Brain trauma induces massive hippocampal neuron death linked to a surge in beta-amyloid levels in mice overexpressing mutant amyloid precursor protein. - Wikidata - awgldk - TriplyDB

Brain trauma induces massive hippocampal neuron death linked to a surge in beta-amyloid levels in mice overexpressing mutant amyloid precursor protein.

Brain trauma induces massive hippocampal neuron death linked to a surge in beta-amyloid levels in mice overexpressing mutant amyloid precursor protein.

http://www.wikidata.org/entity/Q35753360

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Chronic Traumatic Encephalopathy in Athletes: Progressive Tauopathy After Repetitive Head Injury Active and passive immunotherapy for neurodegenerative disorders The Neuroprotective Properties of the Amyloid Precursor Protein Following Traumatic Brain Injury Animal models of traumatic brain injury Effects of low-level blast exposure on the nervous system: is there really a controversy?Chronic traumatic encephalopathy: clinical-biomarker correlations and current concepts in pathogenesis.Cerebral blood flow changes after brain injury in human amyloid-beta knock-in mice Acute blast injury reduces brain abeta in two rodent species.High-throughput quantitative histological analysis of Alzheimer's disease pathology using a confocal digital microscanner.Recovery of neurological function despite immediate sleep disruption following diffuse brain injury in the mouse: clinical relevance to medically untreated concussion Genetic vulnerability following traumatic brain injury: the role of apolipoprotein E.Traumatic brain injury: cause or risk of Alzheimer's disease? A review of experimental studies.Recent advances in neurotrauma.Genetic deletion and pharmacological inhibition of Nogo-66 receptor impairs cognitive outcome after traumatic brain injury in mice.Gene-environment interaction research and transgenic mouse models of Alzheimer's disease.Traumatic brain injury reduces soluble extracellular amyloid-β in mice: a methodologically novel combined microdialysis-controlled cortical impact study.Combination therapy of human umbilical cord blood cells and granulocyte colony stimulating factor reduces histopathological and motor impairments in an experimental model of chronic traumatic brain injury.A review and rationale for the use of genetically engineered animals in the study of traumatic brain injury.Modulation of ABCA1 by an LXR agonist reduces β-amyloid levels and improves outcome after traumatic brain injury Potential intervention by vaccinia virus complement control protein of the signals contributing to the progression of central nervous system injury to Alzheimer's disease.Age-related amyloid beta deposition in transgenic mice overexpressing both Alzheimer mutant presenilin 1 and amyloid beta precursor protein Swedish mutant is not associated with global neuronal loss.Morris water maze search strategy analysis in PDAPP mice before and after experimental traumatic brain injury.Lithium reduces BACE1 overexpression, β amyloid accumulation, and spatial learning deficits in mice with traumatic brain injury.Animal models of head trauma Electromagnetic controlled cortical impact device for precise, graded experimental traumatic brain injury Neuroprotective effects of curcumin.Amyloid-β Peptides and Tau Protein as Biomarkers in Cerebrospinal and Interstitial Fluid Following Traumatic Brain Injury: A Review of Experimental and Clinical Studies.Brain injury, neuroinflammation and Alzheimer's disease.Altered Neuroinflammation and Behavior after Traumatic Brain Injury in a Mouse Model of Alzheimer's Disease.Functional outcome is impaired following traumatic brain injury in aging Nogo-A/B-deficient mice.Head trauma and in vivo measures of amyloid and neurodegeneration in a population-based study.Experimental traumatic brain injury induces rapid aggregation and oligomerization of amyloid-beta in an Alzheimer's disease mouse model.Traumatic brain injury and amyloid-β pathology: a link to Alzheimer's disease?The pathophysiology of concussions in youth.Traumatic Brain Injury, Chronic Traumatic Encephalopathy, and Alzheimer's Disease: Common Pathologies Potentiated by Altered Zinc Homeostasis.Disordered APP metabolism and neurovasculature in trauma and aging: Combined risks for chronic neurodegenerative disorders.Controlled cortical impact traumatic brain injury in 3xTg-AD mice causes acute intra-axonal amyloid-β accumulation and independently accelerates the development of tau abnormalities Amyloid-beta dynamics correlate with neurological status in the injured human brain Commentary on "a roadmap for the prevention of dementia II. Leon Thal Symposium 2008." Rationale and recommendations for first evaluating anti-Alzheimer's disease medications in acute brain injury patients.Traumatic brain injuries.

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P2860

Brain trauma induces massive hippocampal neuron death linked to a surge in beta-amyloid levels in mice overexpressing mutant amyloid precursor protein.

http://www.wikidata.org/entity/Q35753360

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Brain trauma induces massive h ...... ant amyloid precursor protein.

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Brain trauma induces massive h ...... ant amyloid precursor protein.

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The American Journal of Pathology

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Correlative Memory Deficits, Abeta Elevation, and Amyloid Plaques in Transgenic Mice

The occult aftermath of boxing

Molecular features of hypothalamic plaques in Alzheimer's disease.

Amyloid precursor protein processing and A beta42 deposition in a transgenic mouse model of Alzheimer disease.

Amyloids beta40 and beta42 are generated intracellularly in cultured human neurons and their secretion increases with maturation.

Re-examination of ex-boxers' brains using immunohistochemistry with antibodies to amyloid beta-protein and tau protein.

Visualization of A beta 42(43) and A beta 40 in senile plaques with end-specific A beta monoclonals: evidence that an initially deposited species is A beta 42(43).

An extensive network of PHF tau-rich dystrophic neurites permeates neocortex and nearly all neuritic and diffuse amyloid plaques in Alzheimer disease.

Early detection of axonal injury after human head trauma using immunocytochemistry for beta-amyloid precursor protein.

Lack of Alzheimer pathology after beta-amyloid protein injections in rat brain.

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