Prion infection impairs the cellular response to oxidative stress
about
Loss of m-AAA protease in mitochondria causes complex I deficiency and increased sensitivity to oxidative stress in hereditary spastic paraplegiaHot spots in prion protein for pathogenic conversionThe human "prion-like" protein Doppel is expressed in both Sertoli cells and spermatozoaExpression of prion protein increases cellular copper binding and antioxidant enzyme activities but not copper deliveryVariety of antiprion compounds discovered through an in silico screen based on cellular-form prion protein structure: Correlation between antiprion activity and binding affinity.Redox control of prion and disease pathogenesis.Characterizing antiprion compounds based on their binding properties to prion proteins: implications as medical chaperones.What is the basis of transmissible spongiform encephalopathy induced neurodegeneration and can it be repaired?Primary blast-induced traumatic brain injury in rats leads to increased prion protein in plasma: a potential biomarker for blast-induced traumatic brain injuryPrion replication occurs in endogenous adult neural stem cells and alters their neuronal fate: involvement of endogenous neural stem cells in prion diseasesPrion protein accumulation and neuroprotection in hypoxic brain damage.Infectious prion protein alters manganese transport and neurotoxicity in a cell culture model of prion disease.Prions impair bioaminergic functions through serotonin- or catecholamine-derived neurotoxins in neuronal cells.Prion Protein Does Not Confer Resistance to Hippocampus-Derived Zpl Cells against the Toxic Effects of Cu2+, Mn2+, Zn2+ and Co2+ Not Supporting a General Protective Role for PrP in Transition Metal Induced Toxicity.Silibinin induces mitochondrial NOX4-mediated endoplasmic reticulum stress response and its subsequent apoptosisMethionine oxidation perturbs the structural core of the prion protein and suggests a generic misfolding pathwayAntioxidative and steroid systems in neurological and psychiatric disorders.Glutathione peroxidase (GPX) activity in blood of ewes on farms in different scrapie categories in IcelandMitochondrial dysfunction, oxidative stress, and major depressive disorderAntioxidants reduce endoplasmic reticulum stress and improve protein secretion.Design of anti- and pro-aggregation variants to assess the effects of methionine oxidation in human prion protein.Prions can infect primary cultured neurons and astrocytes and promote neuronal cell deathMetalloproteins and neuronal death.Prions and manganese: A maddening beast.Proteolytic processing of the prion protein in health and disease.Modelling neurodegeneration in prion disease - applications for drug development.Regulation of proteasomes in prion disease.The potential of mesenchymal stem cell in prion research.Multitarget ligands and theranostics: sharpening the medicinal chemistry sword against prion diseases.Analysis of nucleic acid chaperoning by the prion protein and its inhibition by oligonucleotides.Proteomic Analyses for the Global S-Nitrosylated Proteins in the Brain Tissues of Different Human Prion Diseases.Involvement of Neuroinflammation during Brain Development in Social Cognitive Deficits in Autism Spectrum Disorder and Schizophrenia.Rational approach to an antiprion compound with a multiple mechanism of action.Hybrid lipoic acid derivatives to attack prion disease on multiple fronts.Oxidative stress impairs intracellular events involved in antigen processing and presentation to T cellsScrapie infection of prion protein-deficient cell line upon ectopic expression of mutant prion proteins.Mitochondrial Respiration Is Impaired during Late-Stage Hamster Prion Infection.Styrylbenzoazole derivatives for imaging of prion plaques and treatment of transmissible spongiform encephalopathies.The radical scavenger CR-6 protects SH-SY5Y neuroblastoma cells from oxidative stress-induced apoptosis: effect on survival pathways.Biological and biochemical characteristics of prion strains conserved in persistently infected cell cultures.
P2860
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P2860
Prion infection impairs the cellular response to oxidative stress
description
2000 nî lūn-bûn
@nan
2000年の論文
@ja
2000年論文
@yue
2000年論文
@zh-hant
2000年論文
@zh-hk
2000年論文
@zh-mo
2000年論文
@zh-tw
2000年论文
@wuu
2000年论文
@zh
2000年论文
@zh-cn
name
Prion infection impairs the cellular response to oxidative stress
@ast
Prion infection impairs the cellular response to oxidative stress
@en
type
label
Prion infection impairs the cellular response to oxidative stress
@ast
Prion infection impairs the cellular response to oxidative stress
@en
prefLabel
Prion infection impairs the cellular response to oxidative stress
@ast
Prion infection impairs the cellular response to oxidative stress
@en
P2093
P2860
P50
P356
P1476
Prion infection impairs the cellular response to oxidative stress
@en
P2093
Casanova D
Katamine S
P2860
P304
13937-13942
P356
10.1073/PNAS.250289197
P407
P577
2000-12-01T00:00:00Z