RETRACTED: p73alpha regulation by Chk1 in response to DNA damage
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ATR/Chk1/Smurf1 pathway determines cell fate after DNA damage by controlling RhoB abundanceInhibitory role of Plk1 in the regulation of p73-dependent apoptosis through physical interaction and phosphorylationThe DNA damage pathway regulates innate immune system ligands of the NKG2D receptorRhoJ regulates melanoma chemoresistance by suppressing pathways that sense DNA damageCheckpoint kinase 1 in DNA damage response and cell cycle regulationRoles of Chk1 in cell biology and cancer therapyDeltaNp73 modulates nerve growth factor-mediated neuronal differentiation through repression of TrkAp73 as a pharmaceutical target for cancer therapyTP63 and TP73 in cancer, an unresolved "family" puzzle of complexity, redundancy and hierarchy.Gain of cellular adaptation due to prolonged p53 impairment leads to functional switchover from p53 to p73 during DNA damage in acute myeloid leukemia cellsA p53-derived apoptotic peptide derepresses p73 to cause tumor regression in vivo.Regulation of p73 activity by post-translational modifications.p73, a sophisticated p53 family member in the cancer world.Polo-like kinase 2 acting as a promoter in human tumor cells with an abundance of TAp73.PLK2 phosphorylates and inhibits enriched TAp73 in human osteosarcoma cellsCataloging and organizing p73 interactions in cell cycle arrest and apoptosisTherapeutic prospects for p73 and p63: rising from the shadow of p53.Global analysis of phosphorylation of tau by the checkpoint kinases Chk1 and Chk2 in vitroSignificant associations of mismatch repair gene polymorphisms with clinical outcome of pancreatic cancer.Modulation of the E2F1-driven cancer cell fate by the DNA damage response machinery and potential novel E2F1 targets in osteosarcomasp53 family: Therapeutic targets in neuroblastoma.p53-family proteins and their regulators: hubs and spokes in tumor suppression.p53: the attractive tumor suppressor in the cancer research field.TAp63alpha induces apoptosis by activating signaling via death receptors and mitochondriap73 induction after DNA damage is regulated by checkpoint kinases Chk1 and Chk2.p73-Binding Partners and Their Functional Significance.Ribosomal proteins L11 and L5 activate TAp73 by overcoming MDM2 inhibition.p73 expression is regulated by ribosomal protein RPL26 through mRNA translation and protein stability.IkappaB kinase beta promotes cell survival by antagonizing p53 functions through DeltaNp73alpha phosphorylation and stabilization.Roles of PKC isoforms in the induction of apoptosis elicited by aberrant Ras.TIP60 represses transcriptional activity of p73beta via an MDM2-bridged ternary complex.Oncogenic activity of Cdc6 through repression of the INK4/ARF locus.Identification of protein kinase A catalytic subunit beta as a novel binding partner of p73 and regulation of p73 function.AMPK couples p73 with p53 in cell fate decision.Regulation of p73-mediated apoptosis by c-Jun N-terminal kinase.Mdm2-mediated NEDD8 modification of TAp73 regulates its transactivation function.p73 coordinates with Δ133p53 to promote DNA double-strand break repair.
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RETRACTED: p73alpha regulation by Chk1 in response to DNA damage
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article científic
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article scientifique
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articolo scientifico
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artigo científico
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bilimsel makale
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retracted article
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RETRACTED: p73alpha regulation by Chk1 in response to DNA damage
@en
p73alpha regulation by Chk1 in response to DNA damage.
@nl
type
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RETRACTED: p73alpha regulation by Chk1 in response to DNA damage
@en
p73alpha regulation by Chk1 in response to DNA damage.
@nl
prefLabel
RETRACTED: p73alpha regulation by Chk1 in response to DNA damage
@en
p73alpha regulation by Chk1 in response to DNA damage.
@nl
P2860
P1476
p73alpha regulation by Chk1 in response to DNA damage
@en
P2093
Carol Prives
Susana Gonzalez
P2860
P304
P356
10.1128/MCB.23.22.8161-8171.2003
P407
P577
2003-11-01T00:00:00Z