Overexpression of catalytic subunit p110alpha of phosphatidylinositol 3-kinase increases glucose transport activity with translocation of glucose transporters in 3T3-L1 adipocytes.
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Potential role of 3-phosphoinositide-dependent protein kinase 1 (PDK1) in insulin-stimulated glucose transporter 4 translocation in adipocytesAssociation of the insulin receptor with phospholipase C-gamma (PLCgamma) in 3T3-L1 adipocytes suggests a role for PLCgamma in metabolic signaling by insulinInter-subunit interaction of gastric H+,K+-ATPase prevents reverse reaction of the transport cycle.Protein phosphatase-2C alpha as a positive regulator of insulin sensitivity through direct activation of phosphatidylinositol 3-kinase in 3T3-L1 adipocytesPhosphoinositide 3-kinase accelerates necrotic cell death during hypoxiaExpression of a prenylation-deficient Rab4 inhibits the GLUT4 translocation induced by active phosphatidylinositol 3-kinase and protein kinase BIdentification of the APS protein as a novel insulin receptor substratep85alpha gene generates three isoforms of regulatory subunit for phosphatidylinositol 3-kinase (PI 3-Kinase), p50alpha, p55alpha, and p85alpha, with different PI 3-kinase activity elevating responses to insulin.Munc18c regulates insulin-stimulated glut4 translocation to the transverse tubules in skeletal muscle.3-phosphoinositide-dependent protein kinase 1, an Akt1 kinase, is involved in dephosphorylation of Thr-308 of Akt1 in Chinese hamster ovary cells.Five isoforms of the phosphatidylinositol 3-kinase regulatory subunit exhibit different associations with receptor tyrosine kinases and their tyrosine phosphorylations.Activators of AMP-activated protein kinase enhance GLUT4 translocation and its glucose transport activity in 3T3-L1 adipocytes.Progesterone inhibits glucose uptake by affecting diverse steps of insulin signaling in 3T3-L1 adipocytes.From insulin receptor signalling to Glut 4 translocation abnormalities in obesity and insulin resistance.The role of G proteins in insulin signalling.Protein kinase B/Akt participates in GLUT4 translocation by insulin in L6 myoblastsSpatiotemporal Regulators for Insulin-Stimulated GLUT4 Vesicle Exocytosis.Muscle-specific Pten deletion protects against insulin resistance and diabetesSignaling pathways mediating insulin-stimulated glucose transport.Dual control of muscle cell survival by distinct growth factor-regulated signaling pathways.Paraquat-induced oxidative stress represses phosphatidylinositol 3-kinase activities leading to impaired glucose uptake in 3T3-L1 adipocytes.Transport of deoxy-D-glucose into lymphocytes of patients with polycystic ovary syndrome.Differential regulation of phosphoinositide 3-kinase adapter subunit variants by insulin in human skeletal muscle.Intracellular segregation of phosphatidylinositol-3,4,5-trisphosphate by insulin-dependent actin remodeling in L6 skeletal muscle cellsInsulin-stimulated glucose uptake occurs in specialized cells within the cumulus oocyte complex.Src kinases as targets for B cell acute lymphoblastic leukaemia therapy.Turning signals on and off: GLUT4 traffic in the insulin-signaling highway.Retinoid Chemoprevention: Who Can Benefit?Intracellular localization of phosphatidylinositide 3-kinase and insulin receptor substrate-1 in adipocytes: potential involvement of a membrane skeleton.Differential effects of constitutively active phosphatidylinositol 3-kinase on glucose transport, glycogen synthase activity, and DNA synthesis in 3T3-L1 adipocytes.A target of phosphatidylinositol 3,4,5-trisphosphate with a zinc finger motif similar to that of the ADP-ribosylation-factor GTPase-activating protein and two pleckstrin homology domains.Optogenetic activation reveals distinct roles of PIP3 and Akt in adipocyte insulin action.Beta-glucans in the treatment of diabetes and associated cardiovascular risks.Bidirectional modulation of insulin action by amino acids.Skeletal muscle insulin resistance: the interplay of local lipid excess and mitochondrial dysfunction.Phosphatidylinositol 3-phosphate [PtdIns3P] is generated at the plasma membrane by an inositol polyphosphate 5-phosphatase: endogenous PtdIns3P can promote GLUT4 translocation to the plasma membrane.p110α and p110β isoforms of PI3K signaling: are they two sides of the same coin?A new recombinant pituitary adenylate cyclase-activating peptide-derived peptide efficiently promotes glucose uptake and glucose-dependent insulin secretion.Positive and negative regulation of phosphoinositide 3-kinase-dependent signaling pathways by three different gene products of the p85alpha regulatory subunit.Insulin-responsive compartments containing GLUT4 in 3T3-L1 and CHO cells: regulation by amino acid concentrations
P2860
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P2860
Overexpression of catalytic subunit p110alpha of phosphatidylinositol 3-kinase increases glucose transport activity with translocation of glucose transporters in 3T3-L1 adipocytes.
description
1996 nî lūn-bûn
@nan
1996年の論文
@ja
1996年学术文章
@wuu
1996年学术文章
@zh-cn
1996年学术文章
@zh-hans
1996年学术文章
@zh-my
1996年学术文章
@zh-sg
1996年學術文章
@yue
1996年學術文章
@zh
1996年學術文章
@zh-hant
name
Overexpression of catalytic su ...... sporters in 3T3-L1 adipocytes.
@en
Overexpression of catalytic su ...... sporters in 3T3-L1 adipocytes.
@nl
type
label
Overexpression of catalytic su ...... sporters in 3T3-L1 adipocytes.
@en
Overexpression of catalytic su ...... sporters in 3T3-L1 adipocytes.
@nl
prefLabel
Overexpression of catalytic su ...... sporters in 3T3-L1 adipocytes.
@en
Overexpression of catalytic su ...... sporters in 3T3-L1 adipocytes.
@nl
P2093
P2860
P921
P356
P1476
Overexpression of catalytic su ...... sporters in 3T3-L1 adipocytes.
@en
P2093
Ishihara H
Katagiri H
Shibasaki Y
P2860
P304
16987-16990
P356
10.1074/JBC.271.29.16987
P407
P577
1996-07-01T00:00:00Z