Reversible gene knockdown in mice using a tight, inducible shRNA expression system. - Wikidata - awgldk - TriplyDB

Reversible gene knockdown in mice using a tight, inducible shRNA expression system.

Reversible gene knockdown in mice using a tight, inducible shRNA expression system.

http://www.wikidata.org/entity/Q35804666

about

Gamma-secretase activating protein is a therapeutic target for Alzheimer's disease Cardiac and skeletal muscle defects in a mouse model of human Barth syndrome Hematopoietic overexpression of FOG1 does not affect B-cells but reduces the number of circulating eosinophils Reversible suppression of an essential gene in adult mice using transgenic RNA interference Potentiation of insulin-mediated glucose lowering without elevated hypoglycemia risk by a small molecule insulin receptor modulator Characterization of HTT inclusion size, location, and timing in the zQ175 mouse model of Huntington's disease: an in vivo high-content imaging study An inducible RNA interference system for the functional dissection of mouse embryogenesis Inducible transgenic rat model for diabetes mellitus based on shRNA-mediated gene knockdown.Synthetic strategies for studying embryonic development.Inhibition of O-GlcNAcase leads to elevation of O-GlcNAc tau and reduction of tauopathy and cerebrospinal fluid tau in rTg4510 mice Accelerating cancer modeling with RNAi and nongermline genetically engineered mouse models.Genetically engineered mouse models for drug development and preclinical trials.In vivo knockdown of TAK1 accelerates bone marrow proliferation/differentiation and induces systemic inflammation Toxicity modelling of Plk1-targeted therapies in genetically engineered mice and cultured primary mammalian cells.A rapid and scalable system for studying gene function in mice using conditional RNA interference.Yin and Yang of hypothalamic insulin and leptin signaling in regulating white adipose tissue metabolism Conditional brain-specific knockdown of MAPK using Cre/loxP regulated RNA interference.Life in the fast lane: mammalian disease models in the genomics era.Progress on RNAi-based molecular medicines.Tafazzin knockdown in mice leads to a developmental cardiomyopathy with early diastolic dysfunction preceding myocardial noncompaction.The microcosmos of cancer.Central insulin action regulates peripheral glucose and fat metabolism in mice.Site-directed, virus-free, and inducible RNAi in embryonic stem cells Contemporary approaches for modifying the mouse genome RNA interference and antiviral therapy.Inducible and reversible gene silencing by stable integration of an shRNA-encoding lentivirus in transgenic rats.MitoNEET-driven alterations in adipocyte mitochondrial activity reveal a crucial adaptive process that preserves insulin sensitivity in obesity.Hepatic insulin signaling regulates VLDL secretion and atherogenesis in mice.Progress and prospects for genetic modification of nonhuman primate models in biomedical research.Rapid target gene validation in complex cancer mouse models using re-derived embryonic stem cells.The PPAR pan-agonist bezafibrate ameliorates cardiomyopathy in a mouse model of Barth syndrome Modeling human neurodegenerative diseases in transgenic systems.Cancer of mice and men: old twists and new tails.Inducible RNAi system and its application in novel therapeutics.Rapid validation of cancer genes in chimeras derived from established genetically engineered mouse models.Synthetic Tet-inducible small hairpin RNAs targeting hTERT or Bcl-2 inhibit malignant phenotypes of bladder cancer T24 and 5637 cells.Efficient mouse transgenesis using Gateway-compatible ROSA26 locus targeting vectors and F1 hybrid ES cells.Overexpression of miR-210 and its significance in ischemic tissue damage.Individual Variation in Conditional β Cell Ablation Mice Contributes Significant Biases in Evaluating β Cell Functional Recovery.Dose-dependent expression of claudin-5 is a modifying factor in schizophrenia.

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P2860

Reversible gene knockdown in mice using a tight, inducible shRNA expression system.

http://www.wikidata.org/entity/Q35804666

description

2007 nî lūn-bûn

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2007年の論文

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2007年論文

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2007年論文

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2007年論文

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2007年論文

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2007年論文

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2007年论文

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2007年论文

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2007年论文

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name

Reversible gene knockdown in mice using a tight, inducible shRNA expression system.

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Reversible gene knockdown in mice using a tight, inducible shRNA expression system.

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type

label

Reversible gene knockdown in mice using a tight, inducible shRNA expression system.

@ast

Reversible gene knockdown in mice using a tight, inducible shRNA expression system.

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prefLabel

Reversible gene knockdown in mice using a tight, inducible shRNA expression system.

@ast

Reversible gene knockdown in mice using a tight, inducible shRNA expression system.

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Nucleic Acids Research

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Reversible gene knockdown in mice using a tight, inducible shRNA expression system.

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P2093

Birgit Küter-Luks

http://www.w3.org/2001/XMLSchema#string

Frieder Schwenk

http://www.w3.org/2001/XMLSchema#string

Jost Seibler

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Sandra Niehaves

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P2860

Cre-lox-regulated conditional RNA interference from transgenes

Single copy shRNA configuration for ubiquitous gene knockdown in mice

Conditional knockdown of Fgfr2 in mice using Cre-LoxP induced RNA interference

Short hairpin RNAs (shRNAs) induce sequence-specific silencing in mammalian cells

Probing tumor phenotypes using stable and regulated synthetic microRNA precursors

Serial passaging and differentiation of myogenic cells isolated from dystrophic mouse muscle

A lentivirus-based system to functionally silence genes in primary mammalian cells, stem cells and transgenic mice by RNA interference

Efficiency of RNA interference in the mouse hematopoietic system varies between cell types and developmental stages.

'Green mice' as a source of ubiquitous green cells.

Rapid generation of inducible mouse mutants

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e54

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scholarly article

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10.1093/NAR/GKM122

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7

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35

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André Kleinridders

Jens C Brüning

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2007-03-21T00:00:00Z

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6430099

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17376804

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1874634

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