Hormone-regulated v-rel estrogen receptor fusion protein: reversible induction of cell transformation and cellular gene expression
about
Conditional transformation of cells and rapid activation of the mitogen-activated protein kinase cascade by an estradiol-dependent human raf-1 protein kinase.AP-1 factors play an important role in transformation induced by the v-rel oncogene.Rel-dependent induction of A1 transcription is required to protect B cells from antigen receptor ligation-induced apoptosisConstitutive and inducible kappa B binding activities in the cytosol of v-Rel-transformed lymphoid cells.Mutations in the DNA-binding and dimerization domains of v-Rel are responsible for altered kappa B DNA-binding complexes in transformed cellsAvian I kappa B alpha is transcriptionally induced by c-Rel and v-Rel with different kineticsIntranuclear localization of human papillomavirus 16 E7 during transformation and preferential binding of E7 to the Rb family member p130v-rel Induces ectopic expression of an adhesion molecule, DM-GRASP, during B-lymphoma developmentInteraction of the v-Rel oncoprotein with NF-kappaB and IkappaB proteins: heterodimers of a transformation-defective v-Rel mutant and NF-2 are functional in vitro and in vivo.v-rel induces expression of three avian immunoregulatory surface receptors more efficiently than c-rel.Evolution of the oncogenic potential of v-rel: rel-induced expression of immunoregulatory receptors correlates with tumor development and in vitro transformationTransformation of avian fibroblasts overexpressing the c-rel proto-oncogene and a variant of c-rel lacking 40 C-terminal amino acids.In vivo evolution of c-rel oncogenic potential.Temperature-sensitive transforming mutants of the v-rel oncogene.NF-kappa B p100 is one of the high-molecular-weight proteins complexed with the v-Rel oncoprotein in transformed chicken spleen cells.A modified oestrogen receptor ligand-binding domain as an improved switch for the regulation of heterologous proteins.A mutant v-rel with increased ability to transform B lymphocytes.The v-rel oncogene promotes malignant T-cell leukemia/lymphoma in transgenic mice.Myb proteins inhibit fibroblast transformation by v-RelIdentification of a novel antiapoptotic protein, GAM-1, encoded by the CELO adenovirus.Cells transformed by a v-Myb-estrogen receptor fusion differentiate into multinucleated giant cells.Interferon regulatory factor 1 (IRF-1) mediates cell growth inhibition by transactivation of downstream target genes.The estrogen receptor cooperates with the TGF alpha receptor (c-erbB) in regulation of chicken erythroid progenitor self-renewal.
P2860
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P2860
Hormone-regulated v-rel estrogen receptor fusion protein: reversible induction of cell transformation and cellular gene expression
description
1992 nî lūn-bûn
@nan
1992 թուականի Դեկտեմբերին հրատարակուած գիտական յօդուած
@hyw
1992 թվականի դեկտեմբերին հրատարակված գիտական հոդված
@hy
1992年の論文
@ja
1992年論文
@yue
1992年論文
@zh-hant
1992年論文
@zh-hk
1992年論文
@zh-mo
1992年論文
@zh-tw
1992年论文
@wuu
name
Hormone-regulated v-rel estrog ...... n and cellular gene expression
@ast
Hormone-regulated v-rel estrog ...... n and cellular gene expression
@en
type
label
Hormone-regulated v-rel estrog ...... n and cellular gene expression
@ast
Hormone-regulated v-rel estrog ...... n and cellular gene expression
@en
prefLabel
Hormone-regulated v-rel estrog ...... n and cellular gene expression
@ast
Hormone-regulated v-rel estrog ...... n and cellular gene expression
@en
P2093
P2860
P1433
P1476
Hormone-regulated v-rel estrog ...... n and cellular gene expression
@en
P2093
G Boehmelt
G Mellitzer
P J Enrietto
P2860
P304
P407
P577
1992-12-01T00:00:00Z