Saccharomyces cerevisiae pol30 (proliferating cell nuclear antigen) mutations impair replication fidelity and mismatch repair.
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Functional interaction of proliferating cell nuclear antigen with MSH2-MSH6 and MSH2-MSH3 complexesDistinct Structural Alterations in Proliferating Cell Nuclear Antigen Block DNA Mismatch RepairRad52 forms DNA repair and recombination centers during S phase.Functional studies on the candidate ATPase domains of Saccharomyces cerevisiae MutLalpha.exo1-Dependent mutator mutations: model system for studying functional interactions in mismatch repair.Interaction between PCNA and diubiquitinated Mcm10 is essential for cell growth in budding yeastGenetic analysis of Saccharomyces cerevisiae H2A serine 129 mutant suggests a functional relationship between H2A and the sister-chromatid cohesion partners Csm3-Tof1 for the repair of topoisomerase I-induced DNA damage.Large-scale prediction of Saccharomyces cerevisiae gene function using overlapping transcriptional clusters.Role of proliferating cell nuclear antigen interactions in the mismatch repair-dependent processing of mitotic and meiotic recombination intermediates in yeast.Pol32, a subunit of Saccharomyces cerevisiae DNA polymerase delta, suppresses genomic deletions and is involved in the mutagenic bypass pathway.Unligated Okazaki Fragments Induce PCNA Ubiquitination and a Requirement for Rad59-Dependent Replication Fork Progression.DNA mismatch repair and mutation avoidance pathwaysIn vivo consequences of putative active site mutations in yeast DNA polymerases alpha, epsilon, delta, and zetaPartial reconstitution of human DNA mismatch repair in vitro: characterization of the role of human replication protein AA PLAC8-containing protein from an endomycorrhizal fungus confers cadmium resistance to yeast cells by interacting with Mlh3pRPA and PCNA suppress formation of large deletion errors by yeast DNA polymerase delta.Reconstitution of Saccharomyces cerevisiae DNA polymerase ε-dependent mismatch repair with purified proteins.Mutational spectrum analysis of RNase H(35) deficient Saccharomyces cerevisiae using fluorescence-based directed termination PCR.Interactions of Exo1p with components of MutLalpha in Saccharomyces cerevisiae.Human DNA mismatch repair in vitro operates independently of methylation status at CpG sitesMitotic recombination in yeast: elements controlling its incidence.Efficient repair of large DNA loops in Saccharomyces cerevisiaePCNA and Msh2-Msh6 activate an Mlh1-Pms1 endonuclease pathway required for Exo1-independent mismatch repair.Roles for mismatch repair factors in regulating genetic recombination.Processivity clamp gp45 and ssDNA-binding-protein gp32 modulate the fidelity of bacteriophage RB69 DNA polymerase in a sequence-specific manner, sometimes enhancing and sometimes compromising accuracyMechanisms of mutagenesis in vivo due to imbalanced dNTP pools.EXO1 and MSH6 are high-copy suppressors of conditional mutations in the MSH2 mismatch repair gene of Saccharomyces cerevisiae.Analysis of conditional mutations in the Saccharomyces cerevisiae MLH1 gene in mismatch repair and in meiotic crossing over.Fragile DNA motifs trigger mutagenesis at distant chromosomal loci in saccharomyces cerevisiae.The effects of mismatch repair and RAD1 genes on interchromosomal crossover recombination in Saccharomyces cerevisiae.Different roles of eukaryotic MutS and MutL complexes in repair of small insertion and deletion loops in yeast.A novel function of DNA polymerase zeta regulated by PCNA.The N terminus of Saccharomyces cerevisiae Msh6 is an unstructured tether to PCNAA mutation in EXO1 defines separable roles in DNA mismatch repair and post-replication repair.Palm mutants in DNA polymerases alpha and eta alter DNA replication fidelity and translesion activity.Mutations that increase DNA binding by the processivity factor of herpes simplex virus affect virus production and DNA replication fidelity.DNA polymerase delta, RFC and PCNA are required for repair synthesis of large looped heteroduplexes in Saccharomyces cerevisiae.Ntg2p, a Saccharomyces cerevisiae DNA N-glycosylase/apurinic or apyrimidinic lyase involved in base excision repair of oxidative DNA damage, interacts with the DNA mismatch repair protein Mlh1p. Identification of a Mlh1p binding motif.PCNA dependent cellular activities tolerate dramatic perturbations in PCNA client interactions.Increased rates of genomic deletions generated by mutations in the yeast gene encoding DNA polymerase delta or by decreases in the cellular levels of DNA polymerase delta
P2860
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P2860
Saccharomyces cerevisiae pol30 (proliferating cell nuclear antigen) mutations impair replication fidelity and mismatch repair.
description
1999 nî lūn-bûn
@nan
1999年の論文
@ja
1999年学术文章
@wuu
1999年学术文章
@zh-cn
1999年学术文章
@zh-hans
1999年学术文章
@zh-my
1999年学术文章
@zh-sg
1999年學術文章
@yue
1999年學術文章
@zh
1999年學術文章
@zh-hant
name
Saccharomyces cerevisiae pol30 ...... fidelity and mismatch repair.
@en
Saccharomyces cerevisiae pol30
@nl
type
label
Saccharomyces cerevisiae pol30 ...... fidelity and mismatch repair.
@en
Saccharomyces cerevisiae pol30
@nl
prefLabel
Saccharomyces cerevisiae pol30 ...... fidelity and mismatch repair.
@en
Saccharomyces cerevisiae pol30
@nl
P2093
P2860
P356
P1476
Saccharomyces cerevisiae pol30 ...... fidelity and mismatch repair.
@en
P2093
P2860
P304
P356
10.1128/MCB.19.11.7801
P407
P577
1999-11-01T00:00:00Z