Commonly dysregulated genes in murine APL cells. - Wikidata - wikimedia - TriplyDB

Commonly dysregulated genes in murine APL cells.

Commonly dysregulated genes in murine APL cells.

http://www.wikidata.org/entity/Q42114434

about

High throughput digital quantification of mRNA abundance in primary human acute myeloid leukemia samples High levels of the adhesion molecule CD44 on leukemic cells generate acute myeloid leukemia relapse after withdrawal of the initial transforming event Clara cells attenuate the inflammatory response through regulation of macrophage behavior Effects of the NUP98-DDX10 oncogene on primary human CD34+ cells: role of a conserved helicase motif Specification of the NF-kappaB transcriptional response by p65 phosphorylation and TNF-induced nuclear translocation of IKK epsilon.A protease-resistant PML-RAR{alpha} has increased leukemogenic potential in a murine model of acute promyelocytic leukemia Expression and function of PML-RARA in the hematopoietic progenitor cells of Ctsg-PML-RARA mice PML-RARA can increase hematopoietic self-renewal without causing a myeloproliferative disease in mice.Rara haploinsufficiency modestly influences the phenotype of acute promyelocytic leukemia in mice.Somatic mutations and germline sequence variants in the expressed tyrosine kinase genes of patients with de novo acute myeloid leukemia.A decade of genome-wide gene expression profiling in acute myeloid leukemia: flashback and prospects.Chemosensitization of acute myeloid leukemia (AML) following mobilization by the CXCR4 antagonist AMD3100.Notch signaling in acute promyelocytic leukemia Applications of microarray technology to Acute Myelogenous Leukemia The PML-Interacting Protein DAXX: Histone Loading Gets into the Picture.Computational identification of the normal and perturbed genetic networks involved in myeloid differentiation and acute promyelocytic leukemia.Interaction with RXR is necessary for NPM-RAR-induced myeloid differentiation blockade.

P2860

Q24652901-90AB4544-235E-458D-942F-F59958105C3D Q28299462-A53535B0-AFC3-44BC-B6ED-33F5C061E821 Q33658797-BB7385C3-3F06-4771-9171-00310DD6716C Q33846983-0884E430-FD94-4F39-9CD6-BA1E1E37C4ED Q34189309-6ABD8944-1A30-409D-88BA-239798DA431A Q34310547-810644CF-9ACF-45C7-9E9A-81A84179B1E0 Q34441594-55A41CFE-B75B-4F80-A094-F41BC9C27876 Q34749039-21646CA3-0E54-4B95-BAC6-081C46A8CCA8 Q35866178-E90E3A09-E653-485C-B212-2829D86F0E54 Q36591394-92E54AD8-E17E-4AF9-A657-1A22D9192815 Q37041909-61B0A20E-7A3C-4813-A7F5-A8D00E6A2E78 Q37234638-2E4B1F02-7A9C-4BA7-AF3B-375524A4D1F9 Q37414175-DBB95719-268A-4272-92AC-3F311E314001 Q37437233-B26173F4-1E2E-49C6-9006-F5477DD701B7 Q38113966-02CD6E68-02B6-4281-9152-0C204A0146A1 Q38275870-5C1F2AF0-5DAC-4727-8436-671B408624CD Q42730910-5F0B4DED-5DF0-4DEE-B436-6979386362F8

P2860

Commonly dysregulated genes in murine APL cells.

http://www.wikidata.org/entity/Q42114434

description

2006 nî lūn-bûn

@nan

2006年の論文

@ja

2006年論文

@yue

2006年論文

@zh-hant

2006年論文

@zh-hk

2006年論文

@zh-mo

2006年論文

@zh-tw

2006年论文

@wuu

2006年论文

@zh

2006年论文

@zh-cn

name

Commonly dysregulated genes in murine APL cells.

@en

Commonly dysregulated genes in murine APL cells.

@nl

type

label

Commonly dysregulated genes in murine APL cells.

@en

Commonly dysregulated genes in murine APL cells.

@nl

prefLabel

Commonly dysregulated genes in murine APL cells.

@en

Commonly dysregulated genes in murine APL cells.

@nl

P1433

Q42114434-EF518885-89DF-4E16-A8F5-FE5C9571DE5E

P1476

Q42114434-1DFF6C68-7D65-4F14-9A67-18E5D1F47729

P2093

Q42114434-1F34B141-4B5E-48FE-93CB-9BEFEC1BA5C7

Q42114434-30E20298-9099-4770-AB6F-215372626B00

Q42114434-349CBBE1-B64C-48C0-8245-53A905860101

Q42114434-5E5C3689-32E3-4800-828E-0FBFF767B104

Q42114434-6957C42F-9EA1-4D21-8783-3DC951B1AE6C

Q42114434-CDDB68E9-506A-4C7A-803B-E29836D90834

Q42114434-F8BB41C5-DA61-45CB-B7A1-CEF286C46715

P2860

Q42114434-022A0B09-6CFC-4777-A5D1-7791FAC01CF0

Q42114434-075A1835-EAC2-4CD9-B137-8660D47A4A44

Q42114434-081339F0-2F4A-4B3F-B958-C4BD661E0782

Q42114434-0C3B848C-D463-4EF7-94BB-830665E7C69C

Q42114434-12E9E6BB-4F22-41F3-9653-754E7AF3CE98

Q42114434-1976F585-02B7-4D4E-9942-39EDF59E0B43

Q42114434-1F819872-6DF7-45A8-B969-8EED35F73056

Q42114434-2260723F-FF1C-4C3F-B463-05E04CDA56AF

Q42114434-2548D0A5-89C8-4C82-9B93-8C87AABEE31A

Q42114434-3105AF8A-761A-4571-A784-DB41CD4F9F98

P304

Q42114434-2738AB92-4C94-4C9D-952E-AF3FD2DC998F

P31

Q42114434-A631322B-88E5-4F81-9AF4-9515078FD7B0

P356

Q42114434-256DCACF-CE7E-4FEA-B3EB-742BA7F63156

P407

Q42114434-5B7C9509-5E85-406E-AB9C-A3D4B2C7CE51

P433

Q42114434-D24C89F7-D73D-43B9-BB88-CA67643DF622

P478

Q42114434-9CE9026E-6CE3-424E-B5B2-E2A39443D895

P577

Q42114434-E4977914-1ECF-486B-9BF9-880469C38F6F

P5875

Q42114434-BBC5CEC1-B87A-4FF2-B1F7-DF9A50876E92

P698

Q42114434-FAEBDB2C-80E2-4F6D-A855-8280E1A27BC8

P932

Q42114434-E1691FB3-D666-4E0A-8CCC-612A6CF9C68A

P356

BLOOD-2006-07-036640

P1433

P1476

Commonly dysregulated genes in murine APL cells.

@en

P2093

Daniel C Link

http://www.w3.org/2001/XMLSchema#string

Jacqueline E Payton

http://www.w3.org/2001/XMLSchema#string

John F DiPersio

http://www.w3.org/2001/XMLSchema#string

Mark A Watson

http://www.w3.org/2001/XMLSchema#string

Matthew S Holt

http://www.w3.org/2001/XMLSchema#string

Timothy J Ley

http://www.w3.org/2001/XMLSchema#string

Wenlin Yuan

http://www.w3.org/2001/XMLSchema#string

P2860

Core transcriptional regulatory circuitry in human embryonic stem cells

Unregulated activation of STAT-5, ERK1/2 and c-Fos may contribute to the phenotypic transformation from myelodysplastic syndrome to acute leukaemia

Concordant loss of MTAP and p16/CDKN2A expression in pancreatic intraepithelial neoplasia: evidence of homozygous deletion in a noninvasive precursor lesion

Loss of methylthioadenosine phosphorylase and elevated ornithine decarboxylase is common in pancreatic cancer

Homozygous deletion of the MTAP gene in invasive adenocarcinoma of the pancreas and in periampullary cancer: a potential new target for therapy

Early growth response gene 1 stimulates development of hematopoietic progenitor cells along the macrophage lineage at the expense of the granulocyte and erythroid lineages.

Neutrophil elastase is important for PML-retinoic acid receptor alpha activities in early myeloid cells.

Reduced PU.1 expression causes myeloid progenitor expansion and increased leukemia penetrance in mice expressing PML-RARalpha.

PML/RARalpha and FLT3-ITD induce an APL-like disease in a mouse model

Acute myeloid leukemias with reciprocal rearrangements can be distinguished by specific gene expression profiles

P304

961-970

http://www.w3.org/2001/XMLSchema#string

P31

scholarly article

P356

10.1182/BLOOD-2006-07-036640

http://www.w3.org/2001/XMLSchema#string

P407

P433

3

http://www.w3.org/2001/XMLSchema#string

P478

109

http://www.w3.org/2001/XMLSchema#string

P577

2006-09-28T00:00:00Z

http://www.w3.org/2001/XMLSchema#dateTime

P5875

6785349

http://www.w3.org/2001/XMLSchema#string

P698

17008535

http://www.w3.org/2001/XMLSchema#string

P932

1785140

http://www.w3.org/2001/XMLSchema#string