about
Bin2, a functionally nonredundant member of the BAR adaptor gene familyPTIP associates with MLL3- and MLL4-containing histone H3 lysine 4 methyltransferase complex53BP1 mediates productive and mutagenic DNA repair through distinct phosphoprotein interactions.Distinct roles of GCN5/PCAF-mediated H3K9ac and CBP/p300-mediated H3K18/27ac in nuclear receptor transactivationLoss of function of mouse Pax-Interacting Protein 1-associated glutamate rich protein 1a (Pagr1a) leads to reduced Bmp2 expression and defects in chorion and amnion developmentPTIP promotes chromatin changes critical for immunoglobulin class switch recombinationUTX mediates demethylation of H3K27me3 at muscle-specific genes during myogenesisA PTIP-PA1 subcomplex promotes transcription for IgH class switching independently from the associated MLL3/MLL4 methyltransferase complexHistone demethylases UTX and JMJD3 are required for NKT cell development in miceTranscriptional and epigenetic regulation of PPARγ expression during adipogenesis.Histone H3K27 methyltransferase Ezh2 represses Wnt genes to facilitate adipogenesis.MLL3/MLL4 are required for CBP/p300 binding on enhancers and super-enhancer formation in brown adipogenesis.Spatial clustering for identification of ChIP-enriched regions (SICER) to map regions of histone methylation patterns in embryonic stem cells.Affinity purification of MLL3/MLL4 histone H3K4 methyltransferase complex.Gcn5 and PCAF regulate PPARγ and Prdm16 expression to facilitate brown adipogenesisH3K4 mono- and di-methyltransferase MLL4 is required for enhancer activation during cell differentiation.The DNA damage- and transcription-associated protein paxip1 controls thymocyte development and emigration.Disruption of KMT2D perturbs germinal center B cell development and promotes lymphomagenesis.Enhancer priming by H3K4 methyltransferase MLL4 controls cell fate transition.H3K4 Methyltransferase Activity Is Required for MLL4 Protein Stability.Gcn5 and PCAF negatively regulate interferon-β production through HAT-independent inhibition of TBK1.DNA-damage-induced differentiation of leukaemic cells as an anti-cancer barrier.Epigenetic regulation of adipogenesis by histone methylation.Replication fork stability confers chemoresistance in BRCA-deficient cellsErratum: Replication fork stability confers chemoresistance in BRCA-deficient cells.EZH2 Mediates epigenetic silencing of neuroblastoma suppressor genes CASZ1, CLU, RUNX3, and NGFRGlobal identification of MLL2-targeted loci reveals MLL2's role in diverse signaling pathwaysMechanism for elimination of a tumor suppressor: aberrant splicing of a brain-specific exon causes loss of function of Bin1 in melanoma.H2A.Z facilitates access of active and repressive complexes to chromatin in embryonic stem cell self-renewal and differentiation.UTX inhibition as selective epigenetic therapy against TAL1-driven T-cell acute lymphoblastic leukemiaUTX demethylase activity is required for satellite cell-mediated muscle regeneration.Histone Demethylase KDM6A Controls the Mammary Luminal Lineage through Enzyme-Independent Mechanisms.Mammary-Stem-Cell-Based Somatic Mouse Models Reveal Breast Cancer Drivers Causing Cell Fate DysregulationDistinct Roles of Transcription Factors KLF4, Krox20, and Peroxisome Proliferator-Activated Receptor γ in Adipogenesis.Glucocorticoid Receptor Accelerates, but Is Dispensable for, AdipogenesisAn epigenetic switch induced by Shh signalling regulates gene activation during development and medulloblastoma growth.Heterogeneous expression and functions of androgen receptor co-factors in primary prostate cancer.Histone H3 lysine 4 methyltransferase KMT2D.Targeted disruption of the murine Bin1/Amphiphysin II gene does not disable endocytosis but results in embryonic cardiomyopathy with aberrant myofibril formation.Thyroid hormone-induced juxtaposition of regulatory elements/factors and chromatin remodeling of Crabp1 dependent on MED1/TRAP220.
P50
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P50
description
onderzoeker
@nl
researcher
@en
հետազոտող
@hy
name
Kai Ge
@ast
Kai Ge
@en
Kai Ge
@es
Kai Ge
@nl
Kai Ge
@sl
type
label
Kai Ge
@ast
Kai Ge
@en
Kai Ge
@es
Kai Ge
@nl
Kai Ge
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altLabel
Ge K
@en
prefLabel
Kai Ge
@ast
Kai Ge
@en
Kai Ge
@es
Kai Ge
@nl
Kai Ge
@sl
P106
P31
P3829
P496
0000-0002-7442-5138