Mitochondrial metabolism in cancer metastasis: visualizing tumor cell mitochondria and the "reverse Warburg effect" in positive lymph node tissue. - Wikidata - awgldk - TriplyDB

Mitochondrial metabolism in cancer metastasis: visualizing tumor cell mitochondria and the "reverse Warburg effect" in positive lymph node tissue.

Mitochondrial metabolism in cancer metastasis: visualizing tumor cell mitochondria and the "reverse Warburg effect" in positive lymph node tissue.

http://www.wikidata.org/entity/Q35954906

about

Oncogenes induce the cancer-associated fibroblast phenotype: metabolic symbiosis and "fibroblast addiction" are new therapeutic targets for drug discovery Interplay between sirtuins, MYC and hypoxia-inducible factor in cancer-associated metabolic reprogramming Autophagy: a targetable linchpin of cancer cell metabolism How does the metabolism of tumour cells differ from that of normal cells A small molecule with anticancer and antimetastatic activities induces rapid mitochondrial-associated necrosis in breast cancer.AMPK activity regulates trafficking of mitochondria to the leading edge during cell migration and matrix invasion.Migration and invasion of drug-resistant lung adenocarcinoma cells are dependent on mitochondrial activity Changes in mitochondrial function during EMT induced by TGFβ-1 in pancreatic cancer Teaching the basics of cancer metabolism: Developing antitumor strategies by exploiting the differences between normal and cancer cell metabolism Deregulation of the EGFR/PI3K/PTEN/Akt/mTORC1 pathway in breast cancer: possibilities for therapeutic intervention.Mitochondrial mass, a new metabolic biomarker for stem-like cancer cells: Understanding WNT/FGF-driven anabolic signaling.Autophagy and senescence in cancer-associated fibroblasts metabolically supports tumor growth and metastasis via glycolysis and ketone production Significance of glycolytic metabolism-related protein expression in colorectal cancer, lymph node and hepatic metastasis.CDK inhibitors (p16/p19/p21) induce senescence and autophagy in cancer-associated fibroblasts, "fueling" tumor growth via paracrine interactions, without an increase in neo-angiogenesis.Mitochondrial fission induces glycolytic reprogramming in cancer-associated myofibroblasts, driving stromal lactate production, and early tumor growth.Suppression of mitochondrial respiration with auraptene inhibits the progression of renal cell carcinoma: involvement of HIF-1α degradation.Mitochondria "fuel" breast cancer metabolism: fifteen markers of mitochondrial biogenesis label epithelial cancer cells, but are excluded from adjacent stromal cells Mitochondrial dysfunction in breast cancer cells prevents tumor growth: understanding chemoprevention with metformin.Impaired mitochondrial metabolism and mammary carcinogenesis CYC1 Predicts Poor Prognosis in Patients with Breast Cancer.Compartment-specific activation of PPARγ governs breast cancer tumor growth, via metabolic reprogramming and symbiosis.Metabolic control analysis of respiration in human cancer tissue.Monocarboxylate transporters in the brain and in cancer.Oncogenes and inflammation rewire host energy metabolism in the tumor microenvironment: RAS and NFκB target stromal MCT4.Antidepressant fluoxetine and its potential against colon tumors.β-alanine suppresses malignant breast epithelial cell aggressiveness through alterations in metabolism and cellular acidity in vitro.Mitochondrial mutations and metabolic adaptation in pancreatic cancer.Mitochondrial Dynamics in Retinal Ganglion Cell Axon Regeneration and Growth Cone Guidance.JNK1 stress signaling is hyper-activated in high breast density and the tumor stroma: connecting fibrosis, inflammation, and stemness for cancer prevention.Autophagic tumor stroma: mechanisms and roles in tumor growth and progression.Mitochondrial dysfunction and cancer metastasis.Mitochondria in relation to cancer metastasis.Targeting lactate metabolism for cancer therapeutics.Canonical and non-canonical Hedgehog signalling and the control of metabolism.Hitting the Bull's-Eye in Metastatic Cancers-NSAIDs Elevate ROS in Mitochondria, Inducing Malignant Cell Death.Mitochondrial Respiration in Human Colorectal and Breast Cancer Clinical Material Is Regulated Differently.Mitochondria and the hallmarks of cancer.Mitochondria and the non-genetic origins of cell-to-cell variability: More is different.Metabolic reprogramming underlies metastatic potential in an obesity-responsive murine model of metastatic triple negative breast cancer.A holistic view of cancer bioenergetics: mitochondrial function and respiration play fundamental roles in the development and progression of diverse tumors.

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Mitochondrial metabolism in cancer metastasis: visualizing tumor cell mitochondria and the "reverse Warburg effect" in positive lymph node tissue.

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Nancy J Philp

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Neal Flomenberg

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P2860

Autophagy in cancer associated fibroblasts promotes tumor cell survival: Role of hypoxia, HIF1 induction and NFκB activation in the tumor stromal microenvironment

Ketones and lactate "fuel" tumor growth and metastasis: Evidence that epithelial cancer cells use oxidative mitochondrial metabolism

The autophagic tumor stroma model of cancer: Role of oxidative stress and ketone production in fueling tumor cell metabolism

Oxidative stress in cancer associated fibroblasts drives tumor-stroma co-evolution: A new paradigm for understanding tumor metabolism, the field effect and genomic instability in cancer cells

Loss of stromal caveolin-1 expression predicts poor clinical outcome in triple negative and basal-like breast cancers

Stromal cell expression of caveolin-1 predicts outcome in breast cancer

An absence of stromal caveolin-1 expression predicts early tumor recurrence and poor clinical outcome in human breast cancers

Understanding the Warburg effect and the prognostic value of stromal caveolin-1 as a marker of a lethal tumor microenvironment

Stromal caveolin-1 levels predict early DCIS progression to invasive breast cancer

Mitochondrial respiratory-chain diseases

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Ubaldo E Martinez-Outschoorn

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