The murine N-ras gene is not essential for growth and development. - Wikidata - awgldk - TriplyDB

The murine N-ras gene is not essential for growth and development.

The murine N-ras gene is not essential for growth and development.

http://www.wikidata.org/entity/Q34650223

about

K-Ras4B phosphorylation at Ser181 is inhibited by calmodulin and modulates K-Ras activity and function R-Ras3/M-Ras induces neuronal differentiation of PC12 cells through cell-type-specific activation of the mitogen-activated protein kinase cascade.A vascular gene trap screen defines RasGRP3 as an angiogenesis-regulated gene required for the endothelial response to phorbol esters K-ras/PI3K-Akt signaling is essential for zebrafish hematopoiesis and angiogenesis Calmodulin binds to K-Ras, but not to H- or N-Ras, and modulates its downstream signaling H-Ras and phosphoinositide 3-kinase cooperate to induce alpha(1,3)-fucosyltransferase VII expression in Jurkat T cells H-Ras signaling and K-Ras signaling are differentially dependent on endocytosis.Suprachiasmatic nucleus circadian oscillatory protein, a novel binding partner of K-Ras in the membrane rafts, negatively regulates MAPK pathway Involvement of a small GTP-binding protein (G protein) regulator, small G protein GDP dissociation stimulator, in antiapoptotic cell survival signaling Ras oncogenes: split personalities MASL1 induces erythroid differentiation in human erythropoietin-dependent CD34+ cells through the Raf/MEK/ERK pathway Ras history: The saga continues.A comprehensive survey of Ras mutations in cancer.Coordinated traffic of Grb2 and Ras during epidermal growth factor receptor endocytosis visualized in living cells.Analysis of k-ras nuclear expression in fibroblasts and mesangial cells Activated Ras alters lens and corneal development through induction of distinct downstream targets.Minireview: physiological and pathological actions of RAS in the ovary.Ras membrane orientation and nanodomain localization generate isoform diversity Elevated phosphate activates N-ras and promotes cell transformation and skin tumorigenesis.Krüppel-like factor 5 is a crucial mediator of intestinal tumorigenesis in mice harboring combined ApcMin and KRASV12 mutations.Control of B cell development by Ras-mediated activation of Raf.R-Ras3, a brain-specific Ras-related protein, activates Akt and promotes cell survival in PC12 cells.Targeted genomic disruption of H-ras and N-ras, individually or in combination, reveals the dispensability of both loci for mouse growth and development Identification of K-ras as the major regulator for cytokine-dependent Akt activation in erythroid progenitors in vivo.Replacement of K-Ras with H-Ras supports normal embryonic development despite inducing cardiovascular pathology in adult mice.A Ras by any other name.BCR-ABL but not JAK2 V617F inhibits erythropoiesis through the Ras signal by inducing p21CIP1/WAF1.The cyclopentenone 15-deoxy-delta 12,14-prostaglandin J2 binds to and activates H-Ras.A plausible function of the prion protein: conjectures and a hypothesis.Structure-energy-based predictions and network modelling of RASopathy and cancer missense mutations Cellular N-Ras promotes cell survival by downregulation of Jun N-terminal protein kinase and p38 Nras overexpression results in granulocytosis, T-cell expansion and early lethality in mice.Ras isoform-specific signaling: location, location, location.Preclinical efficacy of MEK inhibition in Nras-mutant AML.In TCR-stimulated T-cells, N-ras regulates specific genes and signal transduction pathways Serum-dependent transcriptional networks identify distinct functional roles for H-Ras and N-Ras during initial stages of the cell cycle While K-ras is essential for mouse development, expression of the K-ras 4A splice variant is dispensable.Reversible, interrelated mRNA and miRNA expression patterns in the transcriptome of Rasless fibroblasts: functional and mechanistic implications Rb and N-ras function together to control differentiation in the mouse.K-ras is an essential gene in the mouse with partial functional overlap with N-ras.

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P2860

The murine N-ras gene is not essential for growth and development.

http://www.wikidata.org/entity/Q34650223

description

1995 nî lūn-bûn

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1995 թուականի Փետրուարին հրատարակուած գիտական յօդուած

@hyw

1995 թվականի փետրվարին հրատարակված գիտական հոդված

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1995年の論文

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1995年論文

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1995年論文

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1995年論文

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1995年論文

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1995年論文

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1995年论文

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name

The murine N-ras gene is not essential for growth and development.

@ast

The murine N-ras gene is not essential for growth and development.

@en

The murine N-ras gene is not essential for growth and development.

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type

label

The murine N-ras gene is not essential for growth and development.

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The murine N-ras gene is not essential for growth and development.

@en

The murine N-ras gene is not essential for growth and development.

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prefLabel

The murine N-ras gene is not essential for growth and development.

@ast

The murine N-ras gene is not essential for growth and development.

@en

The murine N-ras gene is not essential for growth and development.

@nl

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P1433

Proceedings of the National Academy of Sciences of the United States of America

P1476

The murine N-ras gene is not essential for growth and development.

@en

P2093

A Pellicer

http://www.w3.org/2001/XMLSchema#string

H Umanoff

http://www.w3.org/2001/XMLSchema#string

R Kucherlapati

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W Edelmann

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P2860

The GTPase superfamily: conserved structure and molecular mechanism

Genetic analysis of yeast RAS1 and RAS2 genes.

Requirement for ras proto-oncogene function during serum-stimulated growth of NIH 3T3 cells

Disruption of the proto-oncogene int-2 in mouse embryo-derived stem cells: a general strategy for targeting mutations to non-selectable genes

Site-directed mutagenesis by gene targeting in mouse embryo-derived stem cells

Identification of ras oncogene mutations in the stool of patients with curable colorectal tumors.

A molecular approach to leukemogenesis: mouse lymphomas contain an activated c-ras oncogene

Differential expression of the ras gene family in mice.

Mutational analysis of a ras catalytic domain.

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1709-1713

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10.1073/PNAS.92.5.1709

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5

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92

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1995-02-01T00:00:00Z

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15321273

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7878045

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42589

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