The mouse and human genes encoding the recognition component of the N-end rule pathway
about
Alternative splicing results in differential expression, activity, and localization of the two forms of arginyl-tRNA-protein transferase, a component of the N-end rule pathwayCharacterization of the DOC1/APC10 subunit of the yeast and the human anaphase-promoting complexRING fingers mediate ubiquitin-conjugating enzyme (E2)-dependent ubiquitinationEDD, the human hyperplastic discs protein, has a role in progesterone receptor coactivation and potential involvement in DNA damage responseAssociation of the human papillomavirus type 16 E7 oncoprotein with the 600-kDa retinoblastoma protein-associated factor, p600A family of mammalian E3 ubiquitin ligases that contain the UBR box motif and recognize N-degrons.The N-end rule pathwayCdc53/cullin and the essential Hrt1 RING-H2 subunit of SCF define a ubiquitin ligase module that activates the E2 enzyme Cdc34Female lethality and apoptosis of spermatocytes in mice lacking the UBR2 ubiquitin ligase of the N-end rule pathwaySynthetic heterovalent inhibitors targeting recognition E3 components of the N-end rule pathway'VIT1', a novel gene associated with vitiligoStructural basis of ubiquitin recognition by the ubiquitin-associated (UBA) domain of the ubiquitin ligase EDDRpn4 is a physiological substrate of the Ubr2 ubiquitin ligase.Degradation of HIV-1 integrase by the N-end rule pathwayMuscle cachexia: current concepts of intracellular mechanisms and molecular regulationDeficiency of UBR1, a ubiquitin ligase of the N-end rule pathway, causes pancreatic dysfunction, malformations and mental retardation (Johanson-Blizzard syndrome)Comparative peptide binding studies of the PABC domains from the ubiquitin-protein isopeptide ligase HYD and poly(A)-binding protein. Implications for HYD functionThe ubiquitin-conjugating DNA repair enzyme HR6A is a maternal factor essential for early embryonic development in mice.Construction and analysis of mouse strains lacking the ubiquitin ligase UBR1 (E3alpha) of the N-end rule pathwayAltered activity, social behavior, and spatial memory in mice lacking the NTAN1p amidase and the asparagine branch of the N-end rule pathwayBiochemical and genetic studies of UBR3, a ubiquitin ligase with a function in olfactory and other sensory systemsThe substrate recognition domains of the N-end rule pathway.Modes of regulation of ubiquitin-mediated protein degradation.Modulation of myocardin function by the ubiquitin E3 ligase UBR5.Ubiquitin conjugation by the N-end rule pathway and mRNAs for its components increase in muscles of diabetic ratsUbiquitin ligases of the N-end rule pathway: assessment of mutations in UBR1 that cause the Johanson-Blizzard syndromeUBR2 of the N-end rule pathway is required for chromosome stability via histone ubiquitylation in spermatocytes and somatic cells.Pairs of dipeptides synergistically activate the binding of substrate by ubiquitin ligase through dissociation of its autoinhibitory domain.Impaired neurogenesis and cardiovascular development in mice lacking the E3 ubiquitin ligases UBR1 and UBR2 of the N-end rule pathway.Expression and biochemical characterization of the human enzyme N-terminal asparagine amidohydrolase.Varying intertrial interval reveals temporally defined memory deficits and enhancements in NTAN1-deficient mice.Amino-terminal arginylation targets endoplasmic reticulum chaperone BiP for autophagy through p62 bindingCharacterization of arginylation branch of N-end rule pathway in G-protein-mediated proliferation and signaling of cardiomyocytesPharmacological Modulation of the N-End Rule Pathway and Its Therapeutic Implications.UBR box N-recognin-4 (UBR4), an N-recognin of the N-end rule pathway, and its role in yolk sac vascular development and autophagyEdd, the murine hyperplastic disc gene, is essential for yolk sac vascularization and chorioallantoic fusion.The N-end rule and retroviral infection: no effect on integrase.Multivalency-assisted control of intracellular signaling pathways: application for ubiquitin- dependent N-end rule pathway.Ablation of Arg-tRNA-protein transferases results in defective neural tube development.Protein degradation and memory formation.
P2860
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P2860
The mouse and human genes encoding the recognition component of the N-end rule pathway
description
1998 nî lūn-bûn
@nan
1998 թուականի Յուլիսին հրատարակուած գիտական յօդուած
@hyw
1998 թվականի հուլիսին հրատարակված գիտական հոդված
@hy
1998年の論文
@ja
1998年論文
@yue
1998年論文
@zh-hant
1998年論文
@zh-hk
1998年論文
@zh-mo
1998年論文
@zh-tw
1998年论文
@wuu
name
The mouse and human genes encoding the recognition component of the N-end rule pathway
@ast
The mouse and human genes encoding the recognition component of the N-end rule pathway
@en
The mouse and human genes encoding the recognition component of the N-end rule pathway
@nl
type
label
The mouse and human genes encoding the recognition component of the N-end rule pathway
@ast
The mouse and human genes encoding the recognition component of the N-end rule pathway
@en
The mouse and human genes encoding the recognition component of the N-end rule pathway
@nl
prefLabel
The mouse and human genes encoding the recognition component of the N-end rule pathway
@ast
The mouse and human genes encoding the recognition component of the N-end rule pathway
@en
The mouse and human genes encoding the recognition component of the N-end rule pathway
@nl
P2093
P2860
P3181
P356
P1476
The mouse and human genes encoding the recognition component of the N-end rule pathway
@en
P2093
A Varshavsky
N A Jenkins
N G Copeland
P2860
P304
P3181
P356
10.1073/PNAS.95.14.7898
P407
P577
1998-07-07T00:00:00Z