about
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Targeting lung cancer through inhibition of checkpoint kinases.DNA damage-induced ATM- and Rad-3-related (ATR) kinase activation in non-replicating cells is regulated by the XPB subunit of transcription factor IIH (TFIIH).ATR Kinase Inhibition Protects Non-cycling Cells from the Lethal Effects of DNA Damage and Transcription Stress.ATR inhibitors as a synthetic lethal therapy for tumours deficient in ARID1A.ATR inhibition rewires cellular signaling networks induced by replication stress.DDRprot: a database of DNA damage response-related proteins.ATR Is a Therapeutic Target in Synovial Sarcoma.Local DNA Repair Inhibition for Sustained Radiosensitization of High-Grade Gliomas.
P2860
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P2860
description
2014 թուականի Մայիսին հրատարակուած գիտական յօդուած
@hyw
2014 թվականի մայիսին հրատարակված գիտական հոդված
@hy
artículu científicu espublizáu en 2014
@ast
scientific article (publication date: 9 May 2014)
@en
vedecký článok (publikovaný 2014/05/09)
@sk
vědecký článek publikovaný v roce 2014
@cs
wetenschappelijk artikel (gepubliceerd op 2014/05/09)
@nl
مقالة علمية (نشرت عام 2014)
@ar
name
Chemical strategies for development of ATR inhibitors
@ast
Chemical strategies for development of ATR inhibitors
@en
Chemical strategies for development of ATR inhibitors
@nl
type
label
Chemical strategies for development of ATR inhibitors
@ast
Chemical strategies for development of ATR inhibitors
@en
Chemical strategies for development of ATR inhibitors
@nl
prefLabel
Chemical strategies for development of ATR inhibitors
@ast
Chemical strategies for development of ATR inhibitors
@en
Chemical strategies for development of ATR inhibitors
@nl
P2093
P2860
P3181
P356
P1476
Chemical strategies for development of ATR inhibitors
@en
P2093
Andreas Höglund
Sabin Llona-Minguez
Sylvain A Jacques
Sylvain A. Jacques
Tobias Koolmeister
P2860
P3181
P356
10.1017/ERM.2014.10
P407
P577
2014-01-01T00:00:00Z
2014-05-09T00:00:00Z