Genetic background changes the pattern of forebrain commissure defects in transgenic mice underexpressing the beta-amyloid-precursor protein - Test2 - elhamkhani - TriplyDB

Genetic background changes the pattern of forebrain commissure defects in transgenic mice underexpressing the beta-amyloid-precursor protein

Genetic background changes the pattern of forebrain commissure defects in transgenic mice underexpressing the beta-amyloid-precursor protein

http://www.wikidata.org/entity/Q28587017

about

The Alzheimer's disease-associated amyloid beta-protein is an antimicrobial peptide Disruption of the murine nuclear factor I-A gene (Nfia) results in perinatal lethality, hydrocephalus, and agenesis of the corpus callosum Insights into the physiological function of the β-amyloid precursor protein: beyond Alzheimer's disease Hippocampal network oscillations in APP/APLP2-deficient mice The K-Cl cotransporter KCC3 is mutant in a severe peripheral neuropathy associated with agenesis of the corpus callosum Cortical dysplasia resembling human type 2 lissencephaly in mice lacking all three APP family members Essential roles for the FE65 amyloid precursor protein-interacting proteins in brain development Truncating mutations in APP cause a distinct neurological phenotype.Differences in memory development among C57BL/6NCrl, 129S2/SvPasCrl, and FVB/NCrl mice after delay and trace fear conditioning.COL25A1 triggers and promotes Alzheimer's disease-like pathology in vivo.Alzheimer disease models and human neuropathology: similarities and differences Neuronal untranslated BC1 RNA: targeted gene elimination in mice.Comparative analysis of single and combined APP/APLP knockouts reveals reduced spine density in APP-KO mice that is prevented by APPsα expression.Comparative transcriptome profiling of amyloid precursor protein family members in the adult cortex Function, therapeutic potential and cell biology of BACE proteases: current status and future prospects.Dentate gyrus volume is reduced before onset of plaque formation in PDAPP mice: a magnetic resonance microscopy and stereologic analysis.Daily scheduled high fat meals moderately entrain behavioral anticipatory activity, body temperature, and hypothalamic c-Fos activation.The intracellular threonine of amyloid precursor protein that is essential for docking of Pin1 is dispensable for developmental function.Finding the most appropriate mouse model of juvenile CLN3 (Batten) disease for therapeutic studies: the importance of genetic background and gender.Deficits in axonal transport in hippocampal-based circuitry and the visual pathway in APP knock-out animals witnessed by manganese enhanced MRI.Agenesis of the Corpus Callosum Due to Defective Glial Wedge Formation in Lhx2 Mutant Mice.All in the Family: How the APPs Regulate Neurogenesis Amyloid precursor protein regulates netrin-1-mediated commissural axon outgrowth The protection of acetylcholinesterase inhibitor on β-amyloid-induced the injury of neurite outgrowth via regulating axon guidance related genes expression in neuronal cells.Hippocampal commissure defects in crosses of four inbred mouse strains with absent corpus callosum.Loss of presenilin function is associated with a selective gain of APP function.Amyloid precursor protein maintains constitutive and adaptive plasticity of dendritic spines in adult brain by regulating D-serine homeostasis.Understanding the molecular basis of Alzheimer's disease using a Caenorhabditis elegans model system.Amyloid-modifying therapies for Alzheimer's disease: therapeutic progress and its implications.Biology and pathophysiology of the amyloid precursor protein.Functions of the APP gene family in the nervous system: insights from mouse models.The gene or not the gene--that is the question: understanding the genetically engineered mouse phenotype.Physiological functions of APP family proteins.Similar reliability and equivalent performance of female and male mice in the open field and water-maze place navigation task Expression of endogenous mouse APP modulates β-amyloid deposition in hAPP-transgenic mice The Role of APP in Structural Spine Plasticity.Not just amyloid: physiological functions of the amyloid precursor protein family.GABAergic hyperinnervation of dentate granule cells in the Ts65Dn mouse model of down syndrome: Exploring the role of App.The physiological role of the amyloid precursor protein as an adhesion molecule in the developing nervous system.Transcriptomic profile analysis of mouse neural tube development by RNA-Seq.

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Genetic background changes the pattern of forebrain commissure defects in transgenic mice underexpressing the beta-amyloid-precursor protein

http://www.wikidata.org/entity/Q28587017

description

1999 թուականի Ապրիլին հրատարակուած գիտական յօդուած

@hyw

1999 թվականի ապրիլին հրատարակված գիտական հոդված

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article publié dans les Procee ...... f the United States of America

@fr

artículu científicu espublizáu en 1999

@ast

im April 1999 veröffentlichter wissenschaftlicher Artikel

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scientific journal article

@en

vedecký článok (publikovaný 1999/04/13)

@sk

vědecký článek publikovaný v roce 1999

@cs

wetenschappelijk artikel (gepubliceerd op 1999/04/13)

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наукова стаття, опублікована у квітні 1999

@uk

name

Genetic background changes the ...... beta-amyloid-precursor protein

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Genetic background changes the ...... beta-amyloid-precursor protein

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Genetic background changes the ...... beta-amyloid-precursor protein

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Genetic background changes the ...... beta-amyloid-precursor protein

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Genetic background changes the ...... beta-amyloid-precursor protein

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Genetic background changes the ...... beta-amyloid-precursor protein

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Proceedings of the National Academy of Sciences of the United States of America

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Genetic background changes the ...... beta-amyloid-precursor protein

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C. Weissmann

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D. P. Wolfer

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F. Magara

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M. Stagljar

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P2860

Phenotype of mice lacking functional Deleted in colorectal cancer (Dcc) gene

Neurobehavioral development, adult openfield exploration and swimming navigation learning in mice with a modified beta-amyloid precursor protein gene

Behavioral and anatomical deficits in mice homozygous for a modified beta-amyloid precursor protein gene

Sek4 and Nuk receptors cooperate in guidance of commissural axons and in palate formation

beta-Amyloid precursor protein-deficient mice show reactive gliosis and decreased locomotor activity

Netrin-1 is required for commissural axon guidance in the developing vertebrate nervous system

A rapid, sensitive histochemical stain for myelin in frozen brain sections.

Gene-targeting studies of mammalian behavior: is it the mutation or the background genotype?

MARCKS deficiency in mice leads to abnormal brain development and perinatal death.

Impaired fetal T cell development and perinatal lethality in mice lacking the cAMP response element binding protein

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10200318

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