p53 represses CAAT enhancer-binding protein (C/EBP)-dependent transcription of the albumin gene. A molecular mechanism involved in viral liver infection with implications for hepatocarcinogenesis.
about
The transcription factor p53: not a repressor, solely an activatorPhysical interaction and mutual transrepression between CCAAT/enhancer-binding protein beta and the p53 tumor suppressorSAK, a new polo-like kinase, is transcriptionally repressed by p53 and induces apoptosis upon RNAi silencingNuclear export of phosphorylated C/EBPbeta mediates the inhibition of albumin expression by TNF-alphaThe p53-microRNA-34a axis regulates cellular entry receptors for tumor-associated human herpes viruses.Targeting of p53-transcriptional dysfunction by conditionally replicating adenovirus is not limited by p53-homologues.Adenoviral vector driven by a minimal Rad51 promoter is selective for p53-deficient tumor cells.Apoptosis in diseases of the liver.Tissue-specific and reversible RNA interference in transgenic mice.The inflammation-induced down-regulation of plasma Fetuin-A (alpha2HS-Glycoprotein) in liver results from the loss of interaction between long C/EBP isoforms at two neighbouring binding sitesVersatile functions of p53 protein in multicellular organismsC/EBPalpha is a DNA damage-inducible p53-regulated mediator of the G1 checkpoint in keratinocytes.Wild-type p53 and p73 negatively regulate expression of proliferation related genes.Transcriptional repression by p53 through direct binding to a novel DNA element.The human caspase-8 promoter sustains basal activity through SP1 and ETS-like transcription factors and can be up-regulated by a p53-dependent mechanism.CMV promoter is repressed by p53 and activated by JNK pathway.Tumor-specific adenoviral gene therapy: transcriptional repression of gene expression by utilizing p53-signal transduction pathways.Tumour-specific therapeutic adenovirus vectors: repression of transgene expression in healthy cells by endogenous p53.VP22-mediated intercellular transport of p53 in hepatoma cells in vitro and in vivo.Negative regulation of Chk2 expression by p53 is dependent on the CCAAT-binding transcription factor NF-Y.
P2860
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P2860
p53 represses CAAT enhancer-binding protein (C/EBP)-dependent transcription of the albumin gene. A molecular mechanism involved in viral liver infection with implications for hepatocarcinogenesis.
description
1999 nî lūn-bûn
@nan
1999年の論文
@ja
1999年学术文章
@wuu
1999年学术文章
@zh
1999年学术文章
@zh-cn
1999年学术文章
@zh-hans
1999年学术文章
@zh-my
1999年学术文章
@zh-sg
1999年學術文章
@yue
1999年學術文章
@zh-hant
name
p53 represses CAAT enhancer-bi ...... ions for hepatocarcinogenesis.
@en
p53 represses CAAT enhancer-binding protein
@nl
type
label
p53 represses CAAT enhancer-bi ...... ions for hepatocarcinogenesis.
@en
p53 represses CAAT enhancer-binding protein
@nl
prefLabel
p53 represses CAAT enhancer-bi ...... ions for hepatocarcinogenesis.
@en
p53 represses CAAT enhancer-binding protein
@nl
P2093
P2860
P356
P1476
p53 represses CAAT enhancer-bi ...... ions for hepatocarcinogenesis.
@en
P2093
P2860
P304
32137-32144
P356
10.1074/JBC.274.45.32137
P407
P577
1999-11-01T00:00:00Z