Double-strand break repair in Ku86- and XRCC4-deficient cells. - Wikidata - wikimedia - TriplyDB

Double-strand break repair in Ku86- and XRCC4-deficient cells.

Double-strand break repair in Ku86- and XRCC4-deficient cells.

http://www.wikidata.org/entity/Q39725806

about

Alternative end-joining mechanisms: a historical perspective Ku regulates the non-homologous end joining pathway choice of DNA double-strand break repair in human somatic cells The role of DNA polymerase activity in human non-homologous end joining Ku is a 5'-dRP/AP lyase that excises nucleotide damage near broken ends DNA binding of Xrcc4 protein is associated with V(D)J recombination but not with stimulation of DNA ligase IV activity Association of DNA polymerase mu (pol mu) with Ku and ligase IV: role for pol mu in end-joining double-strand break repair.Chk2 phosphorylation of BRCA1 regulates DNA double-strand break repair Distinct requirements for Ku in N nucleotide addition at V(D)J- and non-V(D)J-generated double-strand breaks Robust chromosomal DNA repair via alternative end-joining in the absence of X-ray repair cross-complementing protein 1 (XRCC1)Deficiency of human BRCA2 leads to impaired homologous recombination but maintains normal nonhomologous end joining Biochemical evidence for Ku-independent backup pathways of NHEJ Effects of camptothecin on double-strand break repair by non-homologous end-joining in DNA mismatch repair-deficient human colorectal cancer cell lines Ku and the Stability of the Genome Homologous Recombination Deficiency: Exploiting the Fundamental Vulnerability of Ovarian Cancer Double-strand break repair-adox: Restoration of suppressed double-strand break repair during mitosis induces genomic instability Balancing self-renewal against genome preservation in stem cells: How do they manage to have the cake and eat it too?DNA Polymerase θ: A Unique Multifunctional End-Joining Machine Microhomology-Mediated End Joining: A Back-up Survival Mechanism or Dedicated Pathway?IgH class switching and translocations use a robust non-classical end-joining pathway The response to and repair of RAG-mediated DNA double-strand breaks Homology and enzymatic requirements of microhomology-dependent alternative end joining Modulation of DNA end joining by nuclear proteins Sibling rivalry: competition between Pol X family members in V(D)J recombination and general double strand break repair ERCC1-XPF endonuclease facilitates DNA double-strand break repair Novel functional requirements for non-homologous DNA end joining in Schizosaccharomyces pombe.MMEJ repair of double-strand breaks (director's cut): deleted sequences and alternative endings WRN regulates pathway choice between classical and alternative non-homologous end joining Extensive ssDNA end formation at DNA double-strand breaks in non-homologous end-joining deficient cells during the S phase Numt-mediated double-strand break repair mitigates deletions during primate genome evolution RAG2's acidic hinge restricts repair-pathway choice and promotes genomic stability.The Mre11/Rad50/Nbs1 complex functions in resection-based DNA end joining in Xenopus laevis Alternative end-joining catalyzes class switch recombination in the absence of both Ku70 and DNA ligase 4.RAG2 mutants alter DSB repair pathway choice in vivo and illuminate the nature of 'alternative NHEJ'.Alternative end-joining catalyzes robust IgH locus deletions and translocations in the combined absence of ligase 4 and Ku70 ATM modulates the loading of recombination proteins onto a chromosomal translocation breakpoint hotspot.Nonhomologous DNA end joining in cell-free systems.53BP1 regulates DNA resection and the choice between classical and alternative end joining during class switch recombination.Suppression of gene amplification and chromosomal DNA integration by the DNA mismatch repair system Coincident In Vitro Analysis of DNA-PK-Dependent and -Independent Nonhomologous End Joining.Canonical non-homologous end joining in mitosis induces genome instability and is suppressed by M-phase-specific phosphorylation of XRCC4

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P2860

Double-strand break repair in Ku86- and XRCC4-deficient cells.

http://www.wikidata.org/entity/Q39725806

description

1998 nî lūn-bûn

@nan

1998年の論文

@ja

1998年論文

@yue

1998年論文

@zh-hant

1998年論文

@zh-hk

1998年論文

@zh-mo

1998年論文

@zh-tw

1998年论文

@wuu

1998年论文

@zh

1998年论文

@zh-cn

name

Double-strand break repair in Ku86- and XRCC4-deficient cells.

@en

Double-strand break repair in Ku86- and XRCC4-deficient cells.

@nl

type

label

Double-strand break repair in Ku86- and XRCC4-deficient cells.

@en

Double-strand break repair in Ku86- and XRCC4-deficient cells.

@nl

prefLabel

Double-strand break repair in Ku86- and XRCC4-deficient cells.

@en

Double-strand break repair in Ku86- and XRCC4-deficient cells.

@nl

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Nucleic Acids Research

P1476

Double-strand break repair in Ku86- and XRCC4-deficient cells.

@en

P2093

D B Roth

http://www.w3.org/2001/XMLSchema#string

E B Kabotyanski

http://www.w3.org/2001/XMLSchema#string

J O Han

http://www.w3.org/2001/XMLSchema#string

L Gomelsky

http://www.w3.org/2001/XMLSchema#string

T D Stamato

http://www.w3.org/2001/XMLSchema#string

P2860

Activity of DNA ligase IV stimulated by complex formation with XRCC4 protein in mammalian cells

Mammalian DNA double-strand break repair protein XRCC4 interacts with DNA ligase IV

Yeast DNA ligase IV mediates non-homologous DNA end joining.

Saccharomyces cerevisiae LIF1: a function involved in DNA double-strand break repair related to mammalian XRCC4.

Ku80: product of the XRCC5 gene and its role in DNA repair and V(D)J recombination

The XRCC4 gene product is a target for and interacts with the DNA-dependent protein kinase

The XRCC4 gene encodes a novel protein involved in DNA double-strand break repair and V(D)J recombination

RAG1 and RAG2 form a stable postcleavage synaptic complex with DNA containing signal ends in V(D)J recombination

The DNA-dependent protein kinase: requirement for DNA ends and association with Ku antigen

Ku protein stimulates DNA end joining by mammalian DNA ligases: a direct role for Ku in repair of DNA double-strand breaks.

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5333-5342

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10.1093/NAR/26.23.5333

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23

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26

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9826756

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147996

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