Human papillomavirus type 16 E7 associates with a histone H1 kinase and with p107 through sequences necessary for transformation.
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Effects of the human papilloma virus HPV-16 E7 oncoprotein on glycolysis and glutaminolysis: role of pyruvate kinase type M2 and the glycolytic-enzyme complexAssociation of p107 with Sp1: genetically separable regions of p107 are involved in regulation of E2F- and Sp1-dependent transcriptionBRCA1-associated growth arrest is RB-dependentModulation of type M2 pyruvate kinase activity by the human papillomavirus type 16 E7 oncoproteinThe association of mammalian DREAM complex and HPV16 E7 proteinsFunctional Roles of E6 and E7 Oncoproteins in HPV-Induced Malignancies at Diverse Anatomical SitesThe human papillomavirus type 16 E7 gene product interacts with and trans-activates the AP1 family of transcription factorsThe human papillomavirus E7 oncoprotein can uncouple cellular differentiation and proliferation in human keratinocytes by abrogating p21Cip1-mediated inhibition of cdk2.Synergism with germ line transcription factor Oct-4: viral oncoproteins share the ability to mimic a stem cell-specific activity.Binding of p110 retinoblastoma protein inhibits nuclear import of simian virus SV40 large tumor antigen.Anchorage-independent transcription of the cyclin A gene induced by the E7 oncoprotein of human papillomavirus type 16.The human papillomavirus type 16 E6 gene alone is sufficient to induce carcinomas in transgenic animals.Destabilization of the retinoblastoma tumor suppressor by human papillomavirus type 16 E7 is not sufficient to overcome cell cycle arrest in human keratinocytes.Involvement of nuclear export in human papillomavirus type 18 E6-mediated ubiquitination and degradation of p53.p53 facilitates pRb cleavage in IL-3-deprived cells: novel pro-apoptotic activity of p53.Inactivation of p16 in human mammary epithelial cells by CpG island methylationGreen tea compound in chemoprevention of cervical cancer.Myc oncogenes: the enigmatic family.Disruption of the G1/S transition in human papillomavirus type 16 E7-expressing human cells is associated with altered regulation of cyclin EHuman papillomavirus oncoproteins E6 and E7 independently abrogate the mitotic spindle checkpoint.Regulation of the human proliferating cell nuclear antigen promoter by the adenovirus E1A-associated protein p107Human papillomaviruses and the interferon responseBiology of human papillomaviruses.Activation of the retinoblastoma tumor suppressor mediates cell cycle inhibition and cell death in specific cervical cancer cell lines.Pathogenesis of human papillomaviruses in differentiating epithelia.p53-dependent G1 arrest involves pRB-related proteins and is disrupted by the human papillomavirus 16 E7 oncoprotein.CCCTC-binding factor recruitment to the early region of the human papillomavirus 18 genome regulates viral oncogene expression.Sequential activation of cyclin E and cyclin A gene expression by human papillomavirus type 16 E7 through sequences necessary for transformation.The Epstein-Barr virus bZIP transcription factor Zta causes G0/G1 cell cycle arrest through induction of cyclin-dependent kinase inhibitorsAbrogation of growth arrest signals by human papillomavirus type 16 E7 is mediated by sequences required for transformation.Perturbation of the p53 response by human papillomavirus type 16 E7.Initiation of DNA synthesis by human papillomavirus E7 oncoproteins is resistant to p21-mediated inhibition of cyclin E-cdk2 activityAutophagy regulates UBC9 levels during viral-mediated tumorigenesis.Intranuclear localization of human papillomavirus 16 E7 during transformation and preferential binding of E7 to the Rb family member p130Separation of C/EBPalpha-mediated proliferation arrest and differentiation pathways.Reversible repression of papillomavirus oncogene expression in cervical carcinoma cells: consequences for the phenotype and E6-p53 and E7-pRB interactionsFunctional importance of complex formation between the retinoblastoma tumor suppressor family and adenovirus E1A proteins as determined by mutational analysis of E1A conserved region 2Comparison of the properties of the E6 and E7 genes of low- and high-risk cutaneous papillomaviruses reveals strongly transforming and high Rb-binding activity for the E7 protein of the low-risk human papillomavirus type 1.Positive and negative regulation of cell proliferation by E2F-1: influence of protein level and human papillomavirus oncoproteinsInduction of tetrasomy by human papillomavirus type 16 E7 protein is independent of pRb binding and disruption of differentiation.
P2860
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P2860
Human papillomavirus type 16 E7 associates with a histone H1 kinase and with p107 through sequences necessary for transformation.
description
1993 nî lūn-bûn
@nan
1993 թուականի Մայիսին հրատարակուած գիտական յօդուած
@hyw
1993 թվականի մայիսին հրատարակված գիտական հոդված
@hy
1993年の論文
@ja
1993年論文
@yue
1993年論文
@zh-hant
1993年論文
@zh-hk
1993年論文
@zh-mo
1993年論文
@zh-tw
1993年论文
@wuu
name
Human papillomavirus type 16 E ...... necessary for transformation.
@ast
Human papillomavirus type 16 E ...... necessary for transformation.
@en
type
label
Human papillomavirus type 16 E ...... necessary for transformation.
@ast
Human papillomavirus type 16 E ...... necessary for transformation.
@en
prefLabel
Human papillomavirus type 16 E ...... necessary for transformation.
@ast
Human papillomavirus type 16 E ...... necessary for transformation.
@en
P2093
P2860
P1433
P1476
Human papillomavirus type 16 E ...... necessary for transformation.
@en
P2093
P2860
P304
P577
1993-05-01T00:00:00Z