Activation of liver X receptors and retinoid X receptors prevents bacterial-induced macrophage apoptosis.
about
The nuclear receptor Rev-erbalpha is a liver X receptor (LXR) target gene driving a negative feedback loop on select LXR-induced pathways in human macrophagesThe retinoid X receptors and their ligandsSimple combinations of lineage-determining transcription factors prime cis-regulatory elements required for macrophage and B cell identitiesLiver X receptors contribute to the protective immune response against Mycobacterium tuberculosis in miceParallel SUMOylation-dependent pathways mediate gene- and signal-specific transrepression by LXRs and PPARgammaLXR-dependent and -independent effects of oxysterols on immunity and tumor growthMitochondrial function and regulation of macrophage sterol metabolism and inflammatory responsesLiver X receptor and peroxisome proliferator-activated receptor as integrators of lipid homeostasis and immunityLiver X Receptor Agonist TO901317 Attenuates Paraquat-Induced Acute Lung Injury through Inhibition of NF-κB and JNK/p38 MAPK Signal Pathways.Differential regulation of gene expression by LXRs in response to macrophage cholesterol loadingAndrogen deprivation by activating the liver X receptor.The scavenger protein apoptosis inhibitor of macrophages (AIM) potentiates the antimicrobial response against Mycobacterium tuberculosis by enhancing autophagy.Stabilization and augmentation of circulating AIM in mice by synthesized IgM-Fc.Role for parasite genetic diversity in differential host responses to Trypanosoma brucei infection.The macrophage LBP gene is an LXR target that promotes macrophage survival and atherosclerosis.Liver X receptors alpha and beta regulate renin expression in vivoAkt2 modulates glucose availability and downstream apoptotic pathways during development.Kdo2-Lipid A of Escherichia coli, a defined endotoxin that activates macrophages via TLR-4.Sterol intermediates of cholesterol biosynthesis inhibit hair growth and trigger an innate immune response in cicatricial alopecia.Circulating AIM as an indicator of liver damage and hepatocellular carcinoma in humans.LXR-mediated inhibition of CD4+ T helper cells.Serum response factor utilizes distinct promoter- and enhancer-based mechanisms to regulate cytoskeletal gene expression in macrophages.Listeria monocytogenes infection induces prosurvival metabolic signaling in macrophages.Apoptosis inhibitor of macrophage (AIM) is required for obesity-associated recruitment of inflammatory macrophages into adipose tissue.Macrophage glucocorticoid receptors regulate Toll-like receptor 4-mediated inflammatory responses by selective inhibition of p38 MAP kinaseAttenuation of neuroinflammation and Alzheimer's disease pathology by liver x receptors.The human CD5L/AIM-CD36 axis: A novel autophagy inducer in macrophages that modulates inflammatory responses.Association of polymorphisms in the MAFB gene and the risk of coronary artery disease and ischemic stroke: a case-control studyInteraction of AIM with insulin-like growth factor-binding protein-4.Myeloid cell-specific ABCA1 deletion protects mice from bacterial infection.Combinatorial roles of nuclear receptors in inflammation and immunity.Expression profiling in APP23 mouse brain: inhibition of Abeta amyloidosis and inflammation in response to LXR agonist treatmentLiver X receptors as integrators of metabolic and inflammatory signalingLipoteichoic acid may affect the pathogenesis of PBC-like bile duct damage and might be involved in systemic multifocal epithelial inflammations in chronic colitis-harboring TCRalpha-/-xAIM-/- mice.Effects of liver X receptor agonist treatment on pulmonary inflammation and host defense.MafB deficiency accelerates the development of obesity in miceNuclear receptors in macrophages: a link between metabolism and inflammation.LXR signaling couples sterol metabolism to proliferation in the acquired immune response.Gastrointestinal inflammation: lessons from metabolic modulators.Coordination of inflammation and metabolism by PPAR and LXR nuclear receptors.
P2860
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P2860
Activation of liver X receptors and retinoid X receptors prevents bacterial-induced macrophage apoptosis.
description
2004 nî lūn-bûn
@nan
2004 թուականի Դեկտեմբերին հրատարակուած գիտական յօդուած
@hyw
2004 թվականի դեկտեմբերին հրատարակված գիտական հոդված
@hy
2004年の論文
@ja
2004年論文
@yue
2004年論文
@zh-hant
2004年論文
@zh-hk
2004年論文
@zh-mo
2004年論文
@zh-tw
2004年论文
@wuu
name
Activation of liver X receptor ...... -induced macrophage apoptosis.
@ast
Activation of liver X receptor ...... -induced macrophage apoptosis.
@en
Activation of liver X receptor ...... -induced macrophage apoptosis.
@nl
type
label
Activation of liver X receptor ...... -induced macrophage apoptosis.
@ast
Activation of liver X receptor ...... -induced macrophage apoptosis.
@en
Activation of liver X receptor ...... -induced macrophage apoptosis.
@nl
prefLabel
Activation of liver X receptor ...... -induced macrophage apoptosis.
@ast
Activation of liver X receptor ...... -induced macrophage apoptosis.
@en
Activation of liver X receptor ...... -induced macrophage apoptosis.
@nl
P2093
P2860
P356
P1476
Activation of liver X receptor ...... -induced macrophage apoptosis.
@en
P2093
Annabel F Valledor
Christopher K Glass
Dominique Sawka-Verhelle
Sumito Ogawa
P2860
P304
17813-17818
P356
10.1073/PNAS.0407749101
P407
P577
2004-12-15T00:00:00Z