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Ionic homeostasis in brain conditioningThe antiepileptic drug levetiracetam suppresses non-convulsive seizure activity and reduces ischemic brain damage in rats subjected to permanent middle cerebral artery occlusionProteolysis of AKAP121 regulates mitochondrial activity during cellular hypoxia and brain ischaemiaAntithrombin reduces ischemic volume, ameliorates neurologic deficits, and prolongs animal survival in both transient and permanent focal ischemia.Proteolytic control of neurite outgrowth inhibitor NOGO-A by the cAMP/PKA pathway.MicroRNA-103-1 selectively downregulates brain NCX1 and its inhibition by anti-miRNA ameliorates stroke damage and neurological deficits.ncx1, ncx2, and ncx3 gene product expression and function in neuronal anoxia and brain ischemia.Pharmacological characterization of the newly synthesized 5-amino-N-butyl-2-(4-ethoxyphenoxy)-benzamide hydrochloride (BED) as a potent NCX3 inhibitor that worsens anoxic injury in cortical neurons, organotypic hippocampal cultures, and ischemic braNCX1 Exchanger Cooperates with Calretinin to Confer Preconditioning-Induced Tolerance Against Cerebral Ischemia in the Striatum.A new cell-penetrating peptide that blocks the autoinhibitory XIP domain of NCX1 and enhances antiporter activity.The NCX3 isoform of the Na+/Ca2+ exchanger contributes to neuroprotection elicited by ischemic postconditioning.Ischemic tolerance modulates TRAIL expression and its receptors and generates a neuroprotected phenotype.Oligosaccharidic fractions derived from Triticum vulgare extract accelerate tissutal repairing processes in in vitro and in vivo models of skin lesions.Targeted acetylation of NF-kappaB/RelA and histones by epigenetic drugs reduces post-ischemic brain injury in mice with an extended therapeutic window.Neurounina-1, a novel compound that increases Na+/Ca2+ exchanger activity, effectively protects against stroke damage.Two sodium/calcium exchanger gene products, NCX1 and NCX3, play a major role in the development of permanent focal cerebral ischemia.A critical role for the potassium-dependent sodium-calcium exchanger NCKX2 in protection against focal ischemic brain damage.Sp3/REST/HDAC1/HDAC2 Complex Represses and Sp1/HIF-1/p300 Complex Activates ncx1 Gene Transcription, in Brain Ischemia and in Ischemic Brain Preconditioning, by Epigenetic Mechanism.Involvement of the potassium-dependent sodium/calcium exchanger gene product NCKX2 in the brain insult induced by permanent focal cerebral ischemia.NCX1 and NCX3: two new effectors of delayed preconditioning in brain ischemia.Targeted disruption of Na+/Ca2+ exchanger 3 (NCX3) gene leads to a worsening of ischemic brain damage.Triticum vulgare extract exerts an anti-inflammatory action in two in vitro models of inflammation in microglial cells.
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description
researcher
@en
wetenschapper
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հետազոտող
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name
Ornella Cuomo
@ast
Ornella Cuomo
@en
Ornella Cuomo
@es
Ornella Cuomo
@nl
type
label
Ornella Cuomo
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Ornella Cuomo
@en
Ornella Cuomo
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Ornella Cuomo
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prefLabel
Ornella Cuomo
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Ornella Cuomo
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Ornella Cuomo
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Ornella Cuomo
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P106
P1153
23008146000
P31
P496
0000-0002-6459-2530