Activation of NF-kappaB by adherent Pseudomonas aeruginosa in normal and cystic fibrosis respiratory epithelial cells.
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Digitoxin mimics gene therapy with CFTR and suppresses hypersecretion of IL-8 from cystic fibrosis lung epithelial cellsImmunostimulatory properties of the emerging pathogen Stenotrophomonas maltophiliaConstruction of predictive promoter models on the example of antibacterial response of human epithelial cellsState of the art: why do the lungs of patients with cystic fibrosis become infected and why can't they clear the infection?Relation of exaggerated cytokine responses of CF airway epithelial cells to PAO1 adherenceProduction of beta-defensin-2 by human colonic epithelial cells induced by Salmonella enteritidis flagella filament structural proteinHyperacidification of cellubrevin endocytic compartments and defective endosomal recycling in cystic fibrosis respiratory epithelial cellsPseudomonas aeruginosa exploits lipid A and muropeptides modification as a strategy to lower innate immunity during cystic fibrosis lung infectionPEI-engineered respirable particles delivering a decoy oligonucleotide to NF-κB: inhibiting MUC2 expression in LPS-stimulated airway epithelial cellsComparative molecular docking analysis of cytoplasmic dynein light chain DYNLL1 with pilin to explore the molecular mechanism of pathogenesis caused by Pseudomonas aeruginosa PAODown-regulated CFTR During Aging Contributes to Benign Prostatic HyperplasiaLung infections associated with cystic fibrosis.IL-17 is a critical component of vaccine-induced protection against lung infection by lipopolysaccharide-heterologous strains of Pseudomonas aeruginosa.The two-component sensor response regulator RoxS/RoxR plays a role in Pseudomonas aeruginosa interactions with airway epithelial cells.NF-kappa B mediates up-regulation of CFTR gene expression in Calu-3 cells by interleukin-1beta.De novo biosynthetic profiling of high abundance proteins in cystic fibrosis lung epithelial cells.Unfractionated heparin reduces the elasticity of sputum from patients with cystic fibrosis.CFTR is a negative regulator of NFkappaB mediated innate immune response.Lack of CFTR in skeletal muscle predisposes to muscle wasting and diaphragm muscle pump failure in cystic fibrosis micePseudomonas aeruginosa biofilm infections in cystic fibrosis: insights into pathogenic processes and treatment strategies.CXC chemokine receptor CXCR2 is essential for protective innate host response in murine Pseudomonas aeruginosa pneumonia.Disruption of interleukin-1β autocrine signaling rescues complex I activity and improves ROS levels in immortalized epithelial cells with impaired cystic fibrosis transmembrane conductance regulator (CFTR) function.TLR-4-mediated innate immunity is reduced in cystic fibrosis airway cells.Cystic fibrosis-related bone disease: insights into a growing problem.Yet another role for the cystic fibrosis transmembrane conductance regulator.Cystic fibrosis: NHLBI Workshop on the Primary Prevention of Chronic Lung Diseases.Cardiac glycosides inhibit TNF-alpha/NF-kappaB signaling by blocking recruitment of TNF receptor-associated death domain to the TNF receptor.Synergistic proinflammatory responses induced by polymicrobial colonization of epithelial surfacesLactoperoxidase. New recognition of an "old" enzyme in airway defenses.Induction of proinflammatory cytokines from human respiratory epithelial cells after stimulation by nontypeable Haemophilus influenzae.CFTR is a pattern recognition molecule that extracts Pseudomonas aeruginosa LPS from the outer membrane into epithelial cells and activates NF-kappa B translocationSERCA2 regulates non-CF and CF airway epithelial cell response to ozoneStaphylococcus aureus agr and sarA functions are required for invasive infection but not inflammatory responses in the lung.Plasma membrane CFTR regulates RANTES expression via its C-terminal PDZ-interacting motif.Normal CFTR inhibits epidermal growth factor receptor-dependent pro-inflammatory chemokine production in human airway epithelial cells.Interaction of pseudomonas aeruginosa with epithelial cells: identification of differentially regulated genes by expression microarray analysis of human cDNAsPotential of anti-inflammatory treatment for cystic fibrosis lung disease.Fas and Fas ligand expression in cystic fibrosis airway epitheliumEnhanced Ca2+ entry due to Orai1 plasma membrane insertion increases IL-8 secretion by cystic fibrosis airwaysThe role of nuclear factor kappa B in the pathogenesis of pulmonary diseases: implications for therapy.
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P2860
Activation of NF-kappaB by adherent Pseudomonas aeruginosa in normal and cystic fibrosis respiratory epithelial cells.
description
article científic
@ca
article scientifique
@fr
articolo scientifico
@it
artigo científico
@pt
bilimsel makale
@tr
scientific article published on June 1998
@en
vedecký článok
@sk
vetenskaplig artikel
@sv
videnskabelig artikel
@da
vědecký článek
@cs
name
Activation of NF-kappaB by adh ...... respiratory epithelial cells.
@en
Activation of NF-kappaB by adh ...... respiratory epithelial cells.
@nl
type
label
Activation of NF-kappaB by adh ...... respiratory epithelial cells.
@en
Activation of NF-kappaB by adh ...... respiratory epithelial cells.
@nl
prefLabel
Activation of NF-kappaB by adh ...... respiratory epithelial cells.
@en
Activation of NF-kappaB by adh ...... respiratory epithelial cells.
@nl
P2093
P2860
P356
P1476
Activation of NF-kappaB by adh ...... respiratory epithelial cells.
@en
P2093
P2860
P304
P356
10.1172/JCI2865
P407
P577
1998-06-01T00:00:00Z