Misfolded major histocompatibility complex class I molecules accumulate in an expanded ER-Golgi intermediate compartment
about
Proteomics of endoplasmic reticulum-Golgi intermediate compartment (ERGIC) membranes from brefeldin A-treated HepG2 cells identifies ERGIC-32, a new cycling protein that interacts with human Erv46EDEM1 reveals a quality control vesicular transport pathway out of the endoplasmic reticulum not involving the COPII exit sitesRoad to ruin: targeting proteins for degradation in the endoplasmic reticulumWhat is the role of alternate splicing in antigen presentation by major histocompatibility complex class I molecules?Inclusion body myopathy-associated mutations in p97/VCP impair endoplasmic reticulum-associated degradationThe oxidoreductase ERp57 efficiently reduces partially folded in preference to fully folded MHC class I moleculesRab1 defines a novel pathway connecting the pre-Golgi intermediate compartment with the cell periphery.Immunolocalization of UDP-glucose:glycoprotein glucosyltransferase indicates involvement of pre-Golgi intermediates in protein quality controlTopology of molecular machines of the endoplasmic reticulum: a compilation of proteomics and cytological data.Probing for membrane domains in the endoplasmic reticulum: retention and degradation of unassembled MHC class I molecules.A striking quality control subcompartment in Saccharomyces cerevisiae: the endoplasmic reticulum-associated compartment.Adopting the rapamycin trapping assay to track the trafficking of murine MHC class I alleles, H-2K(b)Tapasin and other chaperones: models of the MHC class I loading complex.Misfolded major histocompatibility complex class I heavy chains are translocated into the cytoplasm and degraded by the proteasome.Protein quality control: the who's who, the where's and therapeutic escapes.Normal Molecular Specification and Neurodegenerative Disease-Like Death of Spinal Neurons Lacking the SNARE-Associated Synaptic Protein Munc18-1.Proteasomes, TAP, and endoplasmic reticulum-associated aminopeptidase associated with antigen processing control CD4+ Th cell responses by regulating indirect presentation of MHC class II-restricted cytoplasmic antigensEndoplasmic reticulum quality control of asialoglycoprotein receptor H2a involves a determinant for retention and not retrieval.Apoprotein B100 has a prolonged interaction with the translocon during which its lipidation and translocation change from dependence on the microsomal triglyceride transfer protein to independence.Degradation of aggrecan precursors within a specialized subcompartment of the chicken chondrocyte endoplasmic reticulum.Hereditary hemochromatosis: effects of C282Y and H63D mutations on association with beta2-microglobulin, intracellular processing, and cell surface expression of the HFE protein in COS-7 cells.Immunoisolation and characterization of a subdomain of the endoplasmic reticulum that concentrates proteins involved in COPII vesicle biogenesis.Pathogen evasion strategies for the major histocompatibility complex class I assembly pathway.Endoplasmic reticulum stress: its role in disease and novel prospects for therapy.Protein N-glycosylation, protein folding, and protein quality control.The Gp78 ubiquitin ligase: probing endoplasmic reticulum complexity.An updated view of the intracellular mechanisms regulating cross-presentation.A novel quality control compartment derived from the endoplasmic reticulumThe comings and goings of MHC class I molecules herald a new dawn in cross-presentation.Viral glycoproteins accumulate in newly formed annulate lamellae following infection of lymphoid cells by human herpesvirus 6.Targeting TEAD/YAP-transcription-dependent necrosis, TRIAD, ameliorates Huntington's disease pathology.Down-regulation of MHC class I by bovine papillomavirus E5 oncoproteins.Glycosylation increases potassium channel stability and surface expression in mammalian cells.Trafficking and folding defects in hereditary spherocytosis mutants of the human red cell anion exchanger.Selective fast degradation of cytochrome P-450 2E1 in serum-deprived hepatoma cells by a mechanism sensitive to inhibitors of vesicular transport.Cellular processing limits the heterologous expression of secretory component in mammalian cells.Intracellular assembly and degradation of apolipoprotein B-100-containing lipoproteins in digitonin-permeabilized HEP G2 cells.A cellular mechanism governing the severity of Pelizaeus-Merzbacher disease.Alpha-1-antitrypsin deficiency: biochemistry and clinical manifestations.Review: alpha 1-antitrypsin deficiency associated liver disease.
P2860
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P2860
Misfolded major histocompatibility complex class I molecules accumulate in an expanded ER-Golgi intermediate compartment
description
1995 nî lūn-bûn
@nan
1995年の論文
@ja
1995年学术文章
@wuu
1995年学术文章
@zh-cn
1995年学术文章
@zh-hans
1995年学术文章
@zh-my
1995年学术文章
@zh-sg
1995年學術文章
@yue
1995年學術文章
@zh
1995年學術文章
@zh-hant
name
Misfolded major histocompatibi ...... Golgi intermediate compartment
@ast
Misfolded major histocompatibi ...... Golgi intermediate compartment
@en
type
label
Misfolded major histocompatibi ...... Golgi intermediate compartment
@ast
Misfolded major histocompatibi ...... Golgi intermediate compartment
@en
prefLabel
Misfolded major histocompatibi ...... Golgi intermediate compartment
@ast
Misfolded major histocompatibi ...... Golgi intermediate compartment
@en
P2093
P2860
P356
P1476
Misfolded major histocompatibi ...... Golgi intermediate compartment
@en
P2093
H L Ploegh
H M van Santen
R Leijendekker
P2860
P304
P356
10.1083/JCB.131.6.1403
P407
P433
P577
1995-12-01T00:00:00Z