Prevention of nephritis in major histocompatibility complex class II-deficient MRL-lpr mice.
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Role of Natural Autoantibodies and Natural IgM Anti-Leucocyte Autoantibodies in Health and DiseaseLinking susceptibility genes and pathogenesis mechanisms using mouse models of systemic lupus erythematosusThe central and multiple roles of B cells in lupus pathogenesis.Superantigens: mechanisms by which they may induce, exacerbate and control autoimmune diseases.Expression and function of Fas during differentiation and activation of B cells.Advances in lupus stemming from the parent-into-F1 modelDonor CD8 T cell activation is critical for greater renal disease severity in female chronic graft-vs.-host mice and is associated with increased splenic ICOS(hi) host CD4 T cells and IL-21 expressionGenetics of systemic autoimmunity in mouse models of lupus.Systemic autoimmunity and lymphoproliferation are associated with excess IL-7 and inhibited by IL-7Rα blockade.Dendritic cells in lupus are not required for activation of T and B cells but promote their expansion, resulting in tissue damageAutocrine IFNγ controls the regulatory function of lymphoproliferative double negative T cellsFcRγ controls the fas-dependent regulatory function of lymphoproliferative double negative T cellsReducing FLI1 levels in the MRL/lpr lupus mouse model impacts T cell function by modulating glycosphingolipid metabolismHDAC inhibition in lupus models.Tetramers reveal IL-17-secreting CD4+ T cells that are specific for U1-70 in lupus and mixed connective tissue disease.Deficiency of fibroblast growth factor-inducible 14 (Fn14) preserves the filtration barrier and ameliorates lupus nephritis.Host modifier genes affect mouse autoimmunity induced by the lpr geneT cells and B cells in lupus nephritis.B Cell-Specific MHC Class II Deletion Reveals Multiple Nonredundant Roles for B Cell Antigen Presentation in Murine Lupus.Role of MHC-linked susceptibility genes in the pathogenesis of human and murine lupus.Statins: immunomodulators for autoimmune rheumatic disease?The role of B cells in lpr/lpr-induced autoimmunity.Greatly reduced lymphoproliferation in lpr mice lacking major histocompatibility complex class I.Autoantibodies produced spontaneously by young 1pr mice carry transforming growth factor beta and suppress cytotoxic T lymphocyte responses.MRL-lpr/lpr mice exhibit a defect in maintaining developmental arrest and follicular exclusion of anti-double-stranded DNA B cells.Major histocompatibility complex class II expression by intrinsic renal cells is required for crescentic glomerulonephritisIL-21 receptor is required for the systemic accumulation of activated B and T lymphocytes in MRL/MpJ-Fas(lpr/lpr)/J mice.Current perspectives on FTY720.Signals via the adaptor MyD88 in B cells and DCs make distinct and synergistic contributions to immune activation and tissue damage in lupusCTL-promoting effects of CD40 stimulation outweigh B cell-stimulatory effects resulting in B cell elimination and disease improvement in a murine model of lupus.ICOS controls effector function but not trafficking receptor expression of kidney-infiltrating effector T cells in murine lupus.Imbalance towards Th1 predominance is associated with acceleration of lupus-like autoimmune syndrome in MRL mice.CD4+ CD7- CD28- T cells are expanded in rheumatoid arthritis and are characterized by autoreactivity.Roles of interferon-gamma and interleukin-4 in murine lupus.Mouse models of systemic lupus erythematosus reveal a complex pathogenesis.Harnessing regulatory T cells for the therapy of lupus and other autoimmune diseases.Role of major histocompatibility complex class II expression by non-hematopoietic cells in autoimmune and inflammatory disorders: facts and fiction.Activation of diverse repertoires of autoreactive T cells enhances the loss of anti-dsDNA B cell tolerance.Lupus autoimmunity: from peptides to particles.The transmembrane form of TNF-alpha drives autoantibody production in the absence of CD154: studies using MRL/Mp-Fas(lpr) mice.
P2860
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P2860
Prevention of nephritis in major histocompatibility complex class II-deficient MRL-lpr mice.
description
1994 nî lūn-bûn
@nan
1994年の論文
@ja
1994年学术文章
@wuu
1994年学术文章
@zh-cn
1994年学术文章
@zh-hans
1994年学术文章
@zh-my
1994年学术文章
@zh-sg
1994年學術文章
@yue
1994年學術文章
@zh
1994年學術文章
@zh-hant
name
Prevention of nephritis in maj ...... ass II-deficient MRL-lpr mice.
@ast
Prevention of nephritis in maj ...... ass II-deficient MRL-lpr mice.
@en
type
label
Prevention of nephritis in maj ...... ass II-deficient MRL-lpr mice.
@ast
Prevention of nephritis in maj ...... ass II-deficient MRL-lpr mice.
@en
prefLabel
Prevention of nephritis in maj ...... ass II-deficient MRL-lpr mice.
@ast
Prevention of nephritis in maj ...... ass II-deficient MRL-lpr mice.
@en
P2093
P2860
P356
P1476
Prevention of nephritis in maj ...... ass II-deficient MRL-lpr mice.
@en
P2093
Glimcher LH
Jevnikar AM
P2860
P304
P356
10.1084/JEM.179.4.1137
P407
P577
1994-04-01T00:00:00Z