Transgenic rabbit model for human troponin I-based hypertrophic cardiomyopathy.
about
Animal and in silico models for the study of sarcomeric cardiomyopathiesSmall and large animal models in cardiac contraction research: advantages and disadvantagesDiastolic dysfunction and thin filament dysregulation resulting from excitation-contraction uncoupling in a mouse model of restrictive cardiomyopathy.Protective effect of geranylgeranylacetone via enhancement of HSPB8 induction in desmin-related cardiomyopathy.Ultrastructural changes, increased oxidative stress, inflammation, and altered cardiac hypertrophic gene expressions in heart tissues of rats exposed to incense smoke.In Vivo Analysis of Troponin C Knock-In (A8V) Mice: Evidence that TNNC1 Is a Hypertrophic Cardiomyopathy Susceptibility GeneLost in transgenesis: a user's guide for genetically manipulating the mouse in cardiac research.Role of the acidic N' region of cardiac troponin I in regulating myocardial function.Phospholamban overexpression in transgenic rabbitsA quantitative comparison of regional myocardial motion in mice, rabbits and humans using in-vivo phase contrast CMR.Enhanced transmural fiber rotation and connexin 43 heterogeneity are associated with an increased upper limit of vulnerability in a transgenic rabbit model of human hypertrophic cardiomyopathy.Allele and species dependent contractile defects by restrictive and hypertrophic cardiomyopathy-linked troponin I mutantsFunctional consequences of the human cardiac troponin I hypertrophic cardiomyopathy mutation R145G in transgenic miceDesigning heart performance by gene transfer.Impaired relaxation is the main manifestation in transgenic mice expressing a restrictive cardiomyopathy mutation, R193H, in cardiac TnI.Increased myofilament Ca2+-sensitivity and arrhythmia susceptibility.On the emerging role of rabbit as human disease model and the instrumental role of novel transgenic tools.In vivo models of cardiac diseases: application to drug development and screening.Inherited cardiomyopathies caused by troponin mutations.Passive ventricular remodeling in cardiac disease: focus on heterogeneity.A One Health Approach to Hypertrophic Cardiomyopathy.On mice, rabbits, and human heart failure.MR tagging demonstrates quantitative differences in regional ventricular wall motion in mice, rats, and men.Proliferative Potential of Cardiomyocytes in Hypertrophic Cardiomyopathy: Correlation with Myocardial Remodeling.
P2860
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P2860
Transgenic rabbit model for human troponin I-based hypertrophic cardiomyopathy.
description
2005 nî lūn-bûn
@nan
2005 թուականի Մայիսին հրատարակուած գիտական յօդուած
@hyw
2005 թվականի մայիսին հրատարակված գիտական հոդված
@hy
2005年の論文
@ja
2005年論文
@yue
2005年論文
@zh-hant
2005年論文
@zh-hk
2005年論文
@zh-mo
2005年論文
@zh-tw
2005年论文
@wuu
name
Transgenic rabbit model for human troponin I-based hypertrophic cardiomyopathy.
@ast
Transgenic rabbit model for human troponin I-based hypertrophic cardiomyopathy.
@en
Transgenic rabbit model for human troponin I-based hypertrophic cardiomyopathy.
@nl
type
label
Transgenic rabbit model for human troponin I-based hypertrophic cardiomyopathy.
@ast
Transgenic rabbit model for human troponin I-based hypertrophic cardiomyopathy.
@en
Transgenic rabbit model for human troponin I-based hypertrophic cardiomyopathy.
@nl
prefLabel
Transgenic rabbit model for human troponin I-based hypertrophic cardiomyopathy.
@ast
Transgenic rabbit model for human troponin I-based hypertrophic cardiomyopathy.
@en
Transgenic rabbit model for human troponin I-based hypertrophic cardiomyopathy.
@nl
P2093
P2860
P1433
P1476
Transgenic rabbit model for human troponin I-based hypertrophic cardiomyopathy.
@en
P2093
Atsushi Sanbe
Bruce Zinman
Donald M Bers
Hanna Osinska
James Gulick
Jeanne James
Jeffrey Robbins
Kenneth S Ginsburg
Lisa Martin
Raisa Klevitsky
P2860
P304
P356
10.1161/01.CIR.0000164234.24957.75
P407
P577
2005-05-02T00:00:00Z