Defining specificity and on-target activity of BH3-mimetics using engineered B-ALL cell lines.
about
Epistatic mutations in PUMA BH3 drive an alternate binding mode to potently and selectively inhibit anti-apoptotic Bfl-1Development of venetoclax for therapy of lymphoid malignancies.BCL2 Inhibitors as Anticancer Drugs: A Plethora of Misleading BH3 Mimetics.Iterative optimization yields Mcl-1-targeting stapled peptides with selective cytotoxicity to Mcl-1-dependent cancer cells.Modulation of Navitoclax Sensitivity by Dihydroartemisinin-Mediated MCL-1 Repression in BCR-ABL+ B-Lineage Acute Lymphoblastic Leukemia.Pathways and mechanisms of venetoclax resistance.Discovery and biological characterization of potent myeloid cell leukemia-1 inhibitors.Discovery of Mcl-1 inhibitors from integrated high throughput and virtual screening
P2860
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P2860
Defining specificity and on-target activity of BH3-mimetics using engineered B-ALL cell lines.
description
2016 nî lūn-bûn
@nan
2016年の論文
@ja
2016年論文
@yue
2016年論文
@zh-hant
2016年論文
@zh-hk
2016年論文
@zh-mo
2016年論文
@zh-tw
2016年论文
@wuu
2016年论文
@zh
2016年论文
@zh-cn
name
Defining specificity and on-ta ...... g engineered B-ALL cell lines.
@en
type
label
Defining specificity and on-ta ...... g engineered B-ALL cell lines.
@en
prefLabel
Defining specificity and on-ta ...... g engineered B-ALL cell lines.
@en
P2093
P2860
P356
P1433
P1476
Defining specificity and on-ta ...... g engineered B-ALL cell lines.
@en
P2093
Anthony Letai
Brian Koss
Joseph T Opferman
Katherine Szarama
Madhavi Bathina
Michael H Cardone
Zaneta Nikolovska-Coleska
P2860
P304
11500-11511
P356
10.18632/ONCOTARGET.7204
P407
P577
2016-02-05T00:00:00Z