FANCM, BRCA1, and BLM cooperatively resolve the replication stress at the ALT telomeres.
about
Diagnosis and treatment of ALT tumors: is Trabectedin a new therapeutic option?BLM and SLX4 play opposing roles in recombination-dependent replication at human telomeres.Breaking the end: Target the replication stress response at the ALT telomeres for cancer therapy.The Guardian of the Genome Revisited: p53 Downregulates Genes Required for Telomere Maintenance, DNA Repair, and Centromere Structure.
P2860
FANCM, BRCA1, and BLM cooperatively resolve the replication stress at the ALT telomeres.
description
2017 nî lūn-bûn
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2017年の論文
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2017年学术文章
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2017年学术文章
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2017年学术文章
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2017年学术文章
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2017年学术文章
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2017年学术文章
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2017年學術文章
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2017年學術文章
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name
FANCM, BRCA1, and BLM cooperatively resolve the replication stress at the ALT telomeres.
@en
FANCM, BRCA1, and BLM cooperatively resolve the replication stress at the ALT telomeres.
@nl
type
label
FANCM, BRCA1, and BLM cooperatively resolve the replication stress at the ALT telomeres.
@en
FANCM, BRCA1, and BLM cooperatively resolve the replication stress at the ALT telomeres.
@nl
prefLabel
FANCM, BRCA1, and BLM cooperatively resolve the replication stress at the ALT telomeres.
@en
FANCM, BRCA1, and BLM cooperatively resolve the replication stress at the ALT telomeres.
@nl
P2093
P2860
P356
P1476
FANCM, BRCA1, and BLM cooperatively resolve the replication stress at the ALT telomeres
@en
P2093
Carl L Schildkraut
Louisa Sethi
William C Drosopoulos
Xiaolei Pan
P2860
P304
E5940-E5949
P356
10.1073/PNAS.1708065114
P407
P577
2017-07-03T00:00:00Z