Ras activity in acinar cells links chronic pancreatitis and pancreatic cancer.
about
Acute pancreatitis accelerates initiation and progression to pancreatic cancer in mice expressing oncogenic Kras in the nestin cell lineageUtilizing past and present mouse systems to engineer more relevant pancreatic cancer modelsAnimal models of gastrointestinal and liver diseases. Animal models of acute and chronic pancreatitisExocrine pancreatic carcinogenesis and autotaxin expressionInflammation to cancer: The molecular biology in the pancreas (Review).Pancreatic cancer and its stroma: a conspiracy theoryOverexpressed galectin-3 in pancreatic cancer induces cell proliferation and invasion by binding Ras and activating Ras signaling.Pancreatitis-diabetes-pancreatic cancer: summary of an NIDDK-NCI workshop.Protein kinase D1 drives pancreatic acinar cell reprogramming and progression to intraepithelial neoplasiaDeregulation of the MiR-193b-KRAS Axis Contributes to Impaired Cell Growth in Pancreatic CancerAn NF-κB pathway-mediated positive feedback loop amplifies Ras activity to pathological levels in mice.Complexity of molecular alterations impacts pancreatic cancer prognosis.Acting on Hormone Receptors with Minimal Side Effect on Cell Proliferation: A Timely Challenge Illustrated with GLP-1R and GPEREVI1 oncogene promotes KRAS pathway through suppression of microRNA-96 in pancreatic carcinogenesis.Pathobiology of pancreatic cancer: implications on therapy.p53 mutations cooperate with oncogenic Kras to promote adenocarcinoma from pancreatic ductal cells.Elevated miR-483-3p expression is an early event and indicates poor prognosis in pancreatic ductal adenocarcinoma.Acinar cell plasticity and development of pancreatic ductal adenocarcinoma.Transcriptional Maintenance of Pancreatic Acinar Identity, Differentiation, and Homeostasis by PTF1A.Activating transcription factor 3 promotes loss of the acinar cell phenotype in response to cerulein-induced pancreatitis in mice.CDKN2B deletion is essential for pancreatic cancer development instead of unmeaningful co-deletion due to juxtaposition to CDKN2A.
P2860
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P2860
Ras activity in acinar cells links chronic pancreatitis and pancreatic cancer.
description
2009 nî lūn-bûn
@nan
2009年の論文
@ja
2009年学术文章
@wuu
2009年学术文章
@zh-cn
2009年学术文章
@zh-hans
2009年学术文章
@zh-my
2009年学术文章
@zh-sg
2009年學術文章
@yue
2009年學術文章
@zh
2009年學術文章
@zh-hant
name
Ras activity in acinar cells links chronic pancreatitis and pancreatic cancer.
@en
Ras activity in acinar cells links chronic pancreatitis and pancreatic cancer.
@nl
type
label
Ras activity in acinar cells links chronic pancreatitis and pancreatic cancer.
@en
Ras activity in acinar cells links chronic pancreatitis and pancreatic cancer.
@nl
prefLabel
Ras activity in acinar cells links chronic pancreatitis and pancreatic cancer.
@en
Ras activity in acinar cells links chronic pancreatitis and pancreatic cancer.
@nl
P2860
P1476
Ras activity in acinar cells links chronic pancreatitis and pancreatic cancer.
@en
P2093
Craig D Logsdon
P2860
P356
10.1016/J.CGH.2009.07.040
P407
P433
P577
2009-11-01T00:00:00Z