Lipid peroxidation dominates the chemistry of DNA adduct formation in a mouse model of inflammation.
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Structure of the 1, N 2 -Etheno-2′-deoxyguanosine Lesion in the 3′-G(εdG)T-5′ Sequence Opposite a One-Base DeletionDefects in purine nucleotide metabolism lead to substantial incorporation of xanthine and hypoxanthine into DNA and RNADifferential effects of reactive nitrogen species on DNA base excision repair initiated by the alkyladenine DNA glycosylaseMacrophage diversity enhances tumor progression and metastasisQuantification of DNA damage products resulting from deamination, oxidation and reaction with products of lipid peroxidation by liquid chromatography isotope dilution tandem mass spectrometry.Column switching HPLC-ESI(+)-MS/MS methods for quantitative analysis of exocyclic dA adducts in the DNA of laboratory animals exposed to 1,3-butadiene.Targeting reactive nitrogen species suppresses hereditary pancreatic cancer.Proteins modified by the lipid peroxidation aldehyde 9,12-dioxo-10(E)-dodecenoic acid in MCF7 breast cancer cells.Repair of endogenous DNA base lesions modulate lifespan in miceIntegrated molecular analysis indicates undetectable change in DNA damage in mice after continuous irradiation at ~ 400-fold natural background radiation.Mutagenic potency of Helicobacter pylori in the gastric mucosa of mice is determined by sex and duration of infection.The biological and metabolic fates of endogenous DNA damage productsNontargeted nucleotide analysis based on benzoylhistamine labeling-MALDI-TOF/TOF-MS: discovery of putative 6-oxo-thymine in DNAInflammation-mediated genetic and epigenetic alterations drive cancer development in the neighboring epithelium upon stromal abrogation of TGF-β signaling.An assay for RNA oxidation induced abasic sites using the Aldehyde Reactive ProbeSimultaneous detection of multiple DNA adducts in human lung samples by isotope-dilution UPLC-MS/MSChemistry meets biology in colitis-associated carcinogenesisRNA oxidation catalyzed by cytochrome c leads to its depurination and cross-linking, which may facilitate cytochrome c release from mitochondria.A filtered database search algorithm for endogenous serum protein carbonyl modifications in a mouse model of inflammation.A critical role for macrophages in promotion of urethane-induced lung carcinogenesisReactive species and DNA damage in chronic inflammation: reconciling chemical mechanisms and biological fates.Inflammation and cancer: chemical approaches to mechanisms, imaging, and treatment.DNA repair is indispensable for survival after acute inflammation.Infection-induced colitis in mice causes dynamic and tissue-specific changes in stress response and DNA damage leading to colon cancer.Increased tRNA modification and gene-specific codon usage regulate cell cycle progression during the DNA damage responseQuantitation of DNA adducts by stable isotope dilution mass spectrometry.AlkB homologue 2-mediated repair of ethenoadenine lesions in mammalian DNA.DNA damage induced by chronic inflammation contributes to colon carcinogenesis in miceMass spectrometry of structurally modified DNA.Increased levels of inosine in a mouse model of inflammationContinuous elimination of oxidized nucleotides is necessary to prevent rapid onset of cellular senescence.Nitric oxide and TNF-alpha trigger colonic inflammation and carcinogenesis in Helicobacter hepaticus-infected, Rag2-deficient miceSteric and electrostatic effects at the C2 atom substituent influence replication and miscoding of the DNA deamination product deoxyxanthosine and analogs by DNA polymerases.Molecular mechanism of recombinant liver fatty acid binding protein's antioxidant activity.Gut microbes define liver cancer risk in mice exposed to chemical and viral transgenic hepatocarcinogens.Radiation metabolomics. 2. Dose- and time-dependent urinary excretion of deaminated purines and pyrimidines after sublethal gamma-radiation exposure in mice.Endonuclease and Exonuclease Activities on Oligodeoxynucleotides Containing Spiroiminodihydantoin Depend on the Sequence Context and the Lesion Stereochemistry.Damage of DNA and proteins by major lipid peroxidation products in genome stability.Redox regulation of inflammation: old elements, a new story.DNA Product Formation in Female Sprague-Dawley Rats Following Polyhalogenated Aromatic Hydrocarbon (PHAH) Exposure.
P2860
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P2860
Lipid peroxidation dominates the chemistry of DNA adduct formation in a mouse model of inflammation.
description
2007 nî lūn-bûn
@nan
2007年の論文
@ja
2007年学术文章
@wuu
2007年学术文章
@zh-cn
2007年学术文章
@zh-hans
2007年学术文章
@zh-my
2007年学术文章
@zh-sg
2007年學術文章
@yue
2007年學術文章
@zh
2007年學術文章
@zh-hant
name
Lipid peroxidation dominates t ...... a mouse model of inflammation.
@en
type
label
Lipid peroxidation dominates t ...... a mouse model of inflammation.
@en
prefLabel
Lipid peroxidation dominates t ...... a mouse model of inflammation.
@en
P2093
P356
P1433
P1476
Lipid peroxidation dominates t ...... a mouse model of inflammation.
@en
P2093
Hongbin Yu
John S Wishnok
Koli Taghizadeh
Steven R Tannenbaum
Xinfeng Zhou
P304
P356
10.1093/CARCIN/BGM037
P407
P577
2007-03-07T00:00:00Z