII regulation of cardiac ryanodine receptors and inositol triphosphate receptors.Role of RyR2 phosphorylation in heart failure and arrhythmias: protein kinase A-mediated hyperphosphorylation of the ryanodine receptor at serine 2808 does not alter cardiac contractility or cause heart failure and arrhythmiasSarcoplasmic reticulum Ca2+ pumping kinetics regulates timing of local Ca2+ releases and spontaneous beating rate of rabbit sinoatrial node pacemaker cells.Transgenic rabbit model for human troponin I-based hypertrophic cardiomyopathy.Ryanodine receptor/calcium release channel PKA phosphorylation: a critical mediator of heart failure progression.Variability in couplon size in rabbit ventricular myocytes.Regulation of cardiac sarcoplasmic reticulum Ca release by luminal [Ca] and altered gating assessed with a mathematical model.The ryanodine receptor in cardiac physiology and disease.Balanced changes in Ca buffering by SERCA and troponin contribute to Ca handling during β-adrenergic stimulation in cardiac myocytes2APB- and JTV519(K201)-sensitive micro Ca2+ waves in arrhythmogenic Purkinje cells that survive in infarcted canine heart.Perturbed atrial calcium handling in an ovine model of heart failure: potential roles for reductions in the L-type calcium current.Effects of increased systolic Ca²⁺ and phospholamban phosphorylation during β-adrenergic stimulation on Ca²⁺ transient kinetics in cardiac myocytes.CaMKIIδC slows [Ca]i decline in cardiac myocytes by promoting Ca sparks.Ryanodine receptor phosphorylation and heart failure: phasing out S2808 and "criminalizing" S2814.Ca2+/calmodulin kinase II increases ryanodine binding and Ca2+-induced sarcoplasmic reticulum Ca2+ release kinetics during beta-adrenergic stimulation.Phosphodiesterase type 3A regulates basal myocardial contractility through interacting with sarcoplasmic reticulum calcium ATPase type 2a signaling complexes in mouse heartAtrial remodelling in atrial fibrillation: CaMKII as a nodal proarrhythmic signalCatestatin (chromogranin A344-364) is a novel cardiosuppressive agent: inhibition of isoproterenol and endothelin signaling in the frog heart.Sex differences in mechanisms of cardiac excitation-contraction couplingPKA phosphorylation of cardiac ryanodine receptor modulates SR luminal Ca2+ sensitivity.The antihypertensive chromogranin a peptide catestatin acts as a novel endocrine/paracrine modulator of cardiac inotropism and lusitropismInteractions between sarco-endoplasmic reticulum and mitochondria in cardiac and skeletal muscle - pivotal roles in Ca²⁺ and reactive oxygen species signaling.Adrenergic regulation of cardiac contractility does not involve phosphorylation of the cardiac ryanodine receptor at serine 2808Ryanodine receptor-mediated arrhythmias and sudden cardiac death.Phosphorylation sites required for regulation of cardiac calcium channels in the fight-or-flight response.AKAP-scaffolding proteins and regulation of cardiac physiology.Beta-adrenergic signaling accelerates and synchronizes cardiac ryanodine receptor response to a single L-type Ca2+ channelNa/K-ATPase--an integral player in the adrenergic fight-or-flight response.Targeted ablation of the histidine-rich Ca(2+)-binding protein (HRC) gene is associated with abnormal SR Ca(2+)-cycling and severe pathology under pressure-overload stressInherited calcium channelopathies in the pathophysiology of arrhythmias.
P2860
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P2860
Modulation of excitation-contraction coupling by isoproterenol in cardiomyocytes with controlled SR Ca2+ load and Ca2+ current trigger.
description
2004 nî lūn-bûn
@nan
2004年の論文
@ja
2004年学术文章
@wuu
2004年学术文章
@zh-cn
2004年学术文章
@zh-hans
2004年学术文章
@zh-my
2004年学术文章
@zh-sg
2004年學術文章
@yue
2004年學術文章
@zh
2004年學術文章
@zh-hant
name
Modulation of excitation-contr ...... load and Ca2+ current trigger.
@en
Modulation of excitation-contr ...... load and Ca2+ current trigger.
@nl
type
label
Modulation of excitation-contr ...... load and Ca2+ current trigger.
@en
Modulation of excitation-contr ...... load and Ca2+ current trigger.
@nl
prefLabel
Modulation of excitation-contr ...... load and Ca2+ current trigger.
@en
Modulation of excitation-contr ...... load and Ca2+ current trigger.
@nl
P2860
P1476
Modulation of excitation-contr ...... load and Ca2+ current trigger.
@en
P2093
Kenneth S Ginsburg
P2860
P304
P356
10.1113/JPHYSIOL.2003.055384
P407
P577
2004-01-14T00:00:00Z