Skeletal muscle dysfunction in muscle-specific LKB1 knockout mice. - Wikidata - awgldk - TriplyDB

Skeletal muscle dysfunction in muscle-specific LKB1 knockout mice.

Skeletal muscle dysfunction in muscle-specific LKB1 knockout mice.

http://www.wikidata.org/entity/Q30494897

about

Two weeks of metformin treatment enhances mitochondrial respiration in skeletal muscle of AMPK kinase dead but not wild type mice Regulation of (pro)renin receptor expression in mIMCD via the GSK-3β-NFAT5-SIRT-1 signaling pathway.The effects of age and muscle contraction on AMPK activity and heterotrimer composition.Chronic AMP-activated protein kinase activation and a high-fat diet have an additive effect on mitochondria in rat skeletal muscle LKB1 destabilizes microtubules in myoblasts and contributes to myoblast differentiation Whole body deletion of AMP-activated protein kinase {beta}2 reduces muscle AMPK activity and exercise capacity.Rev-erb-α modulates skeletal muscle oxidative capacity by regulating mitochondrial biogenesis and autophagy Effect of LKB1 deficiency on mitochondrial content, fibre type and muscle performance in the mouse diaphragm.Characterization of the liver kinase B1-mouse protein-25 -Ste-20-related adaptor protein complex in adult mouse skeletal muscle Inactivation of MARK4, an AMP-activated protein kinase (AMPK)-related kinase, leads to insulin hypersensitivity and resistance to diet-induced obesity Deletion of Lkb1 in Renal Tubular Epithelial Cells Leads to CKD by Altering Metabolism.Liver kinase B1 inhibits the expression of inflammation-related genes postcontraction in skeletal muscle.Mitochondrial and performance adaptations to exercise training in mice lacking skeletal muscle LKB1 Muscle-specific AMPK β1β2-null mice display a myopathy due to loss of capillary density in nonpostural muscles.LKB1 signaling in advancing cell differentiation.Lkb1 regulation of skeletal muscle development, metabolism and muscle progenitor cell homeostasis.Deletion of Lkb1 in adult mice results in body weight reduction and lethality.LKB1 deletion causes early changes in atrial channel expression and electrophysiology prior to atrial fibrillation.Lkb1 is indispensable for skeletal muscle development, regeneration, and satellite cell homeostasis LKB1-regulated adaptive mechanisms are essential for neuronal survival following mitochondrial dysfunction.AMPK in skeletal muscle function and metabolism.Dexamethasone promotes long-term functional recovery of neuromuscular junction in a murine model of tourniquet-induced ischaemia-reperfusion.Adipocyte-specific deletion of Lkb1 and mTOR protects against high-fat diet induced obesity but results in insulin resistance.CXCL10 increases in human skeletal muscle following damage but is not necessary for muscle regeneration.Preclinical characterization of the JAK/STAT inhibitor SGI-1252 on skeletal muscle function, morphology, and satellite cell content.

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Skeletal muscle dysfunction in muscle-specific LKB1 knockout mice.

http://www.wikidata.org/entity/Q30494897

description

2010 nî lūn-bûn

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2010 թուականի Ապրիլին հրատարակուած գիտական յօդուած

@hyw

2010 թվականի ապրիլին հրատարակված գիտական հոդված

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2010年の論文

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2010年論文

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2010年論文

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2010年論文

@zh-hk

2010年論文

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2010年論文

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2010年论文

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name

Skeletal muscle dysfunction in muscle-specific LKB1 knockout mice.

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Skeletal muscle dysfunction in muscle-specific LKB1 knockout mice.

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type

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Skeletal muscle dysfunction in muscle-specific LKB1 knockout mice.

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Skeletal muscle dysfunction in muscle-specific LKB1 knockout mice.

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Skeletal muscle dysfunction in muscle-specific LKB1 knockout mice.

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Skeletal muscle dysfunction in muscle-specific LKB1 knockout mice.

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JAPPLPHYSIOL.01293.2009

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Journal of Applied Physiology

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Skeletal muscle dysfunction in muscle-specific LKB1 knockout mice.

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Allen C Parcell

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Anthony D Mongillo

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Bradley G Evanson

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Brandon B Malan

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Chad R Hancock

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David L Kooyman

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David M Thomson

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Jacob D Brown

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Natasha Fillmore

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Squire Hepworth

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P2860

AMP-activated protein kinase regulates GLUT4 transcription by phosphorylating histone deacetylase 5

LKB1 is a master kinase that activates 13 kinases of the AMPK subfamily, including MARK/PAR-1

Skeletal muscle-selective knockout of LKB1 increases insulin sensitivity, improves glucose homeostasis, and decreases TRB3

A serine/threonine kinase gene defective in Peutz-Jeghers syndrome

AMP-activated protein kinase suppresses protein synthesis in rat skeletal muscle through down-regulated mammalian target of rapamycin (mTOR) signaling

Chronic paraplegia-induced muscle atrophy downregulates the mTOR/S6K1 signaling pathway

SIK1 is a class II HDAC kinase that promotes survival of skeletal myocytes

Vascular abnormalities and deregulation of VEGF in Lkb1-deficient mice

Insulin signalling to mTOR mediated by the Akt/PKB substrate PRAS40

Muscle inactivation of mTOR causes metabolic and dystrophin defects leading to severe myopathy.

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