Resistance to EGFR-TKI can be mediated through multiple signaling pathways converging upon cap-dependent translation in EGFR-wild type NSCLC.
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Pim-1 kinase is a target of miR-486-5p and eukaryotic translation initiation factor 4E, and plays a critical role in lung cancer.EGFR inhibitors and autophagy in cancer treatment.Metabolic glycoengineering sensitizes drug-resistant pancreatic cancer cells to tyrosine kinase inhibitors erlotinib and gefitinib.Targeting EIF4F complex in non-small cell lung cancer cells.NRF2 Promotes Tumor Maintenance by Modulating mRNA Translation in Pancreatic Cancer.
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Resistance to EGFR-TKI can be mediated through multiple signaling pathways converging upon cap-dependent translation in EGFR-wild type NSCLC.
description
2013 nî lūn-bûn
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2013年の論文
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2013年論文
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2013年論文
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2013年論文
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2013年論文
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2013年論文
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2013年论文
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2013年论文
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name
Resistance to EGFR-TKI can be ...... ation in EGFR-wild type NSCLC.
@en
Resistance to EGFR-TKI can be ...... ation in EGFR-wild type NSCLC.
@nl
type
label
Resistance to EGFR-TKI can be ...... ation in EGFR-wild type NSCLC.
@en
Resistance to EGFR-TKI can be ...... ation in EGFR-wild type NSCLC.
@nl
prefLabel
Resistance to EGFR-TKI can be ...... ation in EGFR-wild type NSCLC.
@en
Resistance to EGFR-TKI can be ...... ation in EGFR-wild type NSCLC.
@nl
P2093
P2860
P1476
Resistance to EGFR-TKI can be ...... lation in EGFR-wild type NSCLC
@en
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Ahad A Sadiq
Blake A Jacobson
Joe Jay-Dixon
Robert A Kratzke
P2860
P304
P356
10.1097/JTO.0B013E31829CE963
P577
2013-09-01T00:00:00Z