EBNA1 can link the enhancer element to the initiator element of the Epstein-Barr virus plasmid origin of DNA replication.
about
The cellular proteins that bind specifically to the Epstein-Barr virus origin of plasmid DNA replication belong to a gene familyDual stimulation of Epstein-Barr Virus (EBV)-specific CD4+- and CD8+-T-cell responses by a chimeric antigen construct: potential therapeutic vaccine for EBV-positive nasopharyngeal carcinomaEBNA-1, a bifunctional transcriptional activatorEpstein-Barr virus latent genesEpstein-Barr virus nuclear antigen 1 does not cause lymphoma in C57BL/6J miceZinc coordination is required for and regulates transcription activation by Epstein-Barr nuclear antigen 1A role for DNA polymerase θ in promoting replication through oxidative DNA lesion, thymine glycol, in human cells.Functional analyses of the EBNA1 origin DNA binding protein of Epstein-Barr virus.The minimal replicator of Epstein-Barr virus oriP.Human DNA replication initiation factors, ORC and MCM, associate with oriP of Epstein-Barr virus.EBV latency types adopt alternative chromatin conformations.Constitutive binding of EBNA1 protein to the Epstein-Barr virus replication origin, oriP, with distortion of DNA structure during latent infection.The spacing between adjacent binding sites in the family of repeats affects the functions of Epstein-Barr nuclear antigen 1 in transcription activation and stable plasmid maintenanceHuman origin recognition complex binds to the region of the latent origin of DNA replication of Epstein-Barr virus.Separation of the DNA replication, segregation, and transcriptional activation functions of Epstein-Barr nuclear antigen 1.Cooperative assembly of EBNA1 on the Epstein-Barr virus latent origin of replicationGenetic control of translesion synthesis on leading and lagging DNA strands in plasmids derived from Epstein-Barr virus in human cells.Identification of MEF2B, EBF1, and IL6R as Direct Gene Targets of Epstein-Barr Virus (EBV) Nuclear Antigen 1 Critical for EBV-Infected B-Lymphocyte SurvivalMetaphase chromosome tethering is necessary for the DNA synthesis and maintenance of oriP plasmids but is insufficient for transcription activation by Epstein-Barr nuclear antigen 1.Initiation of latent DNA replication in the Epstein-Barr virus genome can occur at sites other than the genetically defined originIdentification of a short amino acid sequence essential for efficient nuclear targeting of the Epstein-Barr virus nuclear antigen 3A.Effect of number and position of EBNA-1 binding sites in Epstein-Barr virus oriP on the sites of initiation, barrier formation, and termination of replicationEasily unwound DNA sequences and hairpin structures in the Epstein-Barr virus origin of plasmid replication.Identification of EBNA1 amino acid sequences required for the interaction of the functional elements of the Epstein-Barr virus latent origin of DNA replicationSeparation of the complex DNA binding domain of EBNA-1 into DNA recognition and dimerization subdomains of novel structure.The plasmid replicon of Epstein-Barr virus: mechanistic insights into efficient, licensed, extrachromosomal replication in human cells.Identifying sites bound by Epstein-Barr virus nuclear antigen 1 (EBNA1) in the human genome: defining a position-weighted matrix to predict sites bound by EBNA1 in viral genomes.Phosphorylation sites of Epstein-Barr virus EBNA1 regulate its functionThe Epstein-Barr Virus EBNA1 Protein.The linking regions of EBNA1 are essential for its support of replication and transcription.Genetic evidence that EBNA-1 is needed for efficient, stable latent infection by Epstein-Barr virusRep*: a viral element that can partially replace the origin of plasmid DNA synthesis of Epstein-Barr virus.Replication from oriP of Epstein-Barr virus requires exact spacing of two bound dimers of EBNA1 which bend DNA.cis-Acting components of human papillomavirus (HPV) DNA replication: linker substitution analysis of the HPV type 11 origin.Multiple regions within EBNA1 can link DNAs.AP1 enhances polyomavirus DNA replication by promoting T-antigen-mediated unwinding of DNADominant-negative inhibitors of EBNA-1 of Epstein-Barr virus.Conditional gene vectors regulated in cis.Sequence requirements of the Epstein-Barr virus latent origin of DNA replication.The plasmid replicon of EBV consists of multiple cis-acting elements that facilitate DNA synthesis by the cell and a viral maintenance element
P2860
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P2860
EBNA1 can link the enhancer element to the initiator element of the Epstein-Barr virus plasmid origin of DNA replication.
description
1992 nî lūn-bûn
@nan
1992年の論文
@ja
1992年論文
@yue
1992年論文
@zh-hant
1992年論文
@zh-hk
1992年論文
@zh-mo
1992年論文
@zh-tw
1992年论文
@wuu
1992年论文
@zh
1992年论文
@zh-cn
name
EBNA1 can link the enhancer el ...... mid origin of DNA replication.
@ast
EBNA1 can link the enhancer el ...... mid origin of DNA replication.
@en
type
label
EBNA1 can link the enhancer el ...... mid origin of DNA replication.
@ast
EBNA1 can link the enhancer el ...... mid origin of DNA replication.
@en
prefLabel
EBNA1 can link the enhancer el ...... mid origin of DNA replication.
@ast
EBNA1 can link the enhancer el ...... mid origin of DNA replication.
@en
P2860
P1433
P1476
EBNA1 can link the enhancer el ...... mid origin of DNA replication.
@en
P2093
P2860
P304
P407
P577
1992-01-01T00:00:00Z