Genotoxic anti-cancer agents and their relationship to DNA damage, mitosis, and checkpoint adaptation in proliferating cancer cells
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Cynaropicrin: A Comprehensive Research Review and Therapeutic Potential As an Anti-Hepatitis C Virus AgentThe homologous recombination protein RAD51D protects the genome from large deletionsSpecific recognition of guanines in non-duplex regions of nucleic acids with potassium tungstate and hydrogen peroxideRequirement of ATR for maintenance of intestinal stem cells in aging DrosophilaAssessment of cytotoxicity and genotoxicity of stem bark extracts from Canarium odontophyllum Miq. (dabai) against HCT 116 human colorectal cancer cell line.Assessment of the proliferation status of glioblastoma cell and tumour tissue after nanoplatinum treatment.Isoeugenol is a selective potentiator of camptothecin cytotoxicity in vertebrate cells lacking TDP1The Cytotoxicity of Dacarbazine Potentiated by Sea Cucumber Saponin in Resistant B16F10 Melanoma Cells through Apoptosis Induction.Highly activated p53 contributes to selectively increased apoptosis of latently HIV-1 infected cells upon treatment of anticancer drugs.When genome integrity and cell cycle decisions collide: roles of polo kinases in cellular adaptation to DNA damage.Effects of hyperthermia on DNA repair pathways: one treatment to inhibit them all.Identification of Small Molecule Translesion Synthesis Inhibitors That Target the Rev1-CT/RIR Protein-Protein Interaction.Cancer cells that survive checkpoint adaptation contain micronuclei that harbor damaged DNA.NONO regulates the intra-S-phase checkpoint in response to UV radiation.Noncoding RNAs in Therapeutic Resistance of Cancer.Caenorhabditis elegans as a powerful alternative model organism to promote research in genetic toxicology and biomedicine.Reduced kinase activity of polo kinase Cdc5 affects chromosome stability and DNA damage response in S. cerevisiae.Cytotoxic amounts of cisplatin induce either checkpoint adaptation or apoptosis in a concentration-dependent manner in cancer cells.G2/M-Phase Checkpoint Adaptation and Micronuclei Formation as Mechanisms That Contribute to Genomic Instability in Human Cells.Dihydrocoumarin, an HDAC Inhibitor, Increases DNA Damage Sensitivity by Inhibiting Rad52.Targeted inhibition of Hedgehog-GLI signaling by novel acylguanidine derivatives inhibits melanoma cell growth by inducing replication stress and mitotic catastrophe.Correlations between Risk Factors for Breast Cancer and Genetic Instability in Cancer Patients—A Clinical Perspective Study.Camptothecin induces G2/M phase arrest through the ATM-Chk2-Cdc25C axis as a result of autophagy-induced cytoprotection: Implications of reactive oxygen species.Bleomycin-induced genome structural variations in normal, non-tumor cells
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P2860
Genotoxic anti-cancer agents and their relationship to DNA damage, mitosis, and checkpoint adaptation in proliferating cancer cells
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2014 nî lūn-bûn
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2014 թուականի Փետրուարին հրատարակուած գիտական յօդուած
@hyw
2014 թվականի փետրվարին հրատարակված գիտական հոդված
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2014年の論文
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2014年論文
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2014年論文
@zh-hant
2014年論文
@zh-hk
2014年論文
@zh-mo
2014年論文
@zh-tw
2014年论文
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name
Genotoxic anti-cancer agents a ...... in proliferating cancer cells
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Genotoxic anti-cancer agents a ...... in proliferating cancer cells
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Genotoxic anti-cancer agents a ...... in proliferating cancer cells
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type
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Genotoxic anti-cancer agents a ...... in proliferating cancer cells
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Genotoxic anti-cancer agents a ...... in proliferating cancer cells
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Genotoxic anti-cancer agents a ...... in proliferating cancer cells
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prefLabel
Genotoxic anti-cancer agents a ...... in proliferating cancer cells
@ast
Genotoxic anti-cancer agents a ...... in proliferating cancer cells
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Genotoxic anti-cancer agents a ...... in proliferating cancer cells
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P2860
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P356
P1476
Genotoxic anti-cancer agents a ...... in proliferating cancer cells
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P2093
Lucy Swift
Roy Golsteyn
P2860
P304
P3181
P356
10.3390/IJMS15033403
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P5008
P577
2014-02-25T00:00:00Z