v-rel induces expression of three avian immunoregulatory surface receptors more efficiently than c-rel.
about
A novel interferon regulatory factor (IRF), IRF-10, has a unique role in immune defense and is induced by the v-Rel oncoprotein.Mutational analysis of the v-Rel dimerization interface reveals a critical role for v-Rel homodimers in transformationAP-1 factors play an important role in transformation induced by the v-rel oncogene.Interferon regulatory factor 4 contributes to transformation of v-Rel-expressing fibroblastsCell transformation by v-Rel reveals distinct roles of AP-1 family members in Rel/NF-kappaB oncogenesis.Mechanism of telomerase activation by v-Rel and its contribution to transformation.Differential regulation of the inhibitor of apoptosis ch-IAP1 by v-rel and the proto-oncogene c-rel.The relocalization of v-Rel from the nucleus to the cytoplasm coincides with induction of expression of Ikba and nfkb1 and stabilization of I kappa B-alpha.Constitutive and inducible kappa B binding activities in the cytosol of v-Rel-transformed lymphoid cells.Mutations in the DNA-binding and dimerization domains of v-Rel are responsible for altered kappa B DNA-binding complexes in transformed cellsv-rel Induces ectopic expression of an adhesion molecule, DM-GRASP, during B-lymphoma developmentInteraction of the v-Rel oncoprotein with NF-kappaB and IkappaB proteins: heterodimers of a transformation-defective v-Rel mutant and NF-2 are functional in vitro and in vivo.ch-IAP1, a member of the inhibitor-of-apoptosis protein family, is a mediator of the antiapoptotic activity of the v-Rel oncoprotein.Evolution of the oncogenic potential of v-rel: rel-induced expression of immunoregulatory receptors correlates with tumor development and in vitro transformationIn vivo evolution of c-rel oncogenic potential.CAPERalpha is a novel Rel-TAD-interacting factor that inhibits lymphocyte transformation by the potent Rel/NF-kappaB oncoprotein v-Rel.Rel-deficient T cells exhibit defects in production of interleukin 3 and granulocyte-macrophage colony-stimulating factorA mutant v-rel with increased ability to transform B lymphocytes.The v-rel oncogene promotes malignant T-cell leukemia/lymphoma in transgenic mice.PEST-dependent cytoplasmic retention of v-Rel by I(kappa)B-alpha: evidence that I(kappa)B-alpha regulates cellular localization of c-Rel and v-Rel by distinct mechanisms.Activation of gga-miR-155 by reticuloendotheliosis virus T strain and its contribution to transformation.Reticuloendotheliosis Virus Inhibits the Immune Response Acting on Lymphocytes from Peripheral Blood of Chicken.Inhibition of v-rel-Induced Oncogenesis through microRNA Targeting.
P2860
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P2860
v-rel induces expression of three avian immunoregulatory surface receptors more efficiently than c-rel.
description
1994 nî lūn-bûn
@nan
1994年の論文
@ja
1994年論文
@yue
1994年論文
@zh-hant
1994年論文
@zh-hk
1994年論文
@zh-mo
1994年論文
@zh-tw
1994年论文
@wuu
1994年论文
@zh
1994年论文
@zh-cn
name
v-rel induces expression of th ...... s more efficiently than c-rel.
@ast
v-rel induces expression of th ...... s more efficiently than c-rel.
@en
type
label
v-rel induces expression of th ...... s more efficiently than c-rel.
@ast
v-rel induces expression of th ...... s more efficiently than c-rel.
@en
prefLabel
v-rel induces expression of th ...... s more efficiently than c-rel.
@ast
v-rel induces expression of th ...... s more efficiently than c-rel.
@en
P2093
P2860
P1433
P1476
v-rel induces expression of th ...... rs more efficiently than c-rel
@en
P2093
E H Humphries
R Hrdlicková
P2860
P304
P407
P577
1994-01-01T00:00:00Z